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Discovery of GSK8612, a Highly Selective and Potent TBK1 Inhibitor.


Common Neurodegeneration-Associated Proteins Are Physiologically Expressed by Human B Lymphocytes and Are Interconnected via the Inflammation/Autophagy-Related Proteins TRAF6 and SQSTM1

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FIP200 controls the TBK1 activation threshold at SQSTM1/p62-positive condensates


Highlights from Recent Cancer Literature

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Human Metapneumovirus Induces IRF1 via TANK-Binding Kinase 1 and Type I IFN


ALS-FTLD-linked mutations of SQSTM1/p62 disrupt selective autophagy and NFE2L2/NRF2 anti-oxidative stress pathway


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Phosphorylation of the selective autophagy receptor TAX1BP1 by canonical and noncanonical IκB kinases promotes its lysosomal localization and clearance of MAVS aggregates


RalA and RalB relocalization to depolarized mitochondria depends on clathrin-mediated endocytosis and facilitates TBK1 activation

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Quantitative proteomics reveals the selectivity of ubiquitin-binding autophagy receptors in the turnover of damaged lysosomes by lysophagy


Quantitative proteomics reveals the selectivity of ubiquitin-binding autophagy receptors in the turnover of damaged lysosomes by lysophagy


Golgi apparatus-synthesized sulfated glycosaminoglycans mediate polymerization and activation of the cGAMP sensor STING.



STING-ing insights



Innate Immune Signaling Organelles Display Natural and Programmable Signaling Flexibility



TMEM203 is a binding partner and regulator of STING-mediated inflammatory signaling in macrophages



Mutations that TANK a kinase


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Kinase Tbk1 키나세 Tbk1
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