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10.1136/bmjopen-2019-032844

This is proposed to result from non-specific protection against non-vaccine targeted (‘off-target’) infections.

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10.1016/j.ygyno.2019.05.024

The HPV genotyping findings were grouped as: HPV16/18+, other hrHPV+ (HPV31/33/35/39/45/51/52/56/58/59/66/68), non-vaccine targeted hrHPV+ (HPV35/39/51/56/59/66/68), low-risk HPV, and HPV negative.

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10.1016/j.ejps.2019.105096

We developed a vaccine targeted to Human papillomavirus (HPV)-induced tumours based on recombinant Semliki Forest virus (rSFV) encoding a fusion protein of the E6 and E7 proteins of HPV type 16.

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10.1016/j.pvr.2019.100190

, at a median follow up of 7 years, the rates of incident vaccine targeted HPV infection were not significantly different between married two dose schedule (180 days +) and three dose recipients and were lower than in the control group [13].

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10.1111/imr.12807

Here we review the role of ICAM‐1 and EPCR receptor adhering falciparum‐parasites in the development of severe malaria; we discuss our current research to understand the factors involved in the pathogenesis of cerebral malaria and the feasibility of developing a vaccine targeted specifically to prevent this disease.

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Vaccine Targeted

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Oct 11, 2024

What is/are Vaccine Targeted?

Vaccine Targeted - This is proposed to result from non-specific protection against non-vaccine targeted (‘off-target’) infections. ^[1] The HPV genotyping findings were grouped as: HPV16/18+, other hrHPV+ (HPV31/33/35/39/45/51/52/56/58/59/66/68), non-vaccine targeted hrHPV+ (HPV35/39/51/56/59/66/68), low-risk HPV, and HPV negative. ^[2] We developed a vaccine targeted to Human papillomavirus (HPV)-induced tumours based on recombinant Semliki Forest virus (rSFV) encoding a fusion protein of the E6 and E7 proteins of HPV type 16. ^[3] , at a median follow up of 7 years, the rates of incident vaccine targeted HPV infection were not significantly different between married two dose schedule (180 days +) and three dose recipients and were lower than in the control group [13]. ^[4] Here we review the role of ICAM‐1 and EPCR receptor adhering falciparum‐parasites in the development of severe malaria; we discuss our current research to understand the factors involved in the pathogenesis of cerebral malaria and the feasibility of developing a vaccine targeted specifically to prevent this disease. ^[5]