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10.3390/ijms20153775

It is well-established that endothelial dysfunction precedes the onset of cardiovascular disease, therefore NOX are important molecular links between type 2 diabetes and vascular complications.

Endothelial Dysfunction: Is There a Hyperglycemia-Induced Imbalance of NOX and NOS?

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10.18585/INABJ.V11I2.576

Long-term hyperglycemia conditions promote endothelial dysfunction precedes to the development of multiple organ dysfunctions.

The Number and Potency of Endothelial Progenitor Cells in Type 2 Diabetes Mellitus Patients

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10.1139/apnm-2019-0445

These findings indicate that brachial artery dysfunction precedes an inefficient functional sympatholysis.

Impact of High Sodium Intake on Blood Pressure and Functional Sympatholysis during Rhythmic Handgrip Exercise.

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10.1007/s12035-019-1550-1

However, ALS is being redefined as a distal axonopathy, in that neuromuscular junction dysfunction precedes and may even influence motoneuron loss.

Overview of Impaired BDNF Signaling, Their Coupled Downstream Serine-Threonine Kinases and SNARE/SM Complex in the Neuromuscular Junction of the Amyotrophic Lateral Sclerosis Model SOD1-G93A Mice

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10.2147/DMSO.S211253

Conclusions Our findings suggest that HFD-induced microvascular dysfunction precedes the decline in renal function, and could be related to antioxidant machinery defects and inflammation activation in the kidney.

High-fat diet-induced kidney alterations in rats with metabolic syndrome: endothelial dysfunction and decreased antioxidant defense

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Function Precedes - It is well-established that endothelial dysfunction precedes the onset of cardiovascular disease, therefore NOX are important molecular links between type 2 diabetes and vascular complications. [1] Long-term hyperglycemia conditions promote endothelial dysfunction precedes to the development of multiple organ dysfunctions. [2] These findings indicate that brachial artery dysfunction precedes an inefficient functional sympatholysis. [3] However, ALS is being redefined as a distal axonopathy, in that neuromuscular junction dysfunction precedes and may even influence motoneuron loss. [4] Conclusions Our findings suggest that HFD-induced microvascular dysfunction precedes the decline in renal function, and could be related to antioxidant machinery defects and inflammation activation in the kidney. [5]