Intracortical Facilitation(피질내 촉진)란 무엇입니까?
Intracortical Facilitation 피질내 촉진 - Corticomotor excitability, intracortical inhibition, and intracortical facilitation were not modulated by verum or sham acupuncture during, immediately after, or 30 min after, recorded from a local or distant hand muscle to the needling site (all P>0. [1] Results: Both TMS-induced long-term potentiation (LTP), intracortical facilitation (ICF) and short-interval ICF increased after 2 and 4 weeks of D-Aspartate but not after placebo, suggesting an enhancement of synaptic plasticity reserve and increased trans-synaptic glutamatergic transmission. [2] Conclusions We found different cortical excitability pattern in arm and face transplantation patients; there is an intracortical inhibition in arm transplants but a reduction of intracortical inhibition and intracortical facilitation. [3] Intracortical inhibition was reduced and intracortical facilitation was increased for the agonist muscle only. [4] What is the central question of this study? How does single limb cold water immersion affect corticomotor function and intracortical circuitry in the motor cortex of each cerebral hemisphere? What is the main finding and its importance? Immersion of a single limb in very cold water caused an increase in corticomotor excitability and intracortical facilitation, and a decrease in intracortical inhibition, in the motor cortex of both hemispheres. [5] In this study, we investigated changes in intracortical inhibition, intracortical facilitation, and corticospinal excitability after 10 sessions of cathodal transcutaneous delivery of pulse or direct current stimulation, termed here transspinal (tsPCS, tsDCS), in resting healthy humans. [6] When participants were divided into groups according to writing style (printers, n = 8; cursive writers, n = 8), a task x fatigue x style interaction was revealed for intracortical facilitation (F (1,14) = 9. [7] HighlightsSchizophrenia subjects received tDCS protocol for symptomatic treatmentMotor cortical excitability was measured with TMS‐EMG before and after tDCSShort latency intracortical inhibition increased after a single tDCS sessionNo significant change in intracortical facilitation was observed after tDCS. [8] Significant improvements in response times during multitasking performance and increases in intracortical facilitation (ICF) were seen following light aerobic exercise. [9] Methods In a group of 29 PD patients (Hoehn & Yahr stage range 1–4), measures of M1 excitability (short latency intracortical inhibition [SICI] and intracortical facilitation [ICF], and plasticity (paired associative stimulation [PAS] protocol) were collected. [10] Results showed that high-intensity interval exercise enhanced iTBS plasticity more than rest, evidenced by increased CME and intracortical facilitation, and reduced intracortical inhibition. [11] SICI was markedly reduced at 1 and 3 ms and intracortical facilitation (ICF) was enhanced at 10 ms. [12] Therefore, we compared maximal ECC and CON contractions of the right biceps brachii (BB) muscle for changes in corticospinal excitability, short‐ (SICI) and long‐interval intracortical inhibition (LICI) and intracortical facilitation (ICF) up to 1 hour post‐exercise. [13]피질운동 흥분성, 피질내 억제, 피질내 촉진은 국부 또는 원거리 손 근육에서 자침 부위로 기록되는 동안, 직후 또는 30분 후 verum 또는 sham 침술에 의해 조절되지 않았습니다(모두 P>0. [1] 결과: TMS로 유발된 장기 강화(LTP), 피질내 촉진(ICF) 및 단기 ICF는 모두 D-Aspartate 투여 2주 및 4주 후 증가했지만 위약 투여 후에는 증가하지 않았으며, 이는 시냅스 가소성 예비의 향상과 트랜스포머의 증가를 시사합니다. 시냅스 글루타메이트 전달. [2] 결론 우리는 팔과 안면 이식 환자에서 서로 다른 피질 흥분 패턴을 발견했습니다. 팔 이식에서는 피질내 억제가 있지만 피질내 억제 및 피질내 촉진은 감소합니다. [3] 주동근에 대해서만 피질내 억제가 감소하고 피질내 촉진이 증가하였다. [4] 이 연구의 중심 질문은 무엇입니까? 외지 냉수 침수는 각 대뇌 반구의 운동 피질에서 피질 운동 기능과 피질 내 회로에 어떤 영향을 미칩니 까? 주요 발견과 그 중요성은 무엇입니까? 매우 차가운 물에 한쪽 다리를 담그면 양쪽 반구의 운동 피질에서 피질 운동 흥분성과 피질 내 촉진이 증가하고 피질 내 억제가 감소합니다. [5] 이 연구에서 우리는 건강한 사람을 쉬고 있는 상태에서 여기에서 경척수(tsPCS, tsDCS)라고 하는 펄스 또는 직류 자극의 음극 경피 전달의 10 세션 후 피질내 억제, 피질내 촉진 및 피질척수 흥분성의 변화를 조사했습니다. [6] 참가자를 작문 스타일(프린터, n = 8, 필기체 작가, n = 8)에 따라 그룹으로 나누었을 때, 피질내 촉진(F(1,14) = 9)에 대한 작업 x 피로 x 스타일 상호 작용이 나타났습니다. [7] 하이라이트 정신 분열증 환자는 대증 치료를 위해 tDCS 프로토콜을 받았습니다. tDCS 전후에 TMS-EMG로 운동 피질 흥분성을 측정했습니다.단일 tDCS 세션 후 짧은 잠복기 피질내 억제가 증가했습니다.tDCS 후에 피질내 촉진의 유의한 변화는 관찰되지 않았습니다. [8] 가벼운 유산소 운동 후 멀티태스킹 수행 중 응답 시간이 크게 개선되고 피질내 촉진(ICF)이 증가하는 것으로 나타났습니다. [9] 방법 29명의 PD 환자 그룹(Hoehn & Yahr 단계 범위 1-4)에서 M1 흥분성(짧은 잠복 피질내 억제[SICI] 및 피질내 촉진[ICF] 및 가소성(짝지어 연합 자극[PAS] 프로토콜) 모은. [10] 결과는 고강도 인터벌 운동이 휴식보다 iTBS 가소성을 향상시켰으며, 이는 CME 및 피질내 촉진 증가 및 피질내 억제 감소로 입증되었습니다. [11] SICI는 1ms와 3ms에서 현저하게 감소했고 피질내 촉진(ICF)은 10ms에서 향상되었습니다. [12] 따라서 우리는 오른쪽 상완이두근(BB) 근육의 최대 ECC 및 CON 수축을 1시간 후까지 피질척수 흥분성, 단거리(SICI) 및 장간격 피질내 억제(LICI) 및 피질내 촉진(ICF)의 변화에 대해 비교했습니다. 연습. [13]
transcranial magnetic stimulation 경두개 자기 자극
By using transcranial magnetic stimulation, we assessed the excitability of the primary motor cortex (M1) by measuring short-interval intracortical inhibition (SICI), long intracortical inhibition (LICI) and intracortical facilitation (ICF), sensorimotor interaction using short-latency afferent inhibition, and cerebellar-brain inhibition to test functional connectivity between the cerebellum and contralateral M1. [1] BACKGROUND Recent work suggests that the function of intracortical interneurons activated by transcranial magnetic stimulation (TMS) is modified in older adults, with the circuits generating short-interval intracortical facilitation (SICF) at longer intervals appearing to be particularly affected. [2] Thus, the purpose of this cross-sectional exploratory study was: (i) to compare the motor cortex inhibition indexed by transcranial magnetic stimulation (TMS) measures [short intracortical inhibition (SICI) and intracortical facilitation (ICF)], as well as the function of descending pain modulatory systems (DPMS) among FM, MDD, and healthy subjects (HS); (ii) to compare SICI, ICF, and the role of DPMS evaluated by the change on Numerical Pain Scale (NPS) during the conditioned pain modulation test (CPM-test) between FM and MDD considering the BDNF-adjusted index; (iii) to assess the relationship between the role of DPMS and the BDNF-adjusted index, despite clinical diagnosis. [3] Methods- Fourteen patients with CPSP but no motor weakness were compared with age- and sex-matched healthy controls for motor cortex excitability and sensorimotor interaction assessed by transcranial magnetic stimulation to measure resting motor thresholds, short-interval intracortical inhibition, intracortical facilitation, and afferent inhibitions. [4] Methods Two sisters affected by NPC and cognitive decline underwent neuropsychological tests, PET scans with 18F- Florbetaben and neurophysiological protocols to assess cortex excitability by means of transcranial magnetic stimulation (TMS), such as short-latency afferent inhibition (SAI), short-interval intracortical inhibition (SICI) and intracortical facilitation (ICF). [5] Methods Eighty-four (56 IULF patients <14 days post-trauma and 28 healthy controls) performed a single transcranial magnetic stimulation (TMS) session over M1 (resting motor threshold (rMT); short-intracortical inhibition (SICI); intracortical facilitation (ICF); long-interval cortical inhibition (LICI)). [6] Transcranial magnetic stimulation measures included short-interval intracortical inhibition (2 and 4 milliseconds), long-interval intracortical inhibition (100, 150, and 200 milliseconds), cortical silent period, and intracortical facilitation (10, 15, and 20 milliseconds). [7]경두개 자기 자극을 사용하여 단기 피질내 억제(SICI), 장기 피질내 억제(LICI) 및 피질내 촉진(ICF), 단기 구심성 억제를 이용한 감각운동 상호작용을 측정하여 1차 운동 피질(M1)의 흥분성을 평가했습니다. 소뇌와 반대쪽 M1 사이의 기능적 연결성을 테스트하기 위한 소뇌-뇌 억제. [1] 배경 최근 연구에 따르면 경두개 자기 자극(TMS)에 의해 활성화된 피질 내 개재 뉴런의 기능은 특히 영향을 받는 것으로 보이는 더 긴 간격으로 단시간 피질내 촉진(SICF)을 생성하는 회로가 노인에서 수정됩니다. [2] nan [3] nan [4] nan [5] nan [6] nan [7]
short interval intracortical 짧은 간격의 피질내
STUDY OBJECTIVES Previous studies found an early impairment of the short-interval intracortical inhibition (SICI) and intracortical facilitation (ICF) to transcranial magnetic stimulation (TMS) in Parkinson's disease. [1] The model captured TMS phenomena including a sigmoidal input-output curve, common paired pulse effects (short interval intracortical inhibition, intracortical facilitation, long interval intracortical inhibition) including responses to pharmacological interventions, and a cortical silent period. [2] We applied short interval intracortical inhibition (SICI - GABAAergic transmission), intracortical facilitation (ICF - glutamatergic transmission), long interval intracortical inhibition (LICI - GABABergic transmission), and short latency afferent inhibition (SAI - cholinergic transmission). [3] Here, paired-pulse TMS was used to examine the balance between short-interval intracortical inhibition (SICI) and short-interval intracortical facilitation (SICF) in younger (n=26) and older (n=21) adults. [4] Specifically, we evaluated the motor thresholds that reflect membrane excitability related to the voltage-gated sodium channels in the neurons of the motor system and the ease of activation of motor cortex via glutamatergic networks, and ad hoc TMS protocols to probe inhibitory-GABAergic (short interval intracortical inhibition, SICI; short-latency afferent inhibition, SAI; cortical silent period, CSP) and excitatory intracortical circuitry (intracortical facilitation, ICF). [5]연구 목표 이전 연구는 파킨슨병에서 경두개 자기 자극(TMS)에 대한 단기 피질내 억제(SICI) 및 피질내 촉진(ICF)의 조기 손상을 발견했습니다. [1] 이 모델은 S자형 입출력 곡선, 일반적인 쌍을 이루는 펄스 효과(짧은 간격의 피질내 억제, 피질내 촉진, 긴 간격의 피질내 억제)를 포함하는 TMS 현상, 약리학적 개입에 대한 반응, 피질 침묵 기간을 포착했습니다. [2] nan [3] nan [4] nan [5]
motor evoked potential 모터 유발 전위
Motor evoked potentials (MEPs), cortical silent period (CSP), short- and long-interval intracortical inhibition (SICI and LICI), and intracortical facilitation (ICF) were assessed with TMS monophasic posterior-anterior (monoPA; n = 9), monophasic anterior-posterior (monoAP; n = 7), or biphasic (biAP-PA; n = 7) pulses. [1] M1 excitability was assessed by motor-evoked potential (MEP) recruitment curve with five TMS intensity levels, short-interval intracortical inhibition (SICI), intracortical facilitation (ICF) and cerebellar inhibition (CBI) at the abductor pollicis brevis (APB) muscle of the right hand before and after the EA-M1 paired associative stimulation (PAS). [2] Before and after each session separated at least by 1 week, motor evoked potential (MEP) amplitude, short‐interval intracortical inhibition (SICI), contralateral silent period (SP), and intracortical facilitation (ICF) generated by TMS were measured in the trained and the untrained BBs. [3] We assessed motor evoked potential (MEP) recruitment curves for the first dorsal interosseous (FDI) both at rest and during tonic contraction, intracortical facilitation (ICF), and short‐interval intracortical inhibition (SICI) using transcranial magnetic stimulation (TMS). [4] METHODS Motor evoked potentials (MEPs) elicited by single-pulse TMS, short-interval intracortical inhibition (SICI) and intracortical facilitation (ICF) were evaluated before and 5, 15 and 30 min after cTBS alone or cTBS delivered during β-tACS (cTBS-β) or γ-tACS (cTBS-γ). [5]운동 유발 전위(MEP), 피질 무성 기간(CSP), 장단기 피질내 억제(SICI 및 LICI), 피질내 촉진(ICF)은 TMS 단상 후방-전방(monoPA, n=9)으로 평가되었습니다. monophasic anterior-posterior (monoAP; n=7) 또는 biphasic (biAP-PA; n=7) 펄스. [1] M1 흥분성은 5개의 TMS 강도 수준, 짧은 간격의 피질내 억제(SICI), 피질내 촉진(ICF) 및 소뇌 억제(CBI)의 5개의 TMS 강도 수준을 갖는 운동 유발 전위(MEP) 모집 곡선에 의해 평가되었습니다. EA-M1 짝을 이루는 연관 자극(PAS) 전후의 오른손. [2] nan [3] nan [4] nan [5]
pulse transcranial magnetic 펄스 경두개 자기
Paired-pulse transcranial magnetic stimulation (TMS) measures, including intracortical facilitation (ICF), short interval cortical inhibition (SICI), and cortical silent period (CSP) were measured in an age matched cohort of youth with ASD- (n = 20), ASD + (n = 29), and controls (TDC) (n = 24). [1] Pre- and post-exercise, single- and paired-pulse transcranial magnetic stimulation (TMS) tested motor evoked potentials (MEP), intracortical facilitation (ICF [12 ms]) and short-interval intracortical inhibition (SICI2 [2ms], SICI3 [3ms]). [2] Single and paired pulse transcranial magnetic stimulation studies showed altered excitability and cortical circuits in PD with decreased silent period, short interval intracortical inhibition, intracortical facilitation, long afferent inhibition, interhemispheric inhibition, and cerebellar inhibition, and increased long interval intracortical inhibition and short interval intracortical facilitation. [3] METHODS Paired-pulse transcranial magnetic stimulation (ppTMS) was used to measure cortical inhibitory function including GABAA- and GABAB-receptor-related CI and cortical excitatory function including glutamate-receptor-related intracortical facilitation (ICF). [4]ASD-(n = 20)를 가진 청소년의 연령 일치 코호트에서 피질내 촉진(ICF), 짧은 간격의 피질 억제(SICI) 및 피질 침묵 기간(CSP)을 포함한 쌍-펄스 경두개 자기 자극(TMS) 측정이 측정되었습니다. , ASD + (n = 29) 및 대조군(TDC)(n = 24). [1] 운동 전후, 단일 및 쌍 펄스 경두개 자기 자극(TMS)은 운동 유발 전위(MEP), 피질 내 촉진(ICF [12ms]) 및 단시간 피질 내 억제(SICI2 [2ms], SICI3 [ 3ms]). [2] nan [3] nan [4]
short intracortical inhibition 짧은 피질내 억제
Paired-pulse stimulation was used to assess short intracortical inhibition and intracortical facilitation. [1] Objective: We assessed and compared short intracortical inhibition (SICI) and intracortical facilitation (ICF) testing four different experimental conditions. [2]짝 펄스 자극은 짧은 피질내 억제 및 피질내 촉진을 평가하는 데 사용되었습니다. [1] 목적: 우리는 4가지 다른 실험 조건을 테스트하는 짧은 피질내 억제(SICI)와 피질내 촉진(ICF)을 평가하고 비교했습니다. [2]
cortical silent period 피질 침묵 기간
The motor threshold (MT), cortical silent period (CSP), short interval intracortical inhibition (SICI), intracortical facilitation (ICF), and long interval intracortical inhibition (LICI) are among TMS-derived metrics that are modulated by antiepileptic drugs. [1] Results Compared to controls, patients with OCD showed a shorter cortical silent period and decreased intracortical facilitation. [2]운동 역치(MT), 피질 침묵 기간(CSP), 짧은 간격의 피질내 억제(SICI), 피질내 촉진(ICF), 및 긴 간격의 피질내 억제(LICI)는 항간질 약물에 의해 조절되는 TMS 유래 메트릭 중 하나입니다. [1] 결과 대조군에 비해 강박장애 환자는 피질 침묵 기간이 더 짧고 피질 내 촉진이 감소했습니다. [2]
long interval intracortical 긴 간격의 피질내
the characteristic long-interval intracortical inhibition (LICI) and the I1-wave timed short-interval intracortical facilitation (SICF). [1] GABA-ergic (short- and long-interval intracortical inhibition (SICI and LICI)) and glutamatergic (intracortical and short-interval intracortical facilitation (ICF and SICF)) excitability of the primary motor cortex (M1) and global corticospinal excitability (motor threshold, motor evoked potential recruitment curve (MEP-RC) were compared between the groups. [2]특징적인 장간격 피질내 억제(LICI) 및 I1파 시간 단시간 피질내 촉진(SICF). [1] 일차 운동 피질(M1) 및 전체 피질척수 흥분성(운동 역치)의 GABA 흥분성(단기 및 장간 피질내 억제(SICI 및 LICI)) 및 글루타메이트성(피질내 및 단간격 피질내 촉진(ICF 및 SICF)) 흥분성 , 운동 유발 전위 모집 곡선(MEP-RC)을 그룹 간에 비교했습니다. [2]
resting motor threshold 휴식 모터 역치
METHODS The automatic threshold tracking method was employed to assess the resting motor threshold (RMT), intracortical facilitation (ICF), short-interval intracortical inhibition (SICI) and short-interval intracortical facilitation (SICF) in patients and controls. [1] Net intracortical facilitation involving glutamate was assessed with intracortical facilitation, and corticospinal excitability was measured with the resting motor threshold. [2]행동 양식 자동 역치 추적 방법을 사용하여 환자와 대조군의 휴식 운동 역치(RMT), 피질내 촉진(ICF), 단시간 피질내 억제(SICI) 및 단간 피질내 촉진(SICF)을 평가했습니다. [1] 글루타메이트를 포함하는 피질내 순 촉진은 피질내 촉진으로 평가되었고, 피질척수 흥분성은 휴식기 운동 역치로 측정되었습니다. [2]
short latency afferent 짧은 대기 시간 구심성
Dexterity of the non-dominant (left) hand was assessed using the grooved pegboard task, and measures of corticomotor excitability, intracortical facilitation, short-latency afferent inhibition (SAI), short-interval intracortical inhibition (SICI), and SAI in the presence of SICI (SAIxSICI), were obtained at baseline, post-tDCS, and 0, 30 and 60 min post-MGS. [1]비우세(왼쪽) 손의 손재주는 홈이 있는 페그보드 작업과 피질운동 흥분성, 피질내 촉진, 단기 구심성 억제(SAI), 단시간 피질내 억제(SICI) 및 존재하에서의 SAI 측정을 사용하여 평가되었습니다. SICI(SAIxSICI)는 기준선, tDCS 후 및 MGS 후 0, 30 및 60분에서 얻었습니다. [1]
Interval Intracortical Facilitation 간격 피질내 촉진
BACKGROUND Recent work suggests that the function of intracortical interneurons activated by transcranial magnetic stimulation (TMS) is modified in older adults, with the circuits generating short-interval intracortical facilitation (SICF) at longer intervals appearing to be particularly affected. [1] Intracortical facilitation (ICF) and short-interval intracortical facilitation (SICF) were measured to assess M1 excitatory circuits. [2] the characteristic long-interval intracortical inhibition (LICI) and the I1-wave timed short-interval intracortical facilitation (SICF). [3] GABA-ergic (short- and long-interval intracortical inhibition (SICI and LICI)) and glutamatergic (intracortical and short-interval intracortical facilitation (ICF and SICF)) excitability of the primary motor cortex (M1) and global corticospinal excitability (motor threshold, motor evoked potential recruitment curve (MEP-RC) were compared between the groups. [4] Here, paired-pulse TMS was used to examine the balance between short-interval intracortical inhibition (SICI) and short-interval intracortical facilitation (SICF) in younger (n=26) and older (n=21) adults. [5] METHODS The automatic threshold tracking method was employed to assess the resting motor threshold (RMT), intracortical facilitation (ICF), short-interval intracortical inhibition (SICI) and short-interval intracortical facilitation (SICF) in patients and controls. [6] Single and paired pulse transcranial magnetic stimulation studies showed altered excitability and cortical circuits in PD with decreased silent period, short interval intracortical inhibition, intracortical facilitation, long afferent inhibition, interhemispheric inhibition, and cerebellar inhibition, and increased long interval intracortical inhibition and short interval intracortical facilitation. [7]배경 최근 연구에 따르면 경두개 자기 자극(TMS)에 의해 활성화된 피질 내 개재 뉴런의 기능은 특히 영향을 받는 것으로 보이는 더 긴 간격으로 단시간 피질내 촉진(SICF)을 생성하는 회로가 노인에서 수정됩니다. [1] 피질내 촉진(ICF) 및 단시간 피질내 촉진(SICF)을 측정하여 M1 흥분성 회로를 평가하였다. [2] 특징적인 장간격 피질내 억제(LICI) 및 I1파 시간 단시간 피질내 촉진(SICF). [3] 일차 운동 피질(M1) 및 전체 피질척수 흥분성(운동 역치)의 GABA 흥분성(단기 및 장간 피질내 억제(SICI 및 LICI)) 및 글루타메이트성(피질내 및 단간격 피질내 촉진(ICF 및 SICF)) 흥분성 , 운동 유발 전위 모집 곡선(MEP-RC)을 그룹 간에 비교했습니다. [4] nan [5] 행동 양식 자동 역치 추적 방법을 사용하여 환자와 대조군의 휴식 운동 역치(RMT), 피질내 촉진(ICF), 단시간 피질내 억제(SICI) 및 단간 피질내 촉진(SICF)을 평가했습니다. [6] nan [7]
Increased Intracortical Facilitation 증가된 피질내 촉진
The unaffected hemisphere showed decreased intracortical inhibition in the combined and fluoxetine groups, and increased intracortical facilitation in the fluoxetine group. [1] We identified studies which report a reduced intracortical inhibition and increased intracortical facilitation in the hemisphere contralateral to the PLP. [2]영향을 받지 않은 반구는 결합된 그룹과 플루옥세틴 그룹에서 감소된 피질내 억제를 보였고, 플루옥세틴 그룹에서 증가된 피질내 촉진을 보였다. [1] 우리는 PLP 반대측 반구에서 감소된 피질내 억제 및 증가된 피질내 촉진을 보고하는 연구를 확인했습니다. [2]