術後副甲状腺機能低下症とは何ですか？
Postoperative Hypoparathyroidism 術後副甲状腺機能低下症 - Postoperative hypoparathyroidism is still an urgent problem, as it requires additional treatment and prolongs hospital-stay. [1] OBJECTIVE to determine the usefulness of high urine output as a low-cost early predictor for postoperative hypoparathyroidism. [2] Notwithstanding the actual surgical skill, postoperative hypoparathyroidism still represents its most frequent complication. [3] We present a case report of a 38-year-old woman with postoperative hypoparathyroidism treated with rhPTH who subsequently developed calciphylactic lesions on her abdomen. [4] Advances in intraoperative technology to optimize the vascularization of the parathyroid glands can help to predict and prevent patients from a postoperative hypoparathyroidism. [5] The postoperative hypoparathyroidism rate in the CNs group was significantly lower than that in the control group (P<0. [6] The procedure is generally safe, although a minority of cases could present complications as postoperative hypoparathyroidism or recurrent laryngeal nerve dysfunction [1]. [7] We aimed to investigate the prevalence of postoperative hypoparathyroidism (PoH), the relevant factors, and predictors of transient or permanent hypoparathyroidism. [8] The clinical characteristics of patients with postoperative hypoparathyroidism who recover parathyroid function more than 12 months after surgery have not been studied. [9] In the case of surgery, postoperative hypoparathyroidism was significantly more frequent following more extensive surgical procedures. [10] In this study, we aimed to evaluate the relationship between the recovery time of parathyroid gland function and patient characteristics, preoperative and postoperative electrolyte changes, and intraoperative parathyroid findings in patients with postoperative hypoparathyroidism. [11] BACKGROUND To investigate how number of autotransplanted parathyroid glands (PGs) affects the incidence of postoperative hypoparathyroidism and the recovery of parathyroid function. [12] Background Definitions of postoperative hypoparathyroidism (hypoPT) have never reached consent until the American Thyroid Association (ATA) statement was released, with new characteristics and challenges. [13] However, this modality did not result in the reduction of postoperative hypoparathyroidism or hypocalcemia risk. [14] This study aimed to explore the relationship between parathyroid gland autotransplantation and postoperative hypoparathyroidism. [15] However, as predicted, LT completely eliminated the risk of postoperative hypoparathyroidism. [16] We wished to investigate the association between number of parathyroids identified intraoperatively during TT, and incidence of incidental parathyroidectomy, and postoperative hypoparathyroidism. [17] BACKGROUND Postoperative hypoparathyroidism (PH) is the most common complication after total thyroidectomy. [18] RESULTS The incidence of postoperative hypoparathyroidism (PTH < 15pg/mL) was significantly lower in the NIRAF group during the hospitalization (88 patients: 33. [19] This method could be a cost- and labor-effective alternative to frozen sections to reduce the incidence of postoperative hypoparathyroidism and improve the outcome of primary hyperparathyroidism in low-resource areas. [20] Introduction: Postoperative hypoparathyroidism is one of the most common complications after total thyroidectomy. [21] The authors hypothesized that (1) for CEC patients undergoing PLE with thyroid lobectomy, definitive chemoradiotherapy (CRT) would have some effect on postoperative hypothyroidism, and (2) PLTE may also have some effect on postoperative hypoparathyroidism. [22] The aim of this paper was to determine whether the rate of postoperative hypoparathyroidism (HPT) is influenced by whether surgery is staged. [23] Postoperative hypoparathyroidism was defined as low parathyroid hormone (PTH) levels (<1. [24] Postoperative hypoparathyroidism/hypocalcemia is the most prevalent complication following thyroid and parathyroid surgeries. [25] When compared with other PHPT patients, PC patients were more frequently male and had higher preoperative blood calcium and PTH and lower phosphate levels, larger and heavier parathyroids excised, lower postoperative calcium, and a higher rate of postoperative hypoparathyroidism. [26] Postoperative hypoparathyroidism requiring long-term calcium and 1alpha(OH) D3 treatment was reported in 5% of patients in group A*3/4 and 26% of patients in group B*7/8 (P=0. [27] A study form consisting of patient information on possible risk factors for osteoporosis such as gender, age, menopausal status, smoking, family history of osteoporosis, preoperative thyroid hormone status, postoperative hypoparathyroidism history, mean serum TSH levels, and duration of TSH suppression was created and filled out for each participant. [28] Injury to parathyroid glands during thyroid and parathyroid surgery is common and postoperative hypoparathyroidism represents a serious complication. [29] Objective: To analyze the challenges and the impact of specialized care in diagnosis, treatment and in follow-up of patients with postoperative hypoparathyroidism, with potential to improve teaching and further research possibilities in the subject. [30] 22, respectively), whereas the LC&R group patients had significantly longer operation time, higher blood loss, and a higher rate of postoperative hypoparathyroidism compared with the L group (P <. [31] This is mostly attributed to the lower rates of surgical complications, such as postoperative hypoparathyroidism and airway obstruction. [32] There were five (4, PTxT+A; 1, SPTx) patients with postoperative hypoparathyroidism, one with persistent HPT (SPTx) and 13 with normal parathyroid function (4, TPTx+A; 9, SPTx) six months after PTx. [33] Postoperative hypoparathyroidism is the major complication after surgery for benign thyroid disease, thus requiring more awareness. [34] Conclusions The risk of morbidity concerning the recurrent nerve injury and postoperative hypoparathyroidism increases with the extent of surgery. [35] Conclusion The application of nano-carbon technology in thyroidectomy can completely clear the lymph nodes, reduce the occurrence of incidental parathyroidectomy, and reduce the incidence of postoperative hypoparathyroidism, low parathyroid hormone and hypocalcemia. [36] No association was found between postoperative hypoparathyroidism and autotransplantation of a parathyroid gland. [37] ImportancePostoperative hypoparathyroidism remains a relevant problem after thyroid surgery. [38] The prevalence of postoperative hypoparathyroidism has been studied in registries and in surgical series with highly variable and imprecise results. [39] For the prevention of postoperative hypoparathyroidism, in addition to carefully accounting for anatomical and topographic-anatomical features, a «stress-test» and a method of double visual-instrumental recording of the parathyroid gland-induced fluorescence were used. [40]術後の副甲状腺機能低下症は、追加の治療が必要であり、入院期間が長くなるため、依然として緊急の問題です。 [1] 目的 術後副甲状腺機能低下症の低コストの早期予測因子としての高尿量の有用性を判断すること。 [2] 実際の外科的スキルにもかかわらず、術後副甲状腺機能低下症は依然として最も頻繁な合併症を表しています。 [3] 術後副甲状腺機能低下症の38歳の女性がrhPTHで治療され、その後腹部に石灰化病変を発症した症例報告を提示します。 [4] 副甲状腺の血管新生を最適化するための術中技術の進歩は、患者の術後副甲状腺機能低下症を予測および予防するのに役立ちます。 [5] CNs群の術後副甲状腺機能低下症率は対照群よりも有意に低かった（P <0）。 [6] この手順は一般的に安全ですが、少数の症例では術後の副甲状腺機能低下症または再発性喉頭神経機能障害などの合併症が現れる可能性があります[1]。 [7] 術後副甲状腺機能低下症（PoH）の有病率、関連する要因、および一過性または永続的な副甲状腺機能低下症の予測因子を調査することを目的とした。 [8] 術後12ヶ月以上副甲状腺機能が回復した術後副甲状腺機能低下症患者の臨床的特徴は研究されていません。 [9] 手術の場合、術後の副甲状腺機能低下症は、より広範な外科的処置の後に有意に頻繁でした。 [10] 本研究では、副甲状腺機能の回復時間と患者の特徴、術前および術後の電解質変化、および術後副甲状腺機能低下症患者における術中副甲状腺所見との関係を評価することを目的とした。 [11] バックグラウンド 自己移植された副甲状腺（PG）の数が、術後の副甲状腺機能低下症の発生率と副甲状腺機能の回復にどのように影響するかを調査すること。 [12] 背景術後副甲状腺機能低下症（hypoPT）の定義は、米国甲状腺協会（ATA）の声明が発表されるまで、新しい特徴と課題を伴って同意に達していません。 [13] ただし、このモダリティは、術後の副甲状腺機能低下症または低カルシウム血症のリスクの低下をもたらしませんでした。 [14] この研究は、副甲状腺の自家移植と術後の副甲状腺機能低下症との関係を調査することを目的とした。 [15] しかし、予測されたように、LTは術後副甲状腺機能低下症のリスクを完全に排除しました。 [16] TT中に術中に特定された副甲状腺の数と、偶発的な副甲状腺摘出術の発生率、および術後の副甲状腺機能低下症との関連を調査したかった。 [17] バックグラウンド 術後副甲状腺機能低下症（PH）は、甲状腺全摘術後の最も一般的な合併症です。 [18] 結果 術後副甲状腺機能低下症（PTH <15pg / mL）の発生率は、入院中のNIRAFグループで有意に低かった（88人の患者：33人）。 [19] この方法は、術後の副甲状腺機能低下症の発生率を減らし、資源の少ない地域での原発性副甲状腺機能低下症の結果を改善するために、凍結切片に代わる費用と労力のかかる代替手段となる可能性があります。 [20] はじめに：術後副甲状腺機能低下症は、甲状腺全摘術後の最も一般的な合併症の1つです。 [21] 著者らは、（1）甲状腺葉切除を伴うPLEを受けているCEC患者の場合、根治的化学放射線療法（CRT）が術後甲状腺機能低下症に何らかの影響を及ぼし、（2）PLTEが術後副甲状腺機能低下症にも何らかの影響を与える可能性があると仮定した。 [22] この論文の目的は、術後副甲状腺機能低下症（HPT）の発生率が、手術が段階的に行われるかどうかによって影響を受けるかどうかを判断することでした。 [23] 術後の副甲状腺機能低下症は、副甲状腺ホルモン（PTH）レベルの低下（<1）と定義されました。 [24] 術後の副甲状腺機能低下症/低カルシウム血症は、甲状腺および副甲状腺の手術後の最も一般的な合併症です。 [25] 他のPHPT患者と比較した場合、PC患者は男性であることが多く、術前の血中カルシウムとPTHが高く、リン酸塩レベルが低く、切除された副甲状腺が大きくて重く、術後のカルシウムが低く、術後の副甲状腺機能低下症の発生率が高かった。 [26] nan [27] nan [28] nan [29] nan [30] nan [31] nan [32] nan [33] nan [34] nan [35] nan [36] nan [37] nan [38] nan [39] nan [40]
lymph node dissection
Female sex, neck dissection, the yield of lymph node dissection and incidental parathyroidectomy were significantly associated with postoperative hypoparathyroidism. [1] Univariate analysis showed that postoperative hypoparathyroidism was associated with cervical lymph node dissection, gender, surgical range, operation time, central region lymph node dissection and preoperative PTH level (P 1). [2]女性の性別、頸部郭清術、リンパ節郭清術の収量、および偶発的な副甲状腺摘出術は、術後の副甲状腺機能低下症と有意に関連していた。 [1] nan [2]
Chronic Postoperative Hypoparathyroidism
The chronic postoperative hypoparathyroidism accounts approximately 75% of patients; the genetic, autoimmune or idiopathic forms are significantly less common. [1] We present a case report of a woman with convulsions, which developed due to dissociative (conversion motor) disorder, with chronic postoperative hypoparathyroidism, receiving standard treatment and having normo- and hypercalcemia. [2] One patient had chronic postoperative hypoparathyroidism and one patient remained normocalcemic 4 years after surgery. [3] One patient had chronic postoperative hypoparathyroidism, and one patient remains normocalcemic 4 years after surgery. [4]慢性的な術後副甲状腺機能低下症は患者の約75％を占めます。遺伝的、自己免疫的または特発性の形態はかなり一般的ではありません。 [1] 解離性（転換運動）障害により発症した、慢性の術後副甲状腺機能低下症、標準治療を受け、正常カルシウム血症および高カルシウム血症の女性の症例報告を提示します。 [2] nan [3] nan [4]
Transient Postoperative Hypoparathyroidism
1%) with transient postoperative hypoparathyroidism, and 21 patients (3. [1] The image-based NIRAF device has been shown in one multicenter prospective study to lower the risk of transient postoperative hypoparathyroidism [3]. [2] Transient postoperative hypoparathyroidism with hypocalcaemia was reported in 8 (9. [3]1％）一過性の術後副甲状腺機能低下症、および21人の患者（3。 [1] 画像ベースのNIRAFデバイスは、ある多施設前向き研究で、一過性の術後副甲状腺機能低下症のリスクを低下させることが示されています[3]。 [2] nan [3]
Permanent Postoperative Hypoparathyroidism
Accidental damage or removal of the PG may result in temporary or permanent postoperative hypoparathyroidism and hypocalcemia. [1] 4%) and permanent postoperative hypoparathyroidism (2. [2]偶発的な損傷またはPGの除去は、一時的または永続的な術後副甲状腺機能低下症および低カルシウム血症を引き起こす可能性があります。 [1] nan [2]
Developed Postoperative Hypoparathyroidism
In both groups participants who developed postoperative hypoparathyroidism (parathormone (iPTH) <1. [1] The time intervals for thyroid disorders were 320 cases developed postoperative hypoparathyroidism in eight weeks, 480 cases the second month, and 1000 cases in the first year after surgery. [2]両方のグループで、術後副甲状腺機能低下症（副甲状腺機能低下症（iPTH）<1）を発症した参加者。 [1] nan [2]
Preventing Postoperative Hypoparathyroidism
Identification of the parathyroid glands during surgery is crucial for preventing postoperative hypoparathyroidism. [1] Recognition of normal parathyroid glands using ultrasound can be valuable for preventing postoperative hypoparathyroidism and in increasing the accuracy of postsurgical ultrasound surveillance. [2]手術中の副甲状腺の識別は、術後の副甲状腺機能低下症を予防するために重要です。 [1] 超音波を使用した正常な副甲状腺の認識は、術後の副甲状腺機能低下症を予防し、術後の超音波監視の精度を高めるのに役立ちます。 [2]