Cancer Ags(癌症抗原)研究综述
Cancer Ags 癌症抗原 - The variety of potential effector cells and cancer Ags, along with potential combination therapies, make BsAbs an active area of drug development. [1]各种潜在的效应细胞和癌症抗原，以及潜在的联合疗法，使 BsAbs 成为药物开发的活跃领域。 [1]
Gastric Cancer Ags 胃癌公司
The DDP-resistant human gastric cancer AGS and HGC cell lines, AGS/DDP and HGC-27/DDP, respectively, were established and CTBP1 expression was detected by western blotting. [1] 5 μM significantly reduced total cell viability in human gastric cancer AGS cells. [2] These findings indicated that chrysin potentiated the chemotherapeutic effect of 5-FU in gastric cancer AGS and AGS/FR cells via cell cycle arrest. [3] The present study aimed to investigate whether ASX induces necroptosis by increasing NADPH oxidase activity and ROS levels in gastric cancer AGS cells. [4] Here, we aimed to evaluate the anticancer effect of novel gemini curcumin (Gemini-Cur) on the gastric cancer AGS cells. [5] Methods In this study, we investigated the inhibitory effect of crocin on gastric cancer AGS cells proliferation and explored the underlying mechanism. [6] Under the interaction of intratumoral H2O2, CO and gemcitabine (GEM) were released in situ from the micelles to reduce side effects, and CO significantly sensitized the chemotherapeutic effect of GEM by elevating the level of reactive oxygen species (ROS) in human gastric cancer AGS cells. [7] Furthermore, knockdown of WWTR1 impaired migration of gastric cancer AGS cells. [8] In this study, the cytotoxic effects of curcumin were investigated in gastric cancer AGS and SGC-7901 cell lines by MTT assay, and curcumin-induced morphological changes and cell apoptosis were assessed by using flow cytometry analysis and caspase-3 activity. [9] Moreover, the fibulin-2 overexpression plasmid was constructed and transfected into human gastric cancer AGS and SGC-790 cell lines, so as to observe changes in β-catenin and its downstream indexes. [10] Furthermore, we demonstrated that NKAP knockdown also played an oncogenic role in human gastric cancer AGS and MKN45 cells. [11] The aim of the present study was to investigate the mechanism by which lycopene induces apoptosis of the human gastric cancer AGS cells. [12] Apoptosis was quantified by annexin V/propidium iodide staining of gastric cancer AGS and colorectal cancer DLD-1 cells. [13] This study aims to investigate whether lycopene inhibits proliferation and induces apoptosis in gastric cancer AGS cells by suppressing the EGFR/Ras/MAPK and NF-κB-COX-2 signaling axis. [14] pylori infection in gastric cancer AGS cells. [15] We verified applicability of CyTOF in gastric cancer cells, and analyzed the responses of seventeen proteins to chemoradiotherapy in human gastric cancer AGS cells. [16] In our research, we observed the biological effect of salidroside on human gastric cancer AGS cells. [17] We investigated the effect of a lentivirus-mediated knock-down of ANXA2 on the proliferation, invasion and migration of gastric cancer AGS cells. [18] zedoaria rhizome was found to show a cytotoxic effect against gastric cancer AGS cells. [19] All halo-δ-lactones and δ-hydroxy-γ-lactones were highly cytotoxic against gastric cancer AGS cells with IC50 values in the range of 0. [20]分别建立抗DDP人胃癌AGS和HGC细胞系AGS/DDP和HGC-27/DDP，并通过蛋白质印迹检测CTBP1的表达。 [1] 5 μM 显着降低人胃癌 AGS 细胞的总细胞活力。 [2] 这些发现表明，白杨素通过细胞周期阻滞增强了 5-FU 在胃癌 AGS 和 AGS/FR 细胞中的化学治疗作用。 [3] 本研究旨在探讨 ASX 是否通过增加胃癌 AGS 细胞中的 NADPH 氧化酶活性和 ROS 水平来诱导坏死性凋亡。 [4] 在这里，我们旨在评估新型双子姜黄素 (Gemini-Cur) 对胃癌 AGS 细胞的抗癌作用。 [5] 方法在本研究中，我们研究了藏红花素对胃癌AGS细胞增殖的抑制作用，并探讨了其潜在机制。 [6] 在瘤内 H2O2 的相互作用下，CO 和吉西他滨 (GEM) 从胶束中原位释放以减少副作用，并且 CO 通过提高人胃癌 AGS 中的活性氧 (ROS) 水平显着增敏 GEM 的化疗效果细胞。 [7] 此外，WWTR1 的敲低损害了胃癌 AGS 细胞的迁移。 [8] 本研究通过 MTT 法研究姜黄素对胃癌 AGS 和 SGC-7901 细胞系的细胞毒作用，并通过流式细胞术分析和 caspase-3 活性评估姜黄素诱导的形态学变化和细胞凋亡。 [9] 此外，构建fibulin-2过表达质粒并转染人胃癌AGS和SGC-790细胞系，观察β-catenin及其下游指标的变化。 [10] 此外，我们证明了 NKAP 敲低在人胃癌 AGS 和 MKN45 细胞中也发挥了致癌作用。 [11] 本研究旨在探讨番茄红素诱导人胃癌AGS细胞凋亡的机制。 [12] 通过胃癌 AGS 和结肠直肠癌 DLD-1 细胞的膜联蛋白 V/碘化丙啶染色定量细胞凋亡。 [13] 本研究旨在探讨番茄红素是否通过抑制 EGFR/Ras/MAPK 和 NF-κB-COX-2 信号轴来抑制胃癌 AGS 细胞的增殖和诱导凋亡。 [14] 胃癌AGS细胞中的幽门螺杆菌感染。 [15] 我们验证了 CyTOF 在胃癌细胞中的适用性，并分析了 17 种蛋白质对人胃癌 AGS 细胞放化疗的反应。 [16] 在我们的研究中，我们观察了红景天苷对人胃癌AGS细胞的生物学作用。 [17] 我们研究了慢病毒介导的 ANXA2 敲低对胃癌 AGS 细胞增殖、侵袭和迁移的影响。 [18] 发现茯苓根茎对胃癌 AGS 细胞具有细胞毒作用。 [19] 所有卤代-δ-内酯和δ-羟基-γ-内酯均对胃癌AGS细胞具有高度细胞毒性，IC50值在0范围内。 [20]
cancer ags cell 癌症抗原细胞
5 μM significantly reduced total cell viability in human gastric cancer AGS cells. [1] The present study aimed to investigate whether ASX induces necroptosis by increasing NADPH oxidase activity and ROS levels in gastric cancer AGS cells. [2] Here, we aimed to evaluate the anticancer effect of novel gemini curcumin (Gemini-Cur) on the gastric cancer AGS cells. [3] Methods In this study, we investigated the inhibitory effect of crocin on gastric cancer AGS cells proliferation and explored the underlying mechanism. [4] Under the interaction of intratumoral H2O2, CO and gemcitabine (GEM) were released in situ from the micelles to reduce side effects, and CO significantly sensitized the chemotherapeutic effect of GEM by elevating the level of reactive oxygen species (ROS) in human gastric cancer AGS cells. [5] Furthermore, knockdown of WWTR1 impaired migration of gastric cancer AGS cells. [6] The aim of the present study was to investigate the mechanism by which lycopene induces apoptosis of the human gastric cancer AGS cells. [7] This study aims to investigate whether lycopene inhibits proliferation and induces apoptosis in gastric cancer AGS cells by suppressing the EGFR/Ras/MAPK and NF-κB-COX-2 signaling axis. [8] pylori infection in gastric cancer AGS cells. [9] We verified applicability of CyTOF in gastric cancer cells, and analyzed the responses of seventeen proteins to chemoradiotherapy in human gastric cancer AGS cells. [10] In our research, we observed the biological effect of salidroside on human gastric cancer AGS cells. [11] We investigated the effect of a lentivirus-mediated knock-down of ANXA2 on the proliferation, invasion and migration of gastric cancer AGS cells. [12] zedoaria rhizome was found to show a cytotoxic effect against gastric cancer AGS cells. [13] All halo-δ-lactones and δ-hydroxy-γ-lactones were highly cytotoxic against gastric cancer AGS cells with IC50 values in the range of 0. [14]5 μM 显着降低人胃癌 AGS 细胞的总细胞活力。 [1] 本研究旨在探讨 ASX 是否通过增加胃癌 AGS 细胞中的 NADPH 氧化酶活性和 ROS 水平来诱导坏死性凋亡。 [2] 在这里，我们旨在评估新型双子姜黄素 (Gemini-Cur) 对胃癌 AGS 细胞的抗癌作用。 [3] 方法在本研究中，我们研究了藏红花素对胃癌AGS细胞增殖的抑制作用，并探讨了其潜在机制。 [4] 在瘤内 H2O2 的相互作用下，CO 和吉西他滨 (GEM) 从胶束中原位释放以减少副作用，并且 CO 通过提高人胃癌 AGS 中的活性氧 (ROS) 水平显着增敏 GEM 的化疗效果细胞。 [5] 此外，WWTR1 的敲低损害了胃癌 AGS 细胞的迁移。 [6] 本研究旨在探讨番茄红素诱导人胃癌AGS细胞凋亡的机制。 [7] 本研究旨在探讨番茄红素是否通过抑制 EGFR/Ras/MAPK 和 NF-κB-COX-2 信号轴来抑制胃癌 AGS 细胞的增殖和诱导凋亡。 [8] 胃癌AGS细胞中的幽门螺杆菌感染。 [9] 我们验证了 CyTOF 在胃癌细胞中的适用性，并分析了 17 种蛋白质对人胃癌 AGS 细胞放化疗的反应。 [10] 在我们的研究中，我们观察了红景天苷对人胃癌AGS细胞的生物学作用。 [11] 我们研究了慢病毒介导的 ANXA2 敲低对胃癌 AGS 细胞增殖、侵袭和迁移的影响。 [12] 发现茯苓根茎对胃癌 AGS 细胞具有细胞毒作用。 [13] 所有卤代-δ-内酯和δ-羟基-γ-内酯均对胃癌AGS细胞具有高度细胞毒性，IC50值在0范围内。 [14]