4. Epithelial-Mesenchymal
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Pancreas
Research Hotspot & Journal Scope - Epithelial-Mesenchymal

Pancreas - DOI: 10.1097/MPA.0000000000001199
Expression of Epithelial-Mesenchymal Transition Proteins in Pancreatic Anaplastic (Undifferentiated) Carcinoma

Kazuyuki Ishida · Rina Yamashita · Mitsumasa Osakabe · Noriyuki Uesugi · Noriyuki Yamada · Hiroyuki Nitta · Fumiyoshi Fujishima · Fuyuhiko Motoi · Hiroyoshi Suzuki · Hiromune Shimamura · Yutaka Noda · Takashi Sawai · Michiaki Unno · Hironobu Sasano · Akira Sasaki · Tamotsu Sugai ·

Biology Medicine
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Pancreas - DOI: 10.1097/MPA.0000000000001432
Expression of Epithelial-Mesenchymal Transition Markers in Treated Pancreatic Ductal Adenocarcinoma.

Minhua Wang · Jeannelyn S Estrella · Matthew H Katz · Michael Kim · Asif Rashid · Jeffrey E Lee · Anirban Maitra · Ignacio I Wistuba · Robert A Wolff · Gauri R Varadhachary · Huamin Wang ·

Medicine
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Pancreas - DOI: 10.1097/MPA.0000000000001321
Inhibition of LONP1 Suppresses Pancreatic Cancer Progression Via c-Jun N-Terminal Kinase Pathway–Meditated Epithelial-Mesenchymal Transition

Can Liu · Hai Wang · Hao Li · Xiong Chen · Xiang Wu · Bin Lu · Wenling Zhang · Yong Zhou · Gary Guishan Xiao · Ge Gao ·

Chemistry Medicine
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In this exploratory substudy, we investigated whether RPC4046 modulates epithelial-mesenchymal transition (EMT).

High‐titer IgG4 to cow’s milk proteins and relationship to specific IgE in Pediatric Eosinophilic Esophagitis: 407 [10.1016/j.jaci.2018.12.410]

However, whether RKIP would alter the invasion and metastasis of non-small cell lung cancer (NSCLC) and play the role through suppressing epithelial-mesenchymal transition (EMT) remains to be explored.

Overexpression of Raf-1 kinase inhibitor protein inhibits cell invasion and migration in lung cancer cells through suppressing epithelial-mesenchymal transition [10.21037/32579]

Furthermore, Sal administration significantly suppresses epithelial-mesenchymal transition (EMT), as evidenced by a decreased expression of α-SMA, vimentin, TGF-β1, snail, slug, and a largely restored expression of E-cadherin.

Salidroside Ameliorates Renal Interstitial Fibrosis by Inhibiting the TLR4/NF-κB and MAPK Signaling Pathways [10.3390/ijms20051103]

Moreover, EZH2 could promote epithelial-mesenchymal transition by epigenetically silencing CDX1.

The Attenuation of Trophoblast Invasion Caused by the Downregulation of EZH2 Is Involved in the Pathogenesis of Human Recurrent Miscarriage [10.1016/j.omtn.2018.12.011]

Moreover, miR-138-5p overexpression suppressed cell epithelial–mesenchymal transition (EMT) phenomenon of BC by upregulating E-cadherin expression, but downregulating N-cadherin and Vimentin expression.

MicroRNA-138-5p inhibits cell migration, invasion and EMT in breast cancer by directly targeting RHBDD1 [10.1007/s12282-019-00989-w]

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Pancreatic Pancreatitis Patients Cancer Acute Adenocarcinoma Neuroendocrine Tumors Ductal Cell Outcomes Chronic Neoplasms Intraductal Papillary Mucinous Analysis Pancreatectomy Severe Pancreas Endoscopic Review Risk Tumor Islet Survival Impact Total Associated Expression Versus Outcome Imaging Carcinoma Meta-analysis Resectable Cystic Therapy Prognostic Disease Clinical Resonance Autotransplantation Incidence Duct Metastasis Fibrosis Diagnosis Prevalence Resection Systematic Advanced Primary Predicts Treatment Assessment Magnetic Patient Characteristics United States Early Opioid Mortality Computed Neoplasm Line Invasive Retrospective Trial Undergoing Diagnostic Tomography Pancreaticoduodenectomy Performance Predicting Lesions Serum Factors Trends Chemotherapy Metastatic Patterns Kinase Epithelial-Mesenchymal Transition Small Comparative Effect Type Hospitalized Rates Immunohistochemistry Development Recurrent Randomized Postoperative Significance Lymph Node
Pancreas | Academic Accelerator - About the Journal

Research Scope

Pancreas

Research Scope
Pancreatic Pancreatitis Patients Cancer Acute Adenocarcinoma Neuroendocrine Tumors Ductal Cell Outcomes Chronic Neoplasms Intraductal Papillary Mucinous Analysis Pancreatectomy Severe Pancreas
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