Cell Residency 셀 레지던시

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10.1016/J.JACI.2020.12.148

Differentially expressed genes (DEG) in T-cells from affected skin reflected homing (CCR7,S1PR1), T-cell activation and proliferation (TNFRS9, GRAP2, ZYG11A, ZHX1), T-cell effector differentiation (IL-21R), and stress survival (EEF1A1, IFI6) without representation of markers of T-cell residency (ITGAE) or regulation.

영향을 받은 피부의 T 세포에서 차등적으로 발현된 유전자(DEG)는 귀환(CCR7, S1PR1), T 세포 활성화 및 증식(TNFRS9, GRAP2, ZYG11A, ZHX1), T 세포 효과기 분화(IL-21R) 및 스트레스를 반영합니다. T-세포 잔류(ITGAE) 또는 조절의 마커를 나타내지 않는 생존(EEF1A1, IFI6).

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10.1172/jci.insight.153551

In contrast, direct glucocorticoid signaling of donor T-cells promotes chemokine and integrin signatures permissive of preferential circulation and migration into the bone marrow, promoting donor T-cell residency.

대조적으로, 기증자 T 세포의 직접적인 글루코코르티코이드 신호전달은 케모카인 및 인테그린 서명을 촉진하여 우선적인 순환 및 골수로의 이동을 허용하여 기증자 T 세포 체류를 촉진합니다.

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Cell Residency 셀 레지던시

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Jan 30, 2023

Cell Residency(셀 레지던시)란 무엇입니까?

Cell Residency 셀 레지던시 - Differentially expressed genes (DEG) in T-cells from affected skin reflected homing (CCR7,S1PR1), T-cell activation and proliferation (TNFRS9, GRAP2, ZYG11A, ZHX1), T-cell effector differentiation (IL-21R), and stress survival (EEF1A1, IFI6) without representation of markers of T-cell residency (ITGAE) or regulation. ^[1] In contrast, direct glucocorticoid signaling of donor T-cells promotes chemokine and integrin signatures permissive of preferential circulation and migration into the bone marrow, promoting donor T-cell residency. ^[2]
영향을 받은 피부의 T 세포에서 차등적으로 발현된 유전자(DEG)는 귀환(CCR7, S1PR1), T 세포 활성화 및 증식(TNFRS9, GRAP2, ZYG11A, ZHX1), T 세포 효과기 분화(IL-21R) 및 스트레스를 반영합니다. T-세포 잔류(ITGAE) 또는 조절의 마커를 나타내지 않는 생존(EEF1A1, IFI6). ^[1] 대조적으로, 기증자 T 세포의 직접적인 글루코코르티코이드 신호전달은 케모카인 및 인테그린 서명을 촉진하여 우선적인 순환 및 골수로의 이동을 허용하여 기증자 T 세포 체류를 촉진합니다. ^[2]