Iia Inhibits
Iia 억제

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Tan Iia Inhibits

In conclusion, Tan IIA inhibits LPS‑induced inflammatory responses in VSMCs in vitro through the partial suppression of the TLR4/TAK1/NF‑κB signaling pathway.

결론적으로, Tan IIA는 TLR4/TAK1/NF-κB 신호전달 경로의 부분적 억제를 통해 시험관 내 VSMC에서 LPS 유도 염증 반응을 억제합니다.

Tanshinone IIA inhibits lipopolysaccharide‑induced inflammatory responses through the TLR4/TAK1/NF‑κB signaling pathway in vascular smooth muscle cells.

Tan Iia Inhibits

Collectively, present study demonstrates that Tan IIA inhibits Hcy-induced proliferation of VSMCs via miR-145/CD40 signaling.

종합적으로, 현재 연구는 Tan IIA가 miR-145/CD40 신호 전달을 통해 VSMC의 Hcy 유도 증식을 억제한다는 것을 보여줍니다.

Tanshinone ⅡA inhibits homocysteine-induced proliferation of vascular smooth muscle cells via miR-145/CD40 signaling.

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10.1016/j.pupt.2021.101990

CONCLUSION Tan-IIA inhibits the proliferation and inflammatory response of PAECs by downregulating the expression of HMGB1 via the TLR4/NF-κB signalling pathway under hypoxic conditions.

결론 Tan-IIA는 저산소 상태에서 TLR4/NF-κB 신호 전달 경로를 통해 HMGB1의 발현을 하향 조절함으로써 PAEC의 증식 및 염증 반응을 억제합니다.

Tanshinone-IIA inhibits the inflammatory response and proliferation of PAECs under hypoxic conditions by repressing HMGB1 via the TLR4/NF-κB signalling pathway.

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Iia Inhibits Iia 억제 - CONCLUSION Tan-IIA inhibits the proliferation and inflammatory response of PAECs by downregulating the expression of HMGB1 via the TLR4/NF-κB signalling pathway under hypoxic conditions. [1]
결론 Tan-IIA는 저산소 상태에서 TLR4/NF-κB 신호 전달 경로를 통해 HMGB1의 발현을 하향 조절함으로써 PAEC의 증식 및 염증 반응을 억제합니다. [1]

Tan Iia Inhibits

In conclusion, Tan IIA inhibits LPS‑induced inflammatory responses in VSMCs in vitro through the partial suppression of the TLR4/TAK1/NF‑κB signaling pathway. [1] Collectively, present study demonstrates that Tan IIA inhibits Hcy-induced proliferation of VSMCs via miR-145/CD40 signaling. [2]
결론적으로, Tan IIA는 TLR4/TAK1/NF-κB 신호전달 경로의 부분적 억제를 통해 시험관 내 VSMC에서 LPS 유도 염증 반응을 억제합니다. [1] 종합적으로, 현재 연구는 Tan IIA가 miR-145/CD40 신호 전달을 통해 VSMC의 Hcy 유도 증식을 억제한다는 것을 보여줍니다. [2]