Oropharyngeal Aspiration(口咽抽吸)研究综述
Oropharyngeal Aspiration 口咽抽吸 - The protocol is a technically non-intensive and relatively inexpensive method that involves whole-body O3 inhalation followed by oropharyngeal aspiration of apoptotic cells (i. [1] For the in vivo work, C57BL/6J mice were subjected to oropharyngeal aspiration of 200 μg of whole DEP. [2] Oropharyngeal aspiration of PM for 8 weeks induced pulmonary inflammation and pulmonary small artery remodeling in mice, as well as increased serum C‐reactive protein and OPN concentrations and systolic blood pressure (SBP). [3] Our previous study suggested that the oropharyngeal aspiration of ZnONPs could cause eosinophilic airway inflammation and increase T helper 2 (Th2) cytokine expression in the absence of allergens in mice. [4] Methods: Male and female C57BL/6 mice were exposed to multi-walled carbon nanotubes (MWCNTs) or crystalline silica (cSiO2) via oropharyngeal aspiration. [5] 4%, oropharyngeal aspiration were 11. [6] CuO and its carboxylated (CuO COOH), methylaminated (CuO NH3) and PEGylated (CuO PEG) derivatives were administered here on four consecutive days via oropharyngeal aspiration in a mouse model of AAI. [7] 5 every other day by oropharyngeal aspiration for 4 weeks, and then, body and cardiac parameter, containing weight data, cardiac function, ultrastructure, metabolic analysis, and molecular detection were conducted to investigate the PM2. [8] Exposure to 100 µg of PM10 via oropharyngeal aspiration in C56BL/6 mice resulted in pulmonary inflammation across all groups. [9] METHODS We obtained human IPF specimens from patients during lung transplantation and administered bleomycin to male C57BL/6J mice either by oropharyngeal aspiration (1 U/kg) or subcutaneous osmotic infusion (100 U/kg over 7 days). [10] In the first investigation, different doses of graphene nanoplatelets were administered to mice via oropharyngeal aspiration. [11] Mice were exposed to doses (28, 140, or 280 μg/mouse) of UYP, MCA, or CA-C sanding dust using oropharyngeal aspiration. [12] 5, 125, 250, 500, or 1000 µg of SSC dust, or 1000 µg silica (positive control) via oropharyngeal aspiration. [13]该方案是一种技术上非密集且相对便宜的方法，涉及全身 O3 吸入，然后是凋亡细胞的口咽抽吸（即。 [1] 对于体内工作，C57BL/6J 小鼠经口咽抽吸 200μg 全 DEP。 [2] PM 的口咽抽吸 8 周可导致小鼠肺部炎症和肺小动脉重塑，以及血清 C 反应蛋白和 OPN 浓度和收缩压 (SBP) 增加。 [3] 我们之前的研究表明，在没有过敏原的小鼠中，ZnONPs 的口咽吸入可能会导致嗜酸性气道炎症并增加 T 辅助细胞 2 (Th2) 细胞因子的表达。 [4] 方法：雄性和雌性 C57BL/6 小鼠通过口咽抽吸暴露于多壁碳纳米管 (MWCNT) 或结晶二氧化硅 (cSiO2)。 [5] 4%，口咽误吸为 11。 [6] 在 AAI 小鼠模型中，通过口咽抽吸连续四天给予 CuO 及其羧化 (CuO COOH)、甲胺化 (CuO NH3) 和聚乙二醇化 (CuO PEG) 衍生物。 [7] 隔日5次经口咽抽吸4周，然后进行体心参数、体重数据、心功能、超微结构、代谢分析、分子检测等，对PM2.5进行调查。 [8] 在 C56BL/6 小鼠中通过口咽吸入暴露于 100 µg PM10 会导致所有组的肺部炎症。 [9] 方法 我们在肺移植期间从患者那里获得了人类 IPF 标本，并通过口咽抽吸 (1 U/kg) 或皮下渗透输注 (100 U/kg 7 天) 将博来霉素施用于雄性 C57BL/6J 小鼠。 [10] 在第一项研究中，通过口咽抽吸将不同剂量的石墨烯纳米片给予小鼠。 [11] 使用口咽抽吸将小鼠暴露于剂量（28、140 或 280μg/小鼠）的 UYP、MCA 或 CA-C 砂尘。 [12] 通过口咽抽吸 5、125、250、500 或 1000 µg SSC 粉尘，或 1000 µg 二氧化硅（阳性对照）。 [13]