Induces Muscle(근육을 유도)란 무엇입니까?
Induces Muscle 근육을 유도 - Evidence from 21 studies on 402 participants demonstrates that exercise training induces muscle remodelling, and this is accompanied by ECM adaptation. [1] According to the neurophysiology of motion control obtained from experimental studies using animals or humans, the pedunculopontine tegmental nucleus (PPTn) induces muscle tone suppression, and the midbrain locomotor region (MLR) induces muscle tone promotion. [2] Our findings demonstrate that swimming induces muscle damage. [3] Our findings suggest that dexamethasone induces muscle atrophy in zebrafish, inhibiting exploratory behavior by inducing myofiber loss, inhibiting muscle contraction, and causing changes in endurance and velocity. [4] Above 1 g, loading progressively induces muscle lengthening, thus facilitating energy dissipation. [5] We provide further knowledge to understand how exercise induces muscle molecular adaptations towards a slower phenotype, resistant to prolonged trains of stimulation or activity that can be useful as therapeutic tools. [6] IMPACT In people affected by chronic conditions, IMAT accumulation induces muscle mass and strength losses, decline in physical performance, inflammation, and metabolic alterations. [7] FoxO1, a member of the FoxO forkhead type transcription factor family, is markedly upregulated in skeletal muscle in energy-deprived states and induces muscle atrophy via the expression of E3-ubiquitine ligases. [8] The current study shows resistance exercise, but not aerobic exercise, may increase muscle protein synthesis and protein deposition, and induces muscle hypertrophy through LeuRS/mTOR signaling pathway. [9] Skeletal muscle is the primary systemic target organ of stroke that severely induces muscle wasting and weakne. [10] Loss of bone mineral density induces muscle atrophy and increases fracture risk. [11] Passive repetitive stretching induces muscle hypertrophy by regulating signaling pathways involved in muscle protein turnover. [12] ABSTRACT Introduction Resistance exercise training (RET) induces muscle hypertrophy that, when combined with co-temporal protein ingestion, is enhanced. [13] Therefore, our data establish how iNOS induces muscle wasting under cachectic conditions and provide a proof of principle for the repurposing of iNOS inhibitors, such as GW274150 for the treatment of cachexia. [14] On the other hand, lipopolysaccharide (LPS), one of the causative substances of sepsis, induces muscle wasting via toll-like receptors expressed in skeletal muscle. [15] Myasthenia Gravis (MG) is a neuromuscular disorder that induces muscle weakness and fatigue which can be fatal. [16] Blood flow restriction (BFR) exercise is a novel exercise modality in clinical settings, which induces muscle hypertrophy and increases strength with low to moderate training intensity through increased anabolic processes mediated by BFR (usually with cuff inflation). [17] Midazolam is a benzodiazepine that induces muscle relaxation and minimal cardiorespiratory changes. [18] “Previously, several human studies have suggested that exercise can be beneficial in the setting of chronic inflammation, which, in addition to other negative effects, induces muscle wasting and loss of contractile strength. [19] Patients with juvenile idiopathic arthritis (JIA) always experience persistent pain and stiffness which induces muscle weakness, fatigue, and functional limitations. [20] Exercise, which induces muscle remodeling, is beneficial not only for menopause but also for multiple other chronic diseases. [21] Conversely, silencing LMCD1 expression impairs calcium handling and force, and induces muscle fatigue without overt atrophy. [22] A lack of MSTN induces muscle hypertrophy and increases formation of fast-twitch (Type II) muscle fibres. [23] We have recently shown that the acute deletion of the fusion protein Opa1 induces muscle atrophy, systemic inflammatory response, precocious epithelial senescence, and premature death that are caused by muscle-dependent secretion of FGF21. [24] Eccentric cycling training induces muscle hypertrophy and increases joint power output in non-athletes. [25] 1-3 In addition, there is literature suggesting physical exercise induces muscle damage and non-specific inflammatory response manifested Comparison of Some Plasma Inflammation Markers in Elite Master Athletes, Recreational Athletes and Sedentary Males. [26] Neuromuscular electrical stimulation (NMES) combined with blood flow restriction (BFR) induces muscle hypertrophy. [27] Here we show that muscle-specific loss of the pro-fission dynamin related protein (DRP) 1 induces muscle wasting and weakness. [28] Resistance exercise training induces muscle hypertrophy, and recovery between sessions is one of the major determinants of this effect. [29] mTORC1 regulates protein synthesis and degradation and, in skeletal muscles, its activation upon exercise induces muscle growth. [30] Necrosis-inducing anticancer drugs enhance high-mobility group box 1 (HMGB1) release during cell necrosis, and HMGB1-induced autophagy in skeletal muscle induces muscle atrophy. [31] Insulin-like growth factor 1 induces muscle hypertrophy via Nox4, but its function in myoblast fusion remains elusive. [32] Pharmacological elevation of circulating AKG induces muscle hypertrophy, brown adipose tissue (BAT) thermogenesis, and white adipose tissue (WAT) lipolysis in vivo. [33] Our results suggest that EGb761 posttreatment reversed SIM-induces muscle changes possibly through its antioxidant effects, elevation of CoQ10 levels, and antagonizing mitochondrial damage. [34] Although T2D has been reported to induce a catabolic state in skeletal muscle, whether T2D induces muscle wasting in respiratory muscles has not yet been investigated. [35] Here, we tested the long-standing hypothesis that muscle paralysis during cataplexy is caused by recruitment of the brainstem circuit that induces muscle paralysis during REM sleep. [36] In addition, after repeated stimulation with a protocol which induces muscle fatigue, also hind limb muscles show altered relaxation times. [37] , in which the authors demonstrated that fibroblast growth factor 21 (FGF21) induces muscle atrophy and weakness via up-regulation of mitophagy protein Bnip3. [38] We speculate that disruption of the interactions between co-filaments and actin-binding proteins is the primary reason why the K336I mutation induces muscle disease in a dominant fashion. [39] A 2-day exposure to hypoxic conditions induces muscle atrophy, which was significantly worse in Cd34-/- mice compared to wild type (WT). [40] Through the combination of venous occlusion and low-load resistance training, it induces muscle development through a number of proposed mechanisms including anaerobic metabolism, cellular swelling, and induction of type 2 muscle fibers. [41]nan [1] nan [2] nan [3] nan [4] nan [5] nan [6] nan [7] nan [8] nan [9] nan [10] nan [11] nan [12] nan [13] nan [14] nan [15] nan [16] nan [17] nan [18] nan [19] nan [20] 근육의 재형성을 유도하는 운동은 갱년기뿐만 아니라 다른 여러 만성 질환에도 도움이 됩니다. [21] 반대로 LMCD1 발현을 억제하면 칼슘 처리와 힘이 손상되고 명백한 위축 없이 근육 피로가 유발됩니다. [22] MSTN의 결핍은 근육 비대를 유도하고 속근(II형) 근섬유의 형성을 증가시킵니다. [23] 우리는 최근 융합 단백질 Opa1의 급성 결실이 FGF21의 근육 의존적 분비에 의해 유발되는 근육 위축, 전신 염증 반응, 조숙한 상피 노화 및 조기 사망을 유도한다는 것을 보여주었다. [24] 편심 사이클링 훈련은 근육 비대를 유도하고 운동 선수가 아닌 사람들의 관절 파워 출력을 증가시킵니다. [25] 1-3 또한 엘리트 마스터 선수, 레크리에이션 선수 및 앉아있는 남성의 일부 혈장 염증 마커의 비교는 신체 운동이 근육 손상을 유발하고 비특이적 염증 반응을 나타냄을 시사하는 문헌이 있습니다. [26] 혈류 제한(BFR)과 결합된 신경근 전기 자극(NMES)은 근육 비대를 유도합니다. [27] 여기에서 우리는 pro-fission dynamin related protein (DRP) 1의 근육 특이적 손실이 근육 소모와 약화를 유도한다는 것을 보여줍니다. [28] 저항 운동 훈련은 근육 비대를 유발하며 세션 사이의 회복은 이러한 효과의 주요 결정 요인 중 하나입니다. [29] mTORC1은 단백질 합성 및 분해를 조절하고 골격근에서 운동 시 활성화되어 근육 성장을 유도합니다. [30] 괴사 유도 항암제는 세포 괴사 동안 고이동성 그룹 상자 1(HMGB1) 방출을 향상시키고 골격근에서 HMGB1 유도 자가포식을 유도하여 근육 위축을 유도합니다. [31] 인슐린 유사 성장 인자 1은 Nox4를 통해 근육 비대를 유도하지만 근모세포 융합에서 그 기능은 아직 파악하기 어렵습니다. [32] 순환하는 AKG의 약리학적 상승은 생체 내에서 근육 비대, 갈색 지방 조직(BAT) 열발생 및 백색 지방 조직(WAT) 지방분해를 유도합니다. [33] 우리의 결과는 EGb761 후처리 SIM 역전이 항산화 효과, CoQ10 수치 상승 및 미토콘드리아 손상 길항을 통해 근육 변화를 유도할 수 있음을 시사합니다. [34] T2D가 골격근에서 이화작용 상태를 유도하는 것으로 보고되었지만 T2D가 호흡기 근육에서 근육 소모를 유도하는지 여부는 아직 조사되지 않았습니다. [35] 여기에서 우리는 탈력발작 중 근육 마비가 REM 수면 동안 근육 마비를 유도하는 뇌간 회로의 모집에 의해 유발된다는 오랜 가설을 테스트했습니다. [36] 또한, 근육 피로를 유발하는 프로토콜로 반복적인 자극 후에 뒷다리 근육도 변경된 이완 시간을 보여줍니다. [37] , 저자는 섬유아세포 성장 인자 21(FGF21)이 미토파지 단백질 Bnip3의 상향 조절을 통해 근육 위축과 약화를 유도한다는 것을 입증했습니다. [38] 우리는 공동 필라멘트와 액틴 결합 단백질 사이의 상호 작용의 붕괴가 K336I 돌연변이가 근육 질환을 지배적인 방식으로 유도하는 주된 이유라고 추측합니다. [39] 저산소 상태에 2일 노출되면 근육 위축이 유발되며, 이는 야생형(WT)에 비해 Cd34-/- 마우스에서 상당히 더 나쁩니다. [40] 정맥 폐색과 저부하 저항 훈련의 조합을 통해 혐기성 대사, 세포 팽창 및 2형 근섬유의 유도를 포함하여 제안된 여러 메커니즘을 통해 근육 발달을 유도합니다. [41]
Training Induces Muscle
Evidence from 21 studies on 402 participants demonstrates that exercise training induces muscle remodelling, and this is accompanied by ECM adaptation. [1] Eccentric cycling training induces muscle hypertrophy and increases joint power output in non-athletes. [2] Resistance exercise training induces muscle hypertrophy, and recovery between sessions is one of the major determinants of this effect. [3]nan [1] 편심 사이클링 훈련은 근육 비대를 유도하고 운동 선수가 아닌 사람들의 관절 파워 출력을 증가시킵니다. [2] 저항 운동 훈련은 근육 비대를 유발하며 세션 사이의 회복은 이러한 효과의 주요 결정 요인 중 하나입니다. [3]
Exercise Induces Muscle
We provide further knowledge to understand how exercise induces muscle molecular adaptations towards a slower phenotype, resistant to prolonged trains of stimulation or activity that can be useful as therapeutic tools. [1] 1-3 In addition, there is literature suggesting physical exercise induces muscle damage and non-specific inflammatory response manifested Comparison of Some Plasma Inflammation Markers in Elite Master Athletes, Recreational Athletes and Sedentary Males. [2] mTORC1 regulates protein synthesis and degradation and, in skeletal muscles, its activation upon exercise induces muscle growth. [3]nan [1] 1-3 또한 엘리트 마스터 선수, 레크리에이션 선수 및 앉아있는 남성의 일부 혈장 염증 마커의 비교는 신체 운동이 근육 손상을 유발하고 비특이적 염증 반응을 나타냄을 시사하는 문헌이 있습니다. [2] mTORC1은 단백질 합성 및 분해를 조절하고 골격근에서 운동 시 활성화되어 근육 성장을 유도합니다. [3]
1 Induces Muscle
Here we show that muscle-specific loss of the pro-fission dynamin related protein (DRP) 1 induces muscle wasting and weakness. [1] Insulin-like growth factor 1 induces muscle hypertrophy via Nox4, but its function in myoblast fusion remains elusive. [2]여기에서 우리는 pro-fission dynamin related protein (DRP) 1의 근육 특이적 손실이 근육 소모와 약화를 유도한다는 것을 보여줍니다. [1] 인슐린 유사 성장 인자 1은 Nox4를 통해 근육 비대를 유도하지만 근모세포 융합에서 그 기능은 아직 파악하기 어렵습니다. [2]
induces muscle hypertrophy 근육 비대 유도
The current study shows resistance exercise, but not aerobic exercise, may increase muscle protein synthesis and protein deposition, and induces muscle hypertrophy through LeuRS/mTOR signaling pathway. [1] Passive repetitive stretching induces muscle hypertrophy by regulating signaling pathways involved in muscle protein turnover. [2] ABSTRACT Introduction Resistance exercise training (RET) induces muscle hypertrophy that, when combined with co-temporal protein ingestion, is enhanced. [3] Blood flow restriction (BFR) exercise is a novel exercise modality in clinical settings, which induces muscle hypertrophy and increases strength with low to moderate training intensity through increased anabolic processes mediated by BFR (usually with cuff inflation). [4] A lack of MSTN induces muscle hypertrophy and increases formation of fast-twitch (Type II) muscle fibres. [5] Eccentric cycling training induces muscle hypertrophy and increases joint power output in non-athletes. [6] Neuromuscular electrical stimulation (NMES) combined with blood flow restriction (BFR) induces muscle hypertrophy. [7] Resistance exercise training induces muscle hypertrophy, and recovery between sessions is one of the major determinants of this effect. [8] Insulin-like growth factor 1 induces muscle hypertrophy via Nox4, but its function in myoblast fusion remains elusive. [9] Pharmacological elevation of circulating AKG induces muscle hypertrophy, brown adipose tissue (BAT) thermogenesis, and white adipose tissue (WAT) lipolysis in vivo. [10]nan [1] nan [2] nan [3] nan [4] MSTN의 결핍은 근육 비대를 유도하고 속근(II형) 근섬유의 형성을 증가시킵니다. [5] 편심 사이클링 훈련은 근육 비대를 유도하고 운동 선수가 아닌 사람들의 관절 파워 출력을 증가시킵니다. [6] 혈류 제한(BFR)과 결합된 신경근 전기 자극(NMES)은 근육 비대를 유도합니다. [7] 저항 운동 훈련은 근육 비대를 유발하며 세션 사이의 회복은 이러한 효과의 주요 결정 요인 중 하나입니다. [8] 인슐린 유사 성장 인자 1은 Nox4를 통해 근육 비대를 유도하지만 근모세포 융합에서 그 기능은 아직 파악하기 어렵습니다. [9] 순환하는 AKG의 약리학적 상승은 생체 내에서 근육 비대, 갈색 지방 조직(BAT) 열발생 및 백색 지방 조직(WAT) 지방분해를 유도합니다. [10]
induces muscle atrophy 근육 위축 유도
Our findings suggest that dexamethasone induces muscle atrophy in zebrafish, inhibiting exploratory behavior by inducing myofiber loss, inhibiting muscle contraction, and causing changes in endurance and velocity. [1] FoxO1, a member of the FoxO forkhead type transcription factor family, is markedly upregulated in skeletal muscle in energy-deprived states and induces muscle atrophy via the expression of E3-ubiquitine ligases. [2] Loss of bone mineral density induces muscle atrophy and increases fracture risk. [3] We have recently shown that the acute deletion of the fusion protein Opa1 induces muscle atrophy, systemic inflammatory response, precocious epithelial senescence, and premature death that are caused by muscle-dependent secretion of FGF21. [4] Necrosis-inducing anticancer drugs enhance high-mobility group box 1 (HMGB1) release during cell necrosis, and HMGB1-induced autophagy in skeletal muscle induces muscle atrophy. [5] , in which the authors demonstrated that fibroblast growth factor 21 (FGF21) induces muscle atrophy and weakness via up-regulation of mitophagy protein Bnip3. [6] A 2-day exposure to hypoxic conditions induces muscle atrophy, which was significantly worse in Cd34-/- mice compared to wild type (WT). [7]nan [1] nan [2] nan [3] 우리는 최근 융합 단백질 Opa1의 급성 결실이 FGF21의 근육 의존적 분비에 의해 유발되는 근육 위축, 전신 염증 반응, 조숙한 상피 노화 및 조기 사망을 유도한다는 것을 보여주었다. [4] 괴사 유도 항암제는 세포 괴사 동안 고이동성 그룹 상자 1(HMGB1) 방출을 향상시키고 골격근에서 HMGB1 유도 자가포식을 유도하여 근육 위축을 유도합니다. [5] , 저자는 섬유아세포 성장 인자 21(FGF21)이 미토파지 단백질 Bnip3의 상향 조절을 통해 근육 위축과 약화를 유도한다는 것을 입증했습니다. [6] 저산소 상태에 2일 노출되면 근육 위축이 유발되며, 이는 야생형(WT)에 비해 Cd34-/- 마우스에서 상당히 더 나쁩니다. [7]
induces muscle wasting 근육 소모 유발
Skeletal muscle is the primary systemic target organ of stroke that severely induces muscle wasting and weakne. [1] Therefore, our data establish how iNOS induces muscle wasting under cachectic conditions and provide a proof of principle for the repurposing of iNOS inhibitors, such as GW274150 for the treatment of cachexia. [2] On the other hand, lipopolysaccharide (LPS), one of the causative substances of sepsis, induces muscle wasting via toll-like receptors expressed in skeletal muscle. [3] “Previously, several human studies have suggested that exercise can be beneficial in the setting of chronic inflammation, which, in addition to other negative effects, induces muscle wasting and loss of contractile strength. [4] Here we show that muscle-specific loss of the pro-fission dynamin related protein (DRP) 1 induces muscle wasting and weakness. [5] Although T2D has been reported to induce a catabolic state in skeletal muscle, whether T2D induces muscle wasting in respiratory muscles has not yet been investigated. [6]nan [1] nan [2] nan [3] nan [4] 여기에서 우리는 pro-fission dynamin related protein (DRP) 1의 근육 특이적 손실이 근육 소모와 약화를 유도한다는 것을 보여줍니다. [5] T2D가 골격근에서 이화작용 상태를 유도하는 것으로 보고되었지만 T2D가 호흡기 근육에서 근육 소모를 유도하는지 여부는 아직 조사되지 않았습니다. [6]
induces muscle fatigue
Conversely, silencing LMCD1 expression impairs calcium handling and force, and induces muscle fatigue without overt atrophy. [1] In addition, after repeated stimulation with a protocol which induces muscle fatigue, also hind limb muscles show altered relaxation times. [2]반대로 LMCD1 발현을 억제하면 칼슘 처리와 힘이 손상되고 명백한 위축 없이 근육 피로가 유발됩니다. [1] 또한, 근육 피로를 유발하는 프로토콜로 반복적인 자극 후에 뒷다리 근육도 변경된 이완 시간을 보여줍니다. [2]
induces muscle damage
Our findings demonstrate that swimming induces muscle damage. [1] 1-3 In addition, there is literature suggesting physical exercise induces muscle damage and non-specific inflammatory response manifested Comparison of Some Plasma Inflammation Markers in Elite Master Athletes, Recreational Athletes and Sedentary Males. [2]nan [1] 1-3 또한 엘리트 마스터 선수, 레크리에이션 선수 및 앉아있는 남성의 일부 혈장 염증 마커의 비교는 신체 운동이 근육 손상을 유발하고 비특이적 염증 반응을 나타냄을 시사하는 문헌이 있습니다. [2]