Neat1 Regulates
Neat1 규제

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Taken together, this study demonstrated that the lncRNA NEAT1 regulates infantile pneumonia by sponging miR-146b.

종합하면, 이 연구는 lncRNA NEAT1이 miR-146b를 스폰지하여 유아 폐렴을 조절한다는 것을 입증했습니다.

LncRNA NEAT1 Regulates Infantile Pneumonia by Sponging miR-146b

Lncrna Neat1 Regulates

OBJECTIVE The present study was designed to investigate the mechanism by which lncRNA NEAT1 regulates survival and angiogenesis in oxygen-glucose deprivation (OGD)-induced brain microvascular endothelial cells (BMECs).

목적 본 연구는 lncRNA NEAT1이 산소-포도당 결핍(OGD) 유발 뇌 미세혈관 내피 세포(BMEC)에서 생존 및 혈관신생을 조절하는 메커니즘을 조사하기 위해 설계되었습니다.

LncRNA NEAT1 facilitates survival and angiogenesis in oxygen-glucose deprivation (OGD)-induced brain microvascular endothelial cells (BMECs) via targeting miR-377 and upregulating SIRT1, VEGFA, and BCL-XL

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10.1038/s41418-021-00858-0

These findings suggest that NEAT1 regulates the lineage fates of BMSCs by orchestrating mitochondrial function and pluripotency maintenance, and might be a potential therapeutic target for skeletal aging.

이러한 발견은 NEAT1이 미토콘드리아 기능과 다능성 유지를 조정함으로써 BMSC의 혈통 운명을 조절하고 골격 노화에 대한 잠재적인 치료 표적이 될 수 있음을 시사합니다.

LncRNA NEAT1 controls the lineage fates of BMSCs during skeletal aging by impairing mitochondrial function and pluripotency maintenance.

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Neat1 Regulates Neat1 규제 - These findings suggest that NEAT1 regulates the lineage fates of BMSCs by orchestrating mitochondrial function and pluripotency maintenance, and might be a potential therapeutic target for skeletal aging. [1]
이러한 발견은 NEAT1이 미토콘드리아 기능과 다능성 유지를 조정함으로써 BMSC의 혈통 운명을 조절하고 골격 노화에 대한 잠재적인 치료 표적이 될 수 있음을 시사합니다. [1]

Lncrna Neat1 Regulates

Taken together, this study demonstrated that the lncRNA NEAT1 regulates infantile pneumonia by sponging miR-146b. [1] OBJECTIVE The present study was designed to investigate the mechanism by which lncRNA NEAT1 regulates survival and angiogenesis in oxygen-glucose deprivation (OGD)-induced brain microvascular endothelial cells (BMECs). [2]
종합하면, 이 연구는 lncRNA NEAT1이 miR-146b를 스폰지하여 유아 폐렴을 조절한다는 것을 입증했습니다. [1] 목적 본 연구는 lncRNA NEAT1이 산소-포도당 결핍(OGD) 유발 뇌 미세혈관 내피 세포(BMEC)에서 생존 및 혈관신생을 조절하는 메커니즘을 조사하기 위해 설계되었습니다. [2]