Deficiency Regulates 결핍 조절

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10.1101/2021.08.26.457753

Independently of noncanonical NF- κB, NIK deficiency regulates mitochondrial spare respiratory capacity (SRC) and proton leak but establishes higher basal oxygen consumption and glycolytic capacity in preadipocytes and ex vivo adipose tissue.

비표준 NF-κB와 별도로 NIK 결핍은 미토콘드리아 예비 호흡 능력(SRC) 및 양성자 누출을 조절하지만 지방전구 세포 및 생체 외 지방 조직에서 더 높은 기초 산소 소비 및 해당 능력을 확립합니다.

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10.1038/s42003-021-01900-0

They find that gestational vitamin B6 deficiency decreases serotonin levels in pancreatic islets and reduces β-cell proliferation, showing that vitamin B6 deficiency regulates maternal glucose tolerance in a serotonin-dependent manner.

그들은 임신성 비타민 B6 결핍이 췌도에서 세로토닌 수치를 감소시키고 β-세포 증식을 감소시킨다는 것을 발견하여 비타민 B6 결핍이 세로토닌 의존적 방식으로 산모의 포도당 내성을 조절한다는 것을 보여줍니다.

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10.1016/j.redox.2021.102003

Overall, our results reveal how Se deficiency regulates NET formation in the progression of arteritis and support silencing-SelS worsens arteritis.

전반적으로, 우리의 결과는 Se 결핍이 동맥염의 진행에서 NET 형성을 조절하고 침묵-SelS가 동맥염을 악화시키는 방법을 보여줍니다.

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10.1016/j.jaci.2019.05.041

CONCLUSIONS Overall, our study has provided a novel underlying molecular mechanism of how DOCK2 deficiency regulates surface expression of CD21, which leads to downregulation of CD19-mediated BCR signaling and marginal zone B-cell differentiation.

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10.1165/rcmb.2019-0161ED

Studies have shown that decreased expression of TRIM21 promotes cell growth in breast cancer cells (13), Trim21 deficiency regulates NF-kB–dependent proinflammatory cytokine production in fibroblasts (14), and augmented T-helper cell type 17 differentiation in TRIM21 deficiency promotes a more fibrous, stable phenotype of atherosclerotic plaques with high collagen content (15).

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Deficiency Regulates 결핍 조절

Deficiency Regulates 결핍 조절

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Dec 11, 2023

Deficiency Regulates(결핍 조절)란 무엇입니까?

Deficiency Regulates 결핍 조절 - Independently of noncanonical NF- κB, NIK deficiency regulates mitochondrial spare respiratory capacity (SRC) and proton leak but establishes higher basal oxygen consumption and glycolytic capacity in preadipocytes and ex vivo adipose tissue. ^[1] They find that gestational vitamin B6 deficiency decreases serotonin levels in pancreatic islets and reduces β-cell proliferation, showing that vitamin B6 deficiency regulates maternal glucose tolerance in a serotonin-dependent manner. ^[2] Overall, our results reveal how Se deficiency regulates NET formation in the progression of arteritis and support silencing-SelS worsens arteritis. ^[3] CONCLUSIONS Overall, our study has provided a novel underlying molecular mechanism of how DOCK2 deficiency regulates surface expression of CD21, which leads to downregulation of CD19-mediated BCR signaling and marginal zone B-cell differentiation. ^[4] Studies have shown that decreased expression of TRIM21 promotes cell growth in breast cancer cells (13), Trim21 deficiency regulates NF-kB–dependent proinflammatory cytokine production in fibroblasts (14), and augmented T-helper cell type 17 differentiation in TRIM21 deficiency promotes a more fibrous, stable phenotype of atherosclerotic plaques with high collagen content (15). ^[5]
비표준 NF-κB와 별도로 NIK 결핍은 미토콘드리아 예비 호흡 능력(SRC) 및 양성자 누출을 조절하지만 지방전구 세포 및 생체 외 지방 조직에서 더 높은 기초 산소 소비 및 해당 능력을 확립합니다. ^[1] 그들은 임신성 비타민 B6 결핍이 췌도에서 세로토닌 수치를 감소시키고 β-세포 증식을 감소시킨다는 것을 발견하여 비타민 B6 결핍이 세로토닌 의존적 방식으로 산모의 포도당 내성을 조절한다는 것을 보여줍니다. ^[2] 전반적으로, 우리의 결과는 Se 결핍이 동맥염의 진행에서 NET 형성을 조절하고 침묵-SelS가 동맥염을 악화시키는 방법을 보여줍니다. ^[3] nan ^[4] nan ^[5]