Corticostriatal Circuits(皮质纹状体回路)研究综述
Corticostriatal Circuits 皮质纹状体回路 - Food and energy intake were monitored and ex vivo electrophysiology was employed to assess neuroadaptations in lateral hypothalamus (LH) and corticostriatal circuits, previously associated with food intake. [1] Conclusions ASD and BDD share the basis of corticostriatal circuits. [2] laterality) in corticostriatal circuits is a shared feature of numerous neurodevelopmental and psychiatric disorders. [3] Local field potentials (LFPs) simultaneously recorded in prefrontal cortex (PFC) and striatum (Str) demonstrate a decrease in γhigh-band coherent activity and spike-field coherence in corticostriatal circuits of APP/PS1 mice. [4] MCI) based on resting-state functional connectivity (rsFC) of the corticostriatal circuits. [5] The role of corticostriatal circuits in language functions is unclear. [6] We asked whether we could identify the targets of such corticostriatal circuits when the cortical microstimulation sites were explicitly identified as affecting approach or avoidance in the Ap‐Av task. [7] Mutations in ASD-associated SHANK3 in mice (Shank3B−/−) result in the accelerated maturation of corticostriatal circuits during the second and third postnatal weeks. [8] Recent neuroanatomical models add to the traditional view of dysfunction in corticostriatal circuits by proposing alterations in an affective circuit including amygdala-prefrontal connections. [9] Background Corticostriatal circuits (CSC) have been implicated in the presentation of some restricted and repetitive behaviours (RRBs) in children with autism-spectrum disorder (ASD), and preliminary evidence suggests that disruptions in these pathways may be associated with differences in genetic and environmental influences on brain development. [10] The heterogeneity of the corticostriatal circuits, however, also renders the system susceptible to a repertoire of neurological diseases. [11] Furthermore, we detected a lower paired-pulse ratio (PPR) and impaired long-term potentiation (LTP) in corticostriatal circuits in Asic3 null mutant mice as compared with wild-type (WT) littermates. [12] CONCLUSIONS Applying CTN-DBS to ASD rat offspring increased FC and altered the synaptic plasticity in the corticolimbic and the corticostriatal circuits. [13] Shape changes correlated with functional measures subserved by corticostriatal circuits, suggesting that the neostriatum is a potentially useful structural basis for characterisation of endophenotypes of HD. [14] Rewarding music altered NAc connectivity with key nodes of the corticostriatal circuits during pain onset. [15]监测食物和能量摄入，并采用离体电生理学评估下丘脑外侧 (LH) 和皮质纹状体回路中的神经适应，这些回路以前与食物摄入有关。 [1] 结论 ASD 和 BDD 共享皮质纹状体回路的基础。 [2] 皮质纹状体回路中的侧向性）是许多神经发育和精神疾病的共同特征。 [3] 同时记录在前额叶皮层 (PFC) 和纹状体 (Str) 中的局部场电位 (LFP) 表明 APP/PS1 小鼠皮质纹状体回路中的 γ 高频带相干活动和尖峰场相干性降低。 [4] MCI) 基于皮质纹状体电路的静息状态功能连接 (rsFC)。 [5] 皮质纹状体回路在语言功能中的作用尚不清楚。 [6] 当皮层微刺激位点被明确识别为影响 Ap-Av 任务中的接近或回避时，我们询问我们是否可以识别这种皮层纹状体回路的目标。 [7] 小鼠 ASD 相关 SHANK3 的突变 (Shank3B-/-) 导致皮质纹状体回路在出生后第二周和第三周加速成熟。 [8] 最近的神经解剖模型通过提出包括杏仁核-前额叶连接在内的情感回路的改变，增加了皮质纹状体回路功能障碍的传统观点。 [9] 背景 皮质纹状体回路 (CSC) 与自闭症谱系障碍 (ASD) 儿童的一些限制性和重复性行为 (RRB) 的表现有关，初步证据表明，这些通路的中断可能与遗传和环境影响的差异有关关于大脑发育。 [10] 然而，皮质纹状体回路的异质性也使该系统容易受到一系列神经系统疾病的影响。 [11] 此外，与野生型 (WT) 同窝小鼠相比，我们在 Asic3 无效突变小鼠的皮质纹状体回路中检测到较低的配对脉冲比 (PPR) 和受损的长期增强 (LTP)。 [12] 结论 将 CTN-DBS 应用于 ASD 大鼠后代会增加 FC 并改变皮质边缘和皮质纹状体回路中的突触可塑性。 [13] 形状变化与皮质纹状体回路所提供的功能测量相关，表明新纹状体是表征 HD 内表型的潜在有用的结构基础。 [14] 在疼痛发作期间，奖励音乐改变了 NAc 与皮质纹状体回路关键节点的连接。 [15]
corticostriatal circuits contain
ConclusionsThe current data demonstrate that oscillations recorded from corticostriatal circuits contain significant information regarding alcohol drinking in males, but not alcohol drinking in females. [1] Conclusions The current data demonstrate that oscillations recorded from corticostriatal circuits contain significant information regarding alcohol drinking in males, but not alcohol drinking in females. [2]结论目前的数据表明，从皮质纹状体回路记录的振荡包含有关男性饮酒的重要信息，但不包含女性饮酒的重要信息。 [1] 结论 目前的数据表明，从皮质纹状体回路记录的振荡包含有关男性饮酒的重要信息，但不包含女性饮酒的重要信息。 [2]