Introduction to Barley Leaves

Furthermore, aphids’ survival was also not affected when the nymphs fed on leaves supplied with dsRNA via basal cut ends of barley leaves.

The triplex assay was used to assess DNA of barley leaves from New South Wales, South Australia, Tasmania, Victoria and Western Australia, along with DNA of seeds from Western Australia.

Pathogenicity test showed ΔMoade4 was completely nonpathogenic on rice and barley leaves which was attributed by failure of its infectious hyphae to colonize the host cells.

Furthermore, the ΔMocpa1 mutant was nonpathogenic on rice and barley leaves, which was a result of defects in appressorium-mediated penetration and restricted invasive hyphal growth within host cells.

Resistance of plants to ionic toxicity of manganese (II) was judged by the value of antioxidant activity (AOA) of superoxide dismutase (SOD), the content of low-molecular antioxidants (chlorophyll a and b, carotenoids and anthocyanins) in barley leaves.

The consequences of this leaf disease are due to the impact of the infection on the photosynthetic performance of barley leaves.

Alfalfa and barley leaves were not consumed by beetles and did not support beetle reproduction.

The Br concentrations were always higher in barley, while the concentrations of pigments in barley leaves were lower than in leaves of wheat.

The ΔMogln2 mutant failed to produce spores and was nonpathogenic on barley leaves, as it was unable to form an appressorium-like structure from its hyphal tips.

The influence of light and atmospheric CO2 concentration ([CO2]) on the accumulation and localization of PhCs in barley leaves was examined for two varieties with different tolerances to oxidative stress.

Disruption of MoHse1 and MoVps27 caused pleiotropic defects in growth, conidiation, sexual development, and pathogenicity, thereby resulting in loss of virulence in rice and barley leaves.

Drought decreased the relative water content (RWC) and water potential (WP) in barley leaves.

The treatments of the barley leaves with JA and OPDA induced the accumulation of methoxylchalcones, but treatment with 9-KODE did not.

In addition, the nitrogen transporter mutants also showed significantly higher 15N incorporation of insect derived nitrogen in barley leaves in the presence of nutrients.

The main objective of this study was to identify the correlation magnitude (r) between salt stress level and both agronomic parameters and proline accumulation in barley leaves.

Generally, temperature of growth was significant factor influencing on the level of the accumulation of the H+-ATPase and HvPIP1 transcript and the PM H+-ATPase and HvPIP1 protein in barley leaves.

Mutation of the ENHANCED MAGNAPORTHE 1 (emr1) locus which can affect mlo-associated disease susceptibility, leads to a reduction in RLS symptoms on barley leaves but does not reduce R.

Relaxation of the non-photochemical quenching (NPQ) of Chl a fluorescence after illumination corresponding to the return of the part of LHC2 from PS1 to PS2 was observed in barley leaves in a wider range of increasing light intensities than in Arabidopsis leaves.

Pathogenicity assays showed that virulence of the Δfpada1 mutant was dramatically decreased on wheat coleoptiles and barley leaves.

15N from 15NO could be detected in RNA, DNA, and proteins of barley leaves and the 15N levels were significantly higher in HvPgb1.

These include phasiRNA loci that overlap with a significant proportion of receptor-like kinases, suggesting an additional sRNA control mechanism may be active in barley leaves as opposed to predominant R-gene phasiRNA overlap in many eudicots.

The reflectance of the barley leaves was significantly (p ≤ 0.

Disruption of MoYCK1 caused pleiotropic defects in growth, conidiation, conidial germination, and appressorium formation and penetration, therefore resulting in reduced virulence in rice seedlings and barley leaves.