Intracortical Inhibition(피질내 억제)란 무엇입니까?
Intracortical Inhibition 피질내 억제 - Multivariate regression modeling analyses were used to investigate the association of intracortical excitability, measured by percentages of intracortical inhibition (ICI) and facilitation (ICF) with clinical variables. [1] 686), or intracortical inhibition (all P > 0. [2] Thresholds for intracortical inhibition in the TIA-unaffected hemisphere were significantly associated with the clinical diagnosis of TIA. [3] Corticospinal excitability, cortical representation area, and intracortical inhibition and facilitation were assessed by nTMS for a small hand muscle (first dorsal interosseous) before and after revascularization. [4] Specifically, we did not observe any change in spinal motoneuron responsiveness from cervicomedullary stimulations but a specific increase in intracortical inhibition during lengthening vs. [5] RESULTS Both clinical (stroke side (left) and age >14 years) and neurophysiological (intracortical inhibition/facilitation and motor threshold) were associated with responsiveness across treatment groups with positive predictive values (PPV) approaching 80%. [6] With regard to neurophysiology, a decrease in intracortical inhibition for the sensorimotor cortex contralateral to the affected hand has been repetitively verified, which might be related to increased primary somatosensory cortex functional activation for the affected limb. [7] TMS showed significant differences between the “symptomatic” and control groups for intracortical inhibition and paired pulse TMS measures. [8] It is concluded that a model based on intracortical inhibition can account well for the known properties of direction selectivity in carnivores and primates. [9] One of the main hypotheses in this cohort is that the level of intracortical inhibition is related to functional deficits. [10] OBJECTIVE The aim of the present study was to explore the impact of acute and chronic nicotine consumption on measures of intracortical inhibition and facilitation. [11] Intrinsic hand muscles of the dominant UE received significantly more intracortical inhibition (SICI) when the shoulder was in ADD compared to ABD; there was no position-dependent modulation of SICI on the non-dominant side. [12] Paired-pulse protocols including intracortical inhibition and intracortical facilitation (ICI and ICF) are well-established. [13]다변량 회귀 모델링 분석을 사용하여 피질내 억제(ICI) 및 촉진(ICF)의 백분율로 측정한 피질내 흥분성과 임상 변수의 연관성을 조사했습니다. [1] 686), 또는 피질내 억제(모두 P > 0. [2] TIA에 영향을 받지 않는 반구에서 피질내 억제에 대한 역치는 TIA의 임상 진단과 유의하게 연관되었습니다. [3] 혈관 재생 전후에 작은 손 근육(첫 번째 등쪽 골간)에 대해 피질척수 흥분성, 피질 대표 영역, 피질내 억제 및 촉진을 nTMS로 평가했습니다. [4] 특히, 우리는 경추수질 자극으로부터 척추 운동뉴런 반응성의 어떠한 변화도 관찰하지 못하였지만, 연장 동안 대뇌피질내 억제의 특정한 증가를 관찰하였다. [5] 결과 임상적(뇌졸중(왼쪽) 및 14세 이상) 및 신경생리학적(피질내 억제/촉진 및 운동 역치)은 양성 예측값(PPV)이 80%에 육박하는 치료군 전반에 걸친 반응성과 관련이 있었습니다. [6] 신경생리학과 관련하여, 영향을 받은 손의 반대쪽 감각운동 피질에 대한 피질내 억제의 감소가 반복적으로 확인되었으며, 이는 영향을 받는 사지의 증가된 일차 체감각 피질 기능 활성화와 관련될 수 있습니다. [7] TMS는 피질내 억제 및 쌍을 이루는 펄스 TMS 측정에 대해 "증상이 있는" 그룹과 대조군 사이에 상당한 차이를 보여주었습니다. [8] 피질내 억제에 기반한 모델은 육식동물과 영장류에서 알려진 방향 선택성의 특성을 잘 설명할 수 있다고 결론지었습니다. [9] 이 코호트의 주요 가설 중 하나는 피질내 억제 수준이 기능적 결함과 관련이 있다는 것입니다. [10] 목적 현재 연구의 목적은 급성 및 만성 니코틴 소비가 피질내 억제 및 촉진 측정에 미치는 영향을 조사하는 것이었습니다. [11] 우세한 UE의 내재적 손 근육은 ABD에 비해 어깨가 ADD에 있을 때 훨씬 더 많은 피질내 억제(SICI)를 받았습니다. 비지배적 측면에서 SICI의 위치 의존적 변조는 없었다. [12] 피질내 억제 및 피질내 촉진(ICI 및 ICF)을 포함한 짝 펄스 프로토콜은 잘 확립되어 있습니다. [13]
transcranial magnetic stimulation 경두개 자기 자극
We used transcranial magnetic stimulation to measure cerebellar-motor cortex (M1) inhibition (CBI), short-intracortical inhibition (SICI) and short-afferent inhibition (SAI) before, during and after the application of tACS. [1] Transcranial magnetic stimulation (TMS) was used to assess neurophysiological differences between groups including: short- and long-interval intracortical inhibition, intracortical facilitation, short- and long-latency afferent inhibition, afferent facilitation, and transcallosal inhibition (TCI). [2] Two novel short-interval intracortical inhibition (SICI) protocols, assessing SICI across a range of interstimulus intervals (ISIs) using either parallel threshold-tracking transcranial magnetic stimulation (TT-TMS) or automated conventional TMS (cTMS), were recently introduced. [3] Transcranial magnetic stimulation was used to test the input/output curve of motor evoked potentials, intracortical inhibition, and short-latency afferent inhibition. [4] Transcranial magnetic stimulation (TMS) was used to probe rest and active short-latency intracortical inhibition (SICI), interhemispheric inhibition (IHI) and response to paired associative stimulation (PAS). [5] OBJECTIVES The transcranial magnetic stimulation (TMS) technique of threshold-tracking short-interval intracortical inhibition (T-SICI) has been proposed as a diagnostic tool for amyotrophic lateral sclerosis (ALS). [6] Transcranial magnetic stimulation (TMS) is a promising, emerging tool for the study (study and modulate excitability and plasticity, applied in single pulses to investigate corticospinal excitability, pairs of pulses to study intracortical inhibition and facilitation) and potential treatment of ASD. [7] At the neural level, Transcranial Magnetic Stimulation (TMS) allows to investigate motor inhibition within the primary motor cortex (M1), such as the cortical silent period (CSP) which is an index of GABAB-mediated intracortical inhibition within M1. [8] STUDY OBJECTIVES Previous studies found an early impairment of the short-interval intracortical inhibition (SICI) and intracortical facilitation (ICF) to transcranial magnetic stimulation (TMS) in Parkinson's disease. [9] By using transcranial magnetic stimulation, we assessed the excitability of the primary motor cortex (M1) by measuring short-interval intracortical inhibition (SICI), long intracortical inhibition (LICI) and intracortical facilitation (ICF), sensorimotor interaction using short-latency afferent inhibition, and cerebellar-brain inhibition to test functional connectivity between the cerebellum and contralateral M1. [10] However, intracortical inhibition (lateral and recurrent) appeared to be reduced after repetitive transcranial magnetic stimulation, probably as the cause of improved responsiveness. [11] BACKGROUND Inhibitory deficits in motor cortex in schizophrenia have been well demonstrated using short-interval intracortical inhibition (SICI) by transcranial magnetic stimulation. [12] BACKGROUND Decreased short-interval intracortical inhibition (SICI) to transcranial magnetic stimulation (TMS) of the primary motor cortex was described in subjects with restless legs syndrome/Willis-Ekbom disease (RLS/WED). [13]우리는 경두개 자기 자극을 사용하여 tACS 적용 전, 중 및 후에 소뇌-운동 피질(M1) 억제(CBI), 단피질내 억제(SICI) 및 단구심성 억제(SAI)를 측정했습니다. [1] 경두개 자기 자극(TMS)을 사용하여 그룹 간의 신경 생리학적 차이를 평가했습니다. [2] nan [3] nan [4] nan [5] nan [6] nan [7] 신경 수준에서 경두개 자기 자극(TMS)을 사용하면 M1 내에서 GABAB 매개 피질내 억제의 지표인 피질 침묵 기간(CSP)과 같은 1차 운동 피질(M1) 내의 운동 억제를 조사할 수 있습니다. [8] nan [9] nan [10] nan [11] nan [12] nan [13]
motor evoked potential 모터 유발 전위
Baseline motor evoked potential amplitude, motor cortex hemisphere, and motor threshold (MT) significantly predicted short-interval intracortical inhibition response. [1] We evaluated inhibition-facilitation configurations [interstimulus interval: 3 ms; short-latency intracortical inhibition (SICI) and 11 ms; intracortical facilitation (ICF)] with paired electrical stimulation protocols and the effect of TBS paradigm on continuous recording of motor-evoked potentials (MEPs) for quantitative parameters. [2] When the interstimulus interval (ISI) is 1-6 msec, the motor evoked potential (MEP) decreases in amplitude; this decrease is termed "short interval intracortical inhibition" (SICI); when the ISI is 7-30 msec, an increase in MEP amplitude occurs, termed "short interval intracortical facilitation" (SICF). [3] Functional evaluation included the box and blocks test (BBT) and hand grip maximum voluntary contraction (MVC), spinal and cortical motor evoked potential (sMEP and cMEP), and resting motor thresholds (RMT), short interval intracortical inhibition (SICI), and F wave in the abductor pollicis brevis muscle. [4] Motor evoked potentials (MEPs) and short-interval intracortical inhibition (SICI) were measured in the PA and AP current directions in M1 at two time points separated by 25 minutes of rest, as well as immediately and 30 minutes after a 25-minute bout of moderate-intensity cycling exercise. [5] Here, we triggered transcranial magnetic stimuli (TMS) on the up‐ or down‐going phase of a tACS‐imposed alpha oscillation and measured motor evoked potential (MEP) amplitude and short‐interval intracortical inhibition (SICI). [6] In separate experiments, the effects of rPMS on motor evoked potentials (MEPs), short-interval intracortical inhibition (SICI), intracortical facilitation (ICF), direct motor response (M-wave), Hoffmann-reflex, and ballistic wrist extension movements were assessed before and after rPMS. [7] The evaluations included box and blocks test (BBT), maximal voluntary contraction (MVC), F wave persistency, F/M ratio, spinal and cortical motor evoked potentials (MEP), recruitment curves of spinal MEP and cortical MEP and short-interval intracortical inhibition. [8] First, we assessed the effect of action observation on corticospinal excitability (amplitude of motor evoked potentials), short-interval intracortical inhibition, and transcallosal inhibition (ipsilateral silent period). [9] Fatiguing intermittent single-joint exercise causes an increase in corticospinal excitability and a decrease in intracortical inhibition when measured with peripherally recorded motor evoked potentials (MEPs) after transcranial magnetic stimulation (TMS). [10] Indeed, short‐interval intracortical inhibition (SICI) inhibits motor evoked potentials (MEPs), and motor learning has been associated with decreased SICI and increased cortical excitability. [11] Motor evoked potentials (MEPs), cortical silent period (CSP), short- and long-interval intracortical inhibition (SICI and LICI), and intracortical facilitation (ICF) were assessed with TMS monophasic posterior-anterior (monoPA; n = 9), monophasic anterior-posterior (monoAP; n = 7), or biphasic (biAP-PA; n = 7) pulses. [12] Amplitudes of motor evoked potentials (MEPs) and short-interval intracortical inhibition (SICI) evoked using transcranial magnetic stimulation (TMS) were assessed at baseline, after every 10 min of the task (a total of 30 min), and 30 min after the third and final trial. [13]기준선 모터는 잠재적 진폭, 운동 피질 반구 및 운동 역치(MT)를 유발하여 짧은 간격의 피질내 억제 반응을 유의하게 예측했습니다. [1] 우리는 억제 촉진 구성을 평가했습니다[자극간 간격: 3ms; 단시간 피질내 억제(SICI) 및 11ms; 피질내 촉진(ICF)] 쌍을 이루는 전기 자극 프로토콜과 TBS 패러다임이 정량적 매개변수에 대한 운동 유발 전위(MEP)의 연속 기록에 미치는 영향. [2] nan [3] nan [4] nan [5] nan [6] nan [7] nan [8] nan [9] nan [10] nan [11] nan [12] nan [13]
paired pulse transcranial 한 쌍의 맥박 경두개
Paired-pulse transcranial magnetic stimulation (ppTMS) was used to measure motor cortical plasticity via short intracortical inhibition (SICI) and intracortical facilitation (ICF). [1] Long-interval intracortical inhibition (LICI) is a paired-pulse transcranial magnetic stimulation (TMS) paradigm mediated in part by gamma-aminobutyric acid receptor B (GABAB) inhibition. [2] Besides stimulus intensities and interstimulus intervals (ISI), the electric field (E-field) orientation is known to affect both short-interval intracortical inhibition (SICI) and facilitation (SICF) in paired-pulse transcranial magnetic stimulation (TMS). [3] Short-interval intracortical inhibition (SICI) was assessed using paired-pulse transcranial magnetic stimulation during a sustained contraction at 10% of plantar flexor isometric peak torque. [4] OBJECTIVE The study aimed to investigate short-interval intracortical inhibition (SICI) in burns survivors and non-injured controls, and establish whether paired-pulse transcranial magnetic stimulation (TMS) is a sensitive tool to investigate SICI after burn-injury. [5] Paired-pulse transcranial magnetic stimulation was applied to left primary motor cortex to assess the modulation of GABAA-mediated short-interval intracortical inhibition (SICI) during stopping and GABAB-mediated long-interval intracortical inhibition (LICI) during the anticipation of a stop-signal. [6]쌍-펄스 경두개 자기 자극(ppTMS)은 짧은 피질내 억제(SICI) 및 피질내 촉진(ICF)을 통해 운동 피질 가소성을 측정하는 데 사용되었습니다. [1] 장간격 피질내 억제(LICI)는 부분적으로 감마-아미노부티르산 수용체 B(GABAB) 억제에 의해 매개되는 쌍-펄스 경두개 자기 자극(TMS) 패러다임입니다. [2] nan [3] nan [4] nan [5] nan [6]
cortical silent period 피질 침묵 기간
Two studies assessed other cortical excitability measures, such as cortical silent period (CSP), short-interval intracortical inhibition (SICI), and intracortical facilitation (ICF). [1] Previous studies of the duration of the cortical silent period (CSP)—a measure of intracortical inhibition—in migraine patients have yielded conflicting results. [2] Resting motor threshold, cortical silent period, and long-interval intracortical inhibition were measured in both groups. [3] We examined TMS-evoked short- (SICI) and long-interval intracortical inhibition (LICI) and cortical silent period (CSP) as markers of GABAA- (SICI) and GABAB-mediated (LICI and CSP) cortical neurotransmission in symptomatic individuals with mania (n = 40), schizophrenia (n = 76), unipolar depression (n = 86), and OCD (n = 43), and compared them against similar recordings in healthy subjects (n = 125). [4] We provide a brief introduction to the TMS technique; common TMS protocols including single-pulse TMS, paired-pulse TMS, paired associative stimulation, and repetitive TMS; and relevant TMS-derived neurophysiological measurements including resting and active motor threshold, cortical silent period, paired-pulse TMS measures of intracortical inhibition and facilitation, and plasticity metrics after repetitive TMS. [5] The motor threshold (MT), cortical silent period (CSP), short interval intracortical inhibition (SICI), intracortical facilitation (ICF), and long interval intracortical inhibition (LICI) are among TMS-derived metrics that are modulated by antiepileptic drugs. [6]2건의 연구에서는 피질 침묵 기간(CSP), 단시간 피질내 억제(SICI) 및 피질내 촉진(ICF)과 같은 다른 피질 흥분성 측정을 평가했습니다. [1] 편두통 환자에서 피질 내 억제의 척도인 피질 침묵 기간(CSP)의 지속 시간에 대한 이전 연구에서는 상반된 결과가 나왔습니다. [2] nan [3] nan [4] nan [5] nan [6]
short interval intracortical 짧은 간격의 피질내
, short- and long-interval intracortical inhibition (SICI/LICI), short-interval intracortical facilitation (SICF), and short- and long-latency afferent inhibition (SAI/LAI)) provide information about intracortical inhibitory and facilitatory networks. [1] Cortical hyperexcitability was defined by reduced short interval intracortical inhibition (SICI) and increased short interval intracortical facilitation (SICF), index of excitability (IE), and motor evoked potential (MEP) amplitude. [2] We then tested the effects of RAG and LAG stimulation on LM1 short-interval intracortical facilitation(SICF), short-interval intracortical inhibition(SICI) and long-interval intracortical inhibition(LICI). [3] OBJECTIVE Motor cortical (M1) inhibition and facilitation can be studied with short-interval intracortical inhibition (SICI) and short-interval intracortical facilitation (SICF). [4] the characteristic long-interval intracortical inhibition (LICI) and the I1-wave timed short-interval intracortical facilitation (SICF). [5] GABA-ergic (short- and long-interval intracortical inhibition (SICI and LICI)) and glutamatergic (intracortical and short-interval intracortical facilitation (ICF and SICF)) excitability of the primary motor cortex (M1) and global corticospinal excitability (motor threshold, motor evoked potential recruitment curve (MEP-RC) were compared between the groups. [6], 단기 및 장기 피질내 억제(SICI/LICI), 단시간 피질내 촉진(SICF), 단기 및 장기 구심성 억제(SAI/LAI))는 피질내 억제 및 촉진 네트워크에 대한 정보를 제공합니다. [1] 피질의 과흥분성은 짧은 간격의 피질내 억제(SICI) 감소 및 짧은 간격의 피질내 촉진(SICF) 증가, 흥분 지수(IE) 및 운동 유발 전위(MEP) 진폭으로 정의되었습니다. [2] nan [3] nan [4] nan [5] nan [6]
short latency afferent 짧은 대기 시간 구심성
Paired-pulse TMS paradigms include short- and long-interval intracortical inhibition (SICI/LICI), intracortical facilitation (ICF), and short-latency afferent inhibition (SAI), which can assess neurophysiological functions of GABAergic, glutamatergic, and cholinergic neural circuits, respectively. [1] We assessed resting and active motor threshold, short-latency intracortical inhibition (SICI), intracortical facilitation (ICF), short-latency afferent inhibition (SAI), long-latency afferent inhibition, cortical silent period, and interhemispheric inhibition. [2] Intracortical activity was evaluated by means of well-established TMS protocols including short-interval intracortical inhibition (SICI), reflecting GABAA-mediated inhibition, long-interval intracortical inhibition (LICI), a marker of GABAB receptor activity, and short-latency afferent inhibition (SAI) that indexes central cholinergic transmission. [3] Short interval intracortical inhibition (SICI), intracortical facilitation (ICF), long interval intracortical inhibition (LICI), and measures of sensorimotor interaction (short-latency afferent [SAI] and long-latency afferent [LAI] stimulation) were assessed in both hemispheres using pp-TMS paradigms at each time point. [4] Short- and long-interval intracortical inhibition, short-latency afferent inhibition (SAI), and long-latency afferent inhibition were measured using transcranial magnetic stimulation (TMS) as indices of GABAergic activity. [5] We applied short interval intracortical inhibition (SICI - GABAAergic transmission), intracortical facilitation (ICF - glutamatergic transmission), long interval intracortical inhibition (LICI - GABABergic transmission), and short latency afferent inhibition (SAI - cholinergic transmission). [6]Paired-pulse TMS 패러다임에는 GABA성, 글루타메이트성 및 콜린성 신경 회로의 신경 생리학적 기능을 평가할 수 있는 단거리 및 장간격 피질내 억제(SICI/LICI), 피질내 촉진(ICF) 및 단기 구심성 억제(SAI)가 포함됩니다. , 각각. [1] 우리는 휴식 및 활성 운동 역치, 짧은 잠복 피질내 억제(SICI), 피질내 촉진(ICF), 짧은 잠복 구심성 억제(SAI), 긴 잠복 구심성 억제, 피질 침묵 기간 및 반구간 억제를 평가했습니다. [2] nan [3] nan [4] nan [5] nan [6]
increased intracortical facilitation 증가된 피질내 촉진
We systematically investigated the relationship between resting-state intracortical inhibition or facilitation and inhibitory control (indicated by stop signal reaction time, SSRT) to determine whether reduced intracortical inhibition or increased intracortical facilitation was related to the poorer inhibitory control. [1] The unaffected hemisphere showed decreased intracortical inhibition in the combined and fluoxetine groups, and increased intracortical facilitation in the fluoxetine group. [2] We identified studies which report a reduced intracortical inhibition and increased intracortical facilitation in the hemisphere contralateral to the PLP. [3]우리는 피질내 억제 감소 또는 피질내 촉진 증가가 더 나쁜 억제 제어와 관련이 있는지 여부를 결정하기 위해 휴식 상태 피질내 억제 또는 촉진과 억제 제어(정지 신호 반응 시간, SSRT로 표시) 사이의 관계를 체계적으로 조사했습니다. [1] nan [2] nan [3]
input output curve 입력 출력 곡선
The model captured TMS phenomena including a sigmoidal input-output curve, common paired pulse effects (short interval intracortical inhibition, intracortical facilitation, long interval intracortical inhibition) including responses to pharmacological interventions, and a cortical silent period. [1] The model captured TMS phenomena including a sigmoidal input-output curve, common paired pulse effects (short interval intracortical inhibition, intracortical facilitation, long interval intracortical inhibition) including responses to pharmacological interventions, and a cortical silent period. [2]이 모델은 S자형 입출력 곡선, 일반적인 쌍을 이루는 펄스 효과(짧은 간격의 피질내 억제, 피질내 촉진, 긴 간격의 피질내 억제)를 포함하는 TMS 현상, 약리학적 개입에 대한 반응, 피질 침묵 기간을 포착했습니다. [1] nan [2]
primary motor cortex 일차운동피질
Forty-eight healthy older adults received, over the primary motor cortex (M1), tDCS – anodal and sham at least 1 week apart – before, during or after an explicit sequence-learning task with electrophysiological measures of corticospinal excitability (CSE) and short-interval intracortical inhibition (SICI) also obtained. [1]48명의 건강한 노인이 일차 운동 피질(M1)을 통해 tDCS(적어도 1주일 간격으로 양극 및 가짜)를 받았습니다. 피질척수 흥분성(CSE) 및 짧은 -간격 피질내 억제(SICI)도 얻었습니다. [1]