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Research Hotspot & Journal Scope - Cardiac

Thyroid - DOI: 10.1089/thy.2018.0709
High Thyrotropin Is Critical for Cardiac Electrical Remodeling and Arrhythmia Vulnerability in Hypothyroidism

Julieta Fernandez-Ruocco · Monica Gallego · Ainhoa Rodriguez-de-Yurre · Julian Zayas-Arrabal · Leyre Echeazarra · Amaia Alquiza · Victor Fernández-López · Juan M. Rodriguez-Robledo · Oscar Brito · Ygor Schleier · Marisa Sepulveda · Natalia F. Oshiyama · Martin Vila-Petroff · Rosana A. Bassani · Emiliano H. Medei · Oscar Casis ·

Thyroid - DOI: 10.1089/thy.2018.0668
Changes in the Stoichiometry of Uniplex Decrease Mitochondrial Calcium Overload and Contribute to Tolerance of Cardiac Ischemia/Reperfusion Injury in Hypothyroidism

Héctor Chapoy-Villanueva · Christian Silva-Platas · Ana K. Gutiérrez-Rodríguez · Noemí García · Edgar Acuña-Morin · Leticia Elizondo-Montemayor · Yuriana Oropeza-Almazán · Alejandro Aguilar-Saenz · Gerardo García-Rivas ·

Chemistry Medicine

Unexpected cardiac arrest (ICU3) and unplanned extubation (ICU1) decreased significantly with MMC and then increased significantly after MMC, whereas ICU-acquired gastrointestinal bleeding, deep vein thrombosis, and pneumothorax did not differ significantly across the three phases.

Impact of morbidity and mortality conferences on patient outcome [10.4103/roaic.roaic_94_17]

CONCLUSIONS Exercise limitation is related to the inability to increase cardiac output during exercise and is notably due to the degree of chronotropic incompetence.

Heart rate response during exercise predicts exercise tolerance in adults with transposition of the great arteries and atrial switch operation. [10.1016/j.ijcard.2019.07.054]

We previously found that the cardiovascular-expressed K+ voltage-gated channel ancillary subunit KCNE4 sex specifically influences the cardiac RISK/SAFE pathway response to IR and that Kcne4 deletion testosterone dependently exacerbates cardiac IR injury.

Kcne4 deletion sex dependently inhibits the RISK pathway response and exacerbates hepatic ischemia-reperfusion injury in mice. [10.1152/ajpregu.00251.2018]

Herein, we hypothesized pharmacologic UPRmt activation may protect against cardiac ischemia-reperfusion (IR) injury, in an ATF5-dependent manner.

Cardioprotection by the mitochondrial unfolded protein response (UPRmt) requires ATF5. [10.1152/ajpheart.00244.2019]

This case report describes the importance of considering internal trauma, including cardiac trauma, in patients presenting with a penetrating thoracic injury.

Penetrating thoracic wound with cardiac trauma [10.1136/vetreccr-2019-000955]

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