Depressed Left
우울한 왼쪽

Depressed Left(우울한 왼쪽)란 무엇입니까?

Depressed Left 우울한 왼쪽 - Catheter AF ablation is a class I indication for symptomatic patients refractory to at least one oral antiarrhythmic drug, and a class IIa for those without prior drug failure1 It may be considered in asymptomatic patients with depressed left ventricular function. ^[1] MB also has been associated with angina, myocardial infarction, arrhythmia, depressed left ventricular function, left bundle branch block, myocardial stunning, apical ballooning syndrome, early death after cardiac transplantation, and sudden death. ^[2] Cats with EMF-RCM had depressed left ventricular myocardial deformations both in systole (whole longitudinal strain, epicardial longitudinal strain, and endocardial circumferential strain) and diastole (early and late diastolic longitudinal strain rates, and late diastolic circumferential strain rate) compared to controls. ^[3] Depressed left ventricular ejection fraction was found in 17. ^[4] Sibutramine (10 μM) induced Ca2+-release from the sarcoplasmic reticulum under either electrical or caffeine stimulation, whereas it depressed left ventricular developed pressure with a marked decrease in the end-diastolic pressure. ^[5] Previous studies reported depressed left ventricular function in DMD patients which may result from deranged intracellular Ca2+‐handling. ^[6] We report a case of ARNI started as first-choice therapy in a patient with relapsing hairy cell leukaemia (HCL) and HF with depressed left ventricular ejection fraction (LVEF). ^[7] 57m2) with a history of acute myeloid leukemia and anthracycline chemotherapy in infancy developed mildly depressed left ventricular (LV) function at 2 years of age. ^[8] To clear the suspect of an acute coronary syndrome, a radial access coronary angiography was performed: normal coronary arteries but a diffuse severely depressed left ventricular function (ejection fraction 27%) were apparent. ^[9] They presented with severely depressed left ventricle function and underwent heart transplantation with excellent outcomes. ^[10] 40]), whereas depressed left ventricular ejection fraction did not (odds ratio, 0. ^[11] A 77-year-old woman with paced-induced cardiomyopathy and depressed left ventricular ejection fraction was admitted to our hospital for left bundle pacing (LBP) for cardiac resynchronization therapy after loss of capture with his-bundle pacing. ^[12] Transthoracic echocardiography (TTE) revealed depressed left ventricular (LV) systolic function and thick prominent trabeculae in the LV, predominantly in the apex, suggesting LVNC. ^[13] Her subtle clinical presentation could have led to a missed diagnosis as she was also not in a fluid overloaded state consistent with the depressed left ventricular function discovered on transthoracic echo. ^[14] This includes treatment of patients with multivessel disease, unprotected left main, acute myocardial infarction complicated by cardiogenic shock, and depressed left ventricular ejection fraction. ^[15] Echocardiogram demonstrated severely depressed left ventricular (LV) function with severe mitral regurgitation. ^[16] Cardiac magnetic resonance imaging revealed the diagnosis of non-compaction cardiomyopathy with severely depressed left ventricular function. ^[17] Low-flow, low-gradient (LF-LG) aortic stenosis with depressed left ventricular (LV) ejection fraction is a diagnostic challenge that is frequently encountered in the management of valvular heart disease. ^[18] However, the presence of low cardiac output state and acute electrocardiogram (ECG) changes, along with echocardiographic evidence of severely depressed left ventricular systolic function occurring concurrently with the acute mental status changes, pointed toward cardiogenic shock as the etiology of her encephalopathy. ^[19] CTRP9 markedly ameliorated macrophage infiltration and attenuated the inflammatory responses by downregulating interleukin‐1β and interleukin‐6, and upregulating interleukin‐10, in 3 days post‐MI; depressed left atrial fibrosis by decreasing the expressions of collagen types I and III, α‐SMA, and transforming growth factor β1 in 7 days post‐MI possibly through depressing the Toll‐like receptor 4/nuclear factor‐κB and Smad2/3 signaling pathways. ^[20] Depressed left ventricular systolic function (hazard ratio, 2. ^[21] An 84-year old woman presented to the emergency room in cardiogenic shock with severely depressed left ventricular ejection fraction (LVEF 15%) due to anterior, apical and septal akinesia. ^[22] In the echocardiography, depressed left ventricular ejection fraction (30%), akinesia and dilatation of the mid and apical portions of left ventricle were seen without an obstruction in the outflow tract (Video 3). ^[23] Advancements in medical treatment, percutaneous interventions, and device therapy (ICD and CRTD) showed consistent reduction in mortality, mainly in survivors of SCD and in patients with ischemic cardiomyopathy and depressed left ventricular function. ^[24] Coronary angiography revealed no remarkable coronary stenosis, and left ventriculography showed a depressed left ventricular ejection fraction (EF) of 23%. ^[25] Objective The aim of this study was to assess the role of nitrate-augmented technetium-99m (99mTc) sestamibi imaging in the evaluation of viable myocardium in patients who had ischemic heart disease with depressed left ventricle (LV) systolic function. ^[26] Prosthesis-patient mismatch is also associated with pulmonary hypertension and depressed left ventricle systolic function. ^[27] TAVR patients had significantly more comorbidities compared to ConsRx or SAVR patients, including older age, previous myocardial infarction, diabetes, renal failure, and more depressed left ventricular function as assessed by NT-proBNP (all with P  Conclusion SAVR and TAVR were associated with better survival compared to ConsRx in both classical and paradoxical LFLG AS patients. ^[28] GENERAL ELECTROPHYSIOLOGY AP1900005 | A retrospective comparative study of 12 lead electrocardiogram and 2D—Echocardiography derived left ventricular ejection fraction among patients of Fatima University Medical Center Jeriko Henry Aguirrem; Marie Sylvie Easter Gunigundo Fatima University Medical Center, Philippines Introduction: The 12 lead electrocardiogram (12L ECG) is a standard diagnostic test for patients with cardiac diseases especially those with depressed left ventricular ejection fraction. ^[29] He had coronary artery disease with severely depressed left ventricular ejection fraction and had received an implantable cardioverter defibrillator (ICD) for primary prevention 4 years previously. ^[30] Patients with HF in NYHA class II-IV and depressed left ventricular ejection fraction ≤50%, functional capacity ≥4 metabolic equivalents (METs) and≤ 85 years were randomly assigned to EFICAR or optimized standard care groups. ^[31] He was diagnosed with transfusion-associated circulatory overload (TACO) due to the presence of acute pulmonary congestion and depressed left ventricular systolic function. ^[32] Progressive improvements in ICD device has expanded the prophylactic use of ICD to include pre-emptive therapy in a large population of patients with significantly depressed left ventricular systolic function. ^[33] Cardiac echography showed a depressed left ventricular function (ejection fraction 35%). ^[34] Advances in cardiovascular imaging have identified several anatomic and functional variables that can be quantified as continuous variables to predict the risk of developing lethal ventricular tachyarrhythmias in patients with depressed left ventricular (LV) systolic function. ^[35] METHODS A phase I trial was conducted on adult patients suffering from ischemic heart disease with depressed left ventricular ejection fraction who were scheduled to undergo TMR or CABG. ^[36] There were enophthalmos, hypoglobus of the left eye, flat nasal bridge, and depressed left malar eminence. ^[37] OBJECTIVE Predictors of operative outcome in patients with severely depressed left ventricular ejection fraction (LVEF) undergoing coronary artery bypass grafting (CABG) remain poorly defined. ^[38] OBJECTIVE To discern the impact of depressed left ventricular ejection fraction (LVEF) on the outcomes of open descending thoracic aneurysm (DTA) and thoracoabdominal aneurysms (TAAA) repair. ^[39] Bentall procedure was successfully performed, but he had severely depressed left ventricular function and muscle wasting together with vascular inflammation. ^[40] Focused cardiac ultrasound interpretation was dichotomized for the presence or absence of pericardial effusion, depressed left ventricular function, and chamber size abnormalities. ^[41] Patients with AF also had depressed left ventricular (LV) function compared to patients without AF. ^[42] Repeat coronary arteriography showed an absence of significant stenosis of the epicardial coronary arteries (Figure 2), with a depressed left ventricular ejection fraction (32%) at ventriculography. ^[43]