Induces Testicular(고환을 유도)란 무엇입니까?
Induces Testicular 고환을 유도 - In conclusion, these findings strongly reveal that diabetes induces testicular damage, at least in part, by inducing mitochondrial-mediated apoptosis and oxidative stress. [1] Background: Methotrexate (MTX) is an anticancer drug that induces testicular abnormalities but, thyme is antioxidant compound, which scavenges oxidative damage. [2] Nonylphenol (NP) is an environmental contaminant, which induces testicular toxicity through oxidative stress. [3] Collectively, the current results indicate that PDB induces testicular toxicity and male reproductive abnormalities by inducing mitochondrial dysfunction, endoplasmic reticulum stress and calcium imbalance. [4] Exposure to dibutyl phthalate (DBP) induces testicular damage in mammals. [5] Our findings led us to conclude that stress like FR and estradiol induces testicular regression immature male chickens by modulating ir-ERα expression in the HPG axis thereby resulting in reduction in reproductive physiology. [6] Diabetes mellitus induces testicular damage, increases sperm abnormalities, and impairs reproductive dysfunction due to induction of endocrine disturbance and testicular oxidative stress. [7] In conclusion, our results suggest that exposure to Sulfx at the two selected doses induces testicular toxicity and these effects can be ameliorated by supplementation of RES. [8] Collectively, these findings demonstrate that HgCl2 exposure disrupts the reproductive system and induces testicular immunosuppression and fibrosis via inhibition of the CD74 signaling pathway in male mice. [9] Collectively, we demonstrated that flufenoxuron inhibits cell proliferation and alters gene expression in mouse testis cells and induces testicular dysfunction in mice. [10] Studies have reported that HAART induces testicular toxicity and impairs sperm quality. [11] ResultsResults showed that KBrO3 administration induces testicular damage reflected in a significant decrease in total protein, glycogen, sialic acid with a significant increase in MDA level in testicular tissue. [12] We have recently demonstrated that WPS exposure in mice induces testicular toxicity including inflammation and oxidative stress. [13] However, the mechanisms by which Pb induces testicular injury remain unclear. [14] To begin, SRY production from the male gonads induces testicular development and Mullerian regression. [15] Diazinon (DZN), as an organophosphate insecticide, induces testicular toxicity through oxidative stress. [16] Zinc (Zn) plays an important role in spermatogenesis, and carbon tetrachloride (CCl 4 ) induces testicular oxidative damage and cell death. [17] CONCLUSION The present study demonstrates that cadmium induces testicular dysfunction that is attributable to defective G6PD and accompanied by increased lipid peroxidation and impaired NO-dependent endothelial function. [18] The findings of this study show that menadione induces testicular toxicity by depleting the antioxidant defense system leading to perturbation in the testicular function indices. [19]nan [1] nan [2] nan [3] nan [4] nan [5] nan [6] nan [7] nan [8] nan [9] nan [10] nan [11] 결과결과는 KBrO3 투여가 고환 조직에서 MDA 수준의 유의한 증가와 함께 총 단백질, 글리코겐, 시알산의 유의한 감소에 반영된 고환 손상을 유도하는 것으로 나타났습니다. [12] 우리는 최근에 마우스에서 WPS 노출이 염증 및 산화 스트레스를 포함한 고환 독성을 유도한다는 것을 입증했습니다. [13] 그러나 Pb가 고환 손상을 유발하는 메커니즘은 아직 명확하지 않습니다. [14] 시작하려면 남성 생식선의 SRY 생산이 고환 발달과 뮐러 회귀를 유도합니다. [15] 디아지논(DZN)은 유기인계 살충제로서 산화 스트레스를 통해 고환 독성을 유발합니다. [16] 아연(Zn)은 정자 형성에 중요한 역할을 하며 사염화탄소(CCl 4 )는 고환의 산화적 손상과 세포 사멸을 유도합니다. [17] 결론 현재의 연구는 카드뮴이 결함이 있는 G6PD에 기인하고 지질 과산화 증가 및 NO 의존성 내피 기능 손상을 동반하는 고환 기능 장애를 유도한다는 것을 보여줍니다. [18] 이 연구의 결과는 메나디온이 고환 기능 지수의 교란으로 이어지는 항산화 방어 시스템을 고갈시킴으로써 고환 독성을 유도한다는 것을 보여줍니다. [19]
induces testicular toxicity 고환 독성 유발
Nonylphenol (NP) is an environmental contaminant, which induces testicular toxicity through oxidative stress. [1] Collectively, the current results indicate that PDB induces testicular toxicity and male reproductive abnormalities by inducing mitochondrial dysfunction, endoplasmic reticulum stress and calcium imbalance. [2] In conclusion, our results suggest that exposure to Sulfx at the two selected doses induces testicular toxicity and these effects can be ameliorated by supplementation of RES. [3] Studies have reported that HAART induces testicular toxicity and impairs sperm quality. [4] We have recently demonstrated that WPS exposure in mice induces testicular toxicity including inflammation and oxidative stress. [5] Diazinon (DZN), as an organophosphate insecticide, induces testicular toxicity through oxidative stress. [6] The findings of this study show that menadione induces testicular toxicity by depleting the antioxidant defense system leading to perturbation in the testicular function indices. [7]nan [1] nan [2] nan [3] nan [4] 우리는 최근에 마우스에서 WPS 노출이 염증 및 산화 스트레스를 포함한 고환 독성을 유도한다는 것을 입증했습니다. [5] 디아지논(DZN)은 유기인계 살충제로서 산화 스트레스를 통해 고환 독성을 유발합니다. [6] 이 연구의 결과는 메나디온이 고환 기능 지수의 교란으로 이어지는 항산화 방어 시스템을 고갈시킴으로써 고환 독성을 유도한다는 것을 보여줍니다. [7]
induces testicular damage 고환 손상 유도
In conclusion, these findings strongly reveal that diabetes induces testicular damage, at least in part, by inducing mitochondrial-mediated apoptosis and oxidative stress. [1] Exposure to dibutyl phthalate (DBP) induces testicular damage in mammals. [2] Diabetes mellitus induces testicular damage, increases sperm abnormalities, and impairs reproductive dysfunction due to induction of endocrine disturbance and testicular oxidative stress. [3] ResultsResults showed that KBrO3 administration induces testicular damage reflected in a significant decrease in total protein, glycogen, sialic acid with a significant increase in MDA level in testicular tissue. [4]nan [1] nan [2] nan [3] 결과결과는 KBrO3 투여가 고환 조직에서 MDA 수준의 유의한 증가와 함께 총 단백질, 글리코겐, 시알산의 유의한 감소에 반영된 고환 손상을 유도하는 것으로 나타났습니다. [4]
induces testicular dysfunction
Collectively, we demonstrated that flufenoxuron inhibits cell proliferation and alters gene expression in mouse testis cells and induces testicular dysfunction in mice. [1] CONCLUSION The present study demonstrates that cadmium induces testicular dysfunction that is attributable to defective G6PD and accompanied by increased lipid peroxidation and impaired NO-dependent endothelial function. [2]nan [1] 결론 현재의 연구는 카드뮴이 결함이 있는 G6PD에 기인하고 지질 과산화 증가 및 NO 의존성 내피 기능 손상을 동반하는 고환 기능 장애를 유도한다는 것을 보여줍니다. [2]