Introduction to Sinus Syndrome

What is/are Sinus Syndrome?

Sinus Syndrome - Further manifestations of the mitochondrial disorder (MID) were tremor, cerebral atrophy, bilateral basal ganglia, calcification, glaucoma, hypoacusis, short stature, hyperostosis frontalis, hyperthyroidism, sick-sinus syndrome and AV-block-1, and myopathy. ^[1] 8%) had a PPM implanted >30 days postoperatively, at a median of 43 months after surgery (range 0‒181), most often for AV block (n = 13) or sick-sinus syndrome (n = 10). ^[2]

year old woman

Case presentation A 94-year-old woman was diagnosed with sick sinus syndrome and underwent a dual-chamber permanent pacemaker implantation. ^[1] Patient concerns: A subject of the study is a 74-year-old woman with a sick sinus syndrome (SSS) and a previously implanted physiological DDD/R pacemaker. ^[2] A 63-year-old woman with a history of hypertension, sick sinus syndrome, and Hashimoto’s thyroiditis was referred to our facility for evaluationof amass in segment 8of the liver. ^[3] A 74-year-old woman with a history of primary hyperparathyroidism, thyroid nodules, atrial fibrillation and pacemaker placement for sick sinus syndrome presented with fatigue, constipation and persistent lower extremity oedema. ^[4] A 67-year-old woman suffered from severe sick sinus syndrome, and she was fitted with a ventricular demand inhibited pacemaker. ^[5] A 58-year-old woman complained of general fatigue and was diagnosed with sick sinus syndrome (SSS) by ambulatory electrocardiogram, which demonstrated sinus arrest at midnight and paroxysmal atrial fibrillation (AF) at nighttime. ^[6] The patient was an 81-year-old woman with a history of mitral valve repair, atrial fibrillation, and sick sinus syndrome status post–His bundle pacemaker (HBP) implant using a Select Secure lead (model 3830, 69 cm; Medtronic Inc. ^[7] Here, we report a 47-year-old woman with only cavernous sinus syndrome as first manifestation. ^[8] An 83-year-old woman with a past medical history of myelodysplastic syndrome, sick sinus syndrome with a pacemaker (PM), and coronary artery disease was diagnosed at an outside hospital with methicillin-resistant Staphylococcus aureus (MRSA) endocarditis from an infected peripheral line. ^[9]

year old female

Results A 72 year-old female presented with a right-sided cavernous sinus syndrome on a background of a myeloproliferative disorder. ^[1] We report a case of a 54-year-old female, known symptomatic sick sinus syndrome, who had undergone a single-chamber pacemaker (VVI, St. ^[2] Here, we describe the step-wise management approach in a 53-year-old female with a giant, left CCA presenting with headache and cavernous sinus syndrome who was ultimately successfully treated with endovascular coiling and ICA occlusion. ^[3] A 79-year old female with a history of obesity, CABG, ischemic cardiomyopathy, atrial fibrillation and sick sinus syndrome (s/p dual chamber pacemaker) presented to. ^[4] We present a challenging case of a 75-year-old female with a history of paroxysmal atrial fibrillation (PAF) and symptomatic sick sinus syndrome (SSS) who presented for a dual chamber pacemaker implantation and was found to have persistent left superior vena cava and absent right superior vena cava with stenosis of the left subclavian vein. ^[5] Case presentation A 78-year-old female patient had history of chronic rheumatic heart disease and symptomatic sick sinus syndrome. ^[6]

dual chamber pacemaker

Methods: A total of 289 patients who were implanted with a dual-chamber pacemaker due to complete atrioventricular block or symptomatic sick sinus syndrome (SSS) and had no previous history of atrial fibrillation were included in the present study. ^[1] I was diagnosed with a sick sinus syndrome and implanted with a dual-chamber pacemaker. ^[2] A 72-year-old man presented for routine dual chamber pacemaker interrogation 13 years following insertion for sick sinus syndrome. ^[3] An 80-year-old man with a history of dilated hypertrophic cardiomyopathy received a dual-chamber pacemaker for sick sinus syndrome and atrioventricular block in February 2010. ^[4] The man, who had dilated hypertrophic cardiomyopathy, hypertension and chronic heart failure, received a dual-chamber pacemaker for sick sinus syndrome and atrioventricular block in February 2010. ^[5] 57 yo M, PMH of CKD, HIV (controlled on HAART), sick sinus syndrome s/p dual-chamber pacemaker, mitral valve prolapse s/p. ^[6]

degree atrioventricular block

The indications for pacemaker implantation were symptomatic third-degree atrioventricular block in one and sick sinus syndrome in eight patients. ^[1] Advanced second-degree and third-degree atrioventricular blocks, sick sinus syndrome, persistent atrial standstill, and vasovagal syncope are the most common rhythm disturbances that require pacing to either alleviate clinical signs or prolong survival. ^[2] Pacemakers effectively reduce symptoms of bradycardia, prevent syncope in patients with sick sinus syndrome, and reduce mortality in high-degree atrioventricular block. ^[3] Among them, 35 cases were diagnosed as sick sinus syndrome (SSS) and 9 cases were diagnosed as Ⅱ or Ⅲ degree atrioventricular block (AVB) before operation. ^[4]

year old man

The first patient (77-year-old man) was implanted an LTP for sick sinus syndrome. ^[1] We report a case of a 72-year-old man with a previous history of sick sinus syndrome and stable angina pectoris. ^[2] We present a case of a 76-year-old man with sick sinus syndrome, in whom the KODEX-EPD system was successfully used to guide an implant of a dual-chamber pacemaker with HBP. ^[3]

permanent pacemaker implantation

He had permanent pacemaker implantation for sick sinus syndrome three weeks prior. ^[1] He had a medical history including, paroxysmal atrial fibrillation (PAf), sick sinus syndrome after permanent pacemaker implantation, and transient ischemic attack. ^[2] His father and his younger brother had permanent pacemaker implantation because of sick sinus syndrome. ^[3]

superior vena cava

Left superior vena cava draining into the left atrium in the absence of coronary sinus is an anomaly that can appear in heterotaxy syndrome and unroofed coronary sinus syndrome. ^[1]

Sick Sinus Syndrome

Pacing indications were sick sinus syndrome (n = 3) and atrio-ventricular block (n = 11). ^[1] We report a case of acute ischemic nephropathy in a patient with severe renal artery stenosis and bradycardia due to sick sinus syndrome. ^[2] Case presentation A 94-year-old woman was diagnosed with sick sinus syndrome and underwent a dual-chamber permanent pacemaker implantation. ^[3] The indication for temporary pacing was sick sinus syndrome in 135 (40. ^[4] Discussion : in our case, taking into account the secretion of catecholamines, sick sinus syndrome, a high risk of bleeding, and tumor fragmentation, the first stage was to achieve the regression of the tumor by means of embolization of heart tumor suppliying afferent with coils. ^[5] We report a case of a 54-year-old female, known symptomatic sick sinus syndrome, who had undergone a single-chamber pacemaker (VVI, St. ^[6] However, on day 76, he presented with syncope secondary to sick sinus syndrome. ^[7] He was implanted with a permanent pacemaker two months prior due to sick sinus syndrome. ^[8] Here, we present a rare case of severe hypothyroidism patient accompanied by sick sinus syndrome (SSS) as a cardiovascular manifestation needing cardiac pacing and severe mental disorders. ^[9] Sick sinus syndrome (SSS) is a set of diseases with abnormal cardiac pacing, which manifests as diverse cardiac arrhythmias, especially bradycardia. ^[10] Sinus node dysfunction, previously known as sick sinus syndrome, describes disorders related to abnormal conduction and propagation of electrical impulses at the sinoatrial node. ^[11] Aims The aim of this study was to use human genetics to investigate the pathogenesis of sick sinus syndrome (SSS) and the role of risk factors in its development. ^[12] 4 s, which was repeatedly observed 6 times, indicating sick sinus syndrome (SSS). ^[13] This occurred repeatedly, and a diagnosis of sick sinus syndrome was made. ^[14] Methods: A total of 289 patients who were implanted with a dual-chamber pacemaker due to complete atrioventricular block or symptomatic sick sinus syndrome (SSS) and had no previous history of atrial fibrillation were included in the present study. ^[15] Patient concerns: A subject of the study is a 74-year-old woman with a sick sinus syndrome (SSS) and a previously implanted physiological DDD/R pacemaker. ^[16] This article presents a case of sick sinus syndrome in a master shooting athlete and discuss the potential shooting accuracy enhancement obtained by the treatment beyond beta-blockade. ^[17] Pacing was more commonly for sick sinus syndrome in women than men (54. ^[18] Patients with AHREs >6 minutes had higher proportions of sick sinus syndrome, subjects with atrial premature beat >1% on Holter monitoring, and larger left atrium (LA) size than patients with AHREs ≤6 minutes. ^[19] Sick sinus syndrome (SSS) is a disease with bradycardia or arrhythmia. ^[20] Indications for pacemaker included sick sinus syndrome in 41%, atrioventricular block in 36%, cardiac resynchronization therapy in 7%, and refractory atrial fibrillation in 15%. ^[21] He had permanent pacemaker implantation for sick sinus syndrome three weeks prior. ^[22] 53% male), of whom 3 had sick sinus syndrome (SSS) and 31 had high-grade atrioventricular conduction block (AVB). ^[23] Medical history was significant for sick sinus syndrome, hypertension, hyperlipemia and cardiac pacemaker implantation. ^[24] It may be effective in treating sick sinus syndrome (SSS). ^[25] A permanent pacemaker system (DDDR mode, ADAPTA, Medtronic) had been implanted eight years previously with the diagnosis of sick sinus syndrome. ^[26] Radiation therapy (RT) plays a pivotal role in the treatment of locally advanced EC patients, and radiation-induced heart diseases (RIHD) has become a clinically concerned problem, which may involve any structure of heart, including coronary heart disease (CHD), atrial fibrillation (AF), valvular heart disease (VHD), pericardial effusion (PE), heart failure (HF), thromboembolic disease (TED) and atrioventricular block (AVB), sick sinus syndrome (SSS), and etc; some of which may occur after 10−15 years or even more decades. ^[27] Case report 2: An 85yearold man with hypertension, chronic kidney disease, prostate cancer and sick sinus syndrome presented with acute respiratory failure due to COVID-19 pneumonia requiring mechanical ventilation. ^[28] Patients with SCN5A mutations tend to have an earlier onset of symptoms, more noticeable electrophysiological defects (such as sick sinus syndrome and AV blocks), and increased risk of major arrhythmic events especially in Asian and Caucasian populations. ^[29] Background SCN5A mutations may present with different clinical phenotypes such as Brugada syndrome, long QT3 syndrome, sick sinus syndrome, atrial fibrillation, dilated cardiomyopathy, and the least known multifocal ectopic Purkinje-related premature contractions syndrome. ^[30] The patient with partial loss of consciousness was diagnosed with DKA, and she experienced sick sinus syndrome leading to sinus arrest in the presence of regular pulsation maintained by the cardiac pacemaker. ^[31] Multivariate analysis revealed that female gender, low body mass index (BMI), non-paroxysmal AF (PAF), and sick sinus syndrome were independent and significant indicators of non-PV foci. ^[32] Permanent cardiac pacing is a recognized method of treatment of patients with sick sinus syndrome and/or atrioventricular conduction disturbances. ^[33] Sick sinus syndrome (SSS) encompasses a group of conduction disorders characterized by the inability of sinoatrial node to perform its pacemaker function. ^[34] 5%) or sick sinus syndrome (31, 18. ^[35] A 63-year-old woman with a history of hypertension, sick sinus syndrome, and Hashimoto’s thyroiditis was referred to our facility for evaluationof amass in segment 8of the liver. ^[36] 3% of first PM implants, sick sinus syndrome in 16. ^[37] He had a medical history including, paroxysmal atrial fibrillation (PAf), sick sinus syndrome after permanent pacemaker implantation, and transient ischemic attack. ^[38] For example, there is a rhythm in transcripts responsible for the two principal pacemaker mechanisms (membrane and Ca 2+ clocks), transcripts responsible for receptors and signalling pathways known to control pacemaking, transcripts from genes identified by GWAS as determinants of resting heart rate, and transcripts from genes responsible for familial and acquired sick sinus syndrome. ^[39] Here, we present a case in which there was sick sinus syndrome mimicking the DLB-related autonomic dysfunctions. ^[40] We examined the genetic background of a Chinese Han family in which some members presented with complex arrhythmias including sick sinus syndrome, progressive conduction block, atrial fibrillation, atrial standstill and Brugada syndrome. ^[41] For patients with sick sinus syndrome but normal atrioventricular conduction, atrial lead dislocation may cause excessive unnecessary ventricular pacing, resulting in nonphysiological pacing leading to heart failure. ^[42] I was diagnosed with a sick sinus syndrome and implanted with a dual-chamber pacemaker. ^[43] Background: Dextrocardia is a congenital abnormal position of the heart in which the main part of the heart is in the right chest and the long axis of the heart points to the lower right, cases with combination of dextrocardia and sick sinus syndrome are rare. ^[44] Due to a sick sinus syndrome, a double lead pacemaker was implanted and the start of antiarrhythmic medication (flecanide) was initiated, which led to freedom from arrhythmias over the last 12 months. ^[45] A 72-year-old man presented for routine dual chamber pacemaker interrogation 13 years following insertion for sick sinus syndrome. ^[46] However, amyloid sometimes invade the right ventricle (RV), and left (LA) and right (RA) atria to cause ECG changes such as sick sinus syndrome (SSS), arrhythmia, and QRS wave axis deviation. ^[47] His father and his younger brother had permanent pacemaker implantation because of sick sinus syndrome. ^[48] Introduction Sick sinus syndrome (SSS) denotes a collection of cardiac arrhythmias associated with dysfunction of the sinoatrial node that commonly lead to disorders in cardiac rhythm and conduction. ^[49] An 80-year-old man with a history of dilated hypertrophic cardiomyopathy received a dual-chamber pacemaker for sick sinus syndrome and atrioventricular block in February 2010. ^[50]

Silent Sinus Syndrome

PURPOSE The authors' aim was to review the literature in terms of the etiology of the syndrome, the frequency of Silent Sinus Syndrome (SSS) and surgical procedure, as well as to present their own experience. ^[1] A 56-year-old male patient was referred by the Opthalmology clinic with silent sinus syndrome. ^[2] We strongly agree that preoperative radiographic identification of silent sinus syndrome (SSS) and an atelectatic uncinate process are essential for minimizing the risk of orbital injury. ^[3] OBJECTIVE To describe a case of silent sinus syndrome secondary to malignancy and discuss the pertinent clinical findings. ^[4] BACKGROUND Maxillary sinus hypoplasia (MSH), associated with enophthalmos and hypoglobus in the silent sinus syndrome (SSS), is a poorly studied condition. ^[5] Chronic Maxillary Atelectasis (CMA) and Silent Sinus Syndrome (SSS) are conditions that may lie on the same clinical spectrum, since both have similarities in pathophysiology, clinical findings and treatment, although the absence of sinonasal symptoms is the main difference between them. ^[6] These findings were suggestive of ‘silent sinus syndrome’ (figures 3 and 4), causing sagging of the inferior rectus and the surrounding soft tissue, thus causing upgaze limitation and diplopia. ^[7] When associated with hypoglobus or enophthalmos, some authors use the term “silent sinus syndrome”. ^[8] Background The histopathology and microbiology associated with silent sinus syndrome (SSS) have not been well described. ^[9] The article discusses modern ideas about the silent sinus syndrome, its etiology, pathogenesis, clinical course and treatment tactics for patients, presents two clinical observations of patients with silent sinus syndrome and describes the options for surgical treatment and its results. ^[10] find that patients with silent sinus syndrome are more likely to have a history of maxillary dental disease, Editorial. ^[11] Although the checklist addresses the relevant anatomical features and existence of prior sinus surgery or disease adequately, I feel it could be further improved regarding the position of the uncinate process, specifically in the context of the silent sinus syndrome (SSS). ^[12] Symptoms and CT findings corresponded with silent sinus syndrome. ^[13] ABSTRACT Silent sinus syndrome (SSS) is a rare disorder involving bony remodeling of the maxillary sinuses, including collapse of the orbital floor. ^[14] Silent sinus syndrome, also termed imploding antrum syndrome, describes spontaneous enophthalmos arising from contracture of the maxillary sinus in the complete absence of any symptomatic sinonasal disease. ^[15] Silent sinus syndrome (SSS) describes enophthalmos and hypoglobus in association with disease of the maxillary sinus. ^[16] Background: Silent sinus syndrome (SSS) is defined as spontaneous, painless enophthalmos, hypoglobus with orbital floor resorption and maxillary sinus collapse on the ipsilateral side. ^[17] It was only after 30 years that the term silent sinus syndrome was coined by Soparkar [2]. ^[18] Silent sinus syndrome is almost a unilateral condition and is usually diagnosed in patients in their 30´s and 40´s. ^[19] Silent sinus syndrome (SSS) is a rare condition characterized by enophthalmos and hypoglobus that is generally not related to trauma or surgery. ^[20]

Cavernou Sinus Syndrome

Results A 72 year-old female presented with a right-sided cavernous sinus syndrome on a background of a myeloproliferative disorder. ^[1] Any pathology therein can cause a cavernous sinus syndrome that may present as a combination of unilateral ophthalmoplegia, autonomic dysfunction, and trigeminal neuropathy. ^[2] Here, we describe the step-wise management approach in a 53-year-old female with a giant, left CCA presenting with headache and cavernous sinus syndrome who was ultimately successfully treated with endovascular coiling and ICA occlusion. ^[3] In this case report, we describe the case of a patient presenting with a cavernous sinus syndrome secondary to a presumed meningioma. ^[4] Pathology involving the Cavernous Sinus (CS), generally referred to as Cavernous Sinus Syndrome (CSS), can arise from vascular disorders, neoplasms, infections, and non-infectious inflammatory disorders. ^[5] Cavernous sinus syndrome (CSS) is a rare condition that presents with clinical signs and symptoms due to the involvement of the cranial nerves (CN) III, IV, VI, V1, and V2. ^[6] The patient underwent urgent transsphenoidal endoscopic resection of the pituitary mass with a presurgical diagnosis of pituitary tumor with right cavernous sinus syndrome. ^[7] Conclusions We highlight the importance of including Rosai-Dorfman disease as a differential diagnosis in cavernous sinus syndrome and demonstrate a satisfactory long-term response to steroid treatment in this disease. ^[8] These symptoms were suggestive of bilateral cavernous sinus syndrome. ^[9] There is little evidence that associates cavernous sinus syndrome, with numb chin and peripheral facial paralysis syndromes. ^[10] Diffuse Infiltration of Multiple Myeloma With Initial Manifestation of Cavernous Sinus Syndrome Unveiled by 18F-FDG PET/CT[J]. ^[11] Ophthalmic manifestations of NPC usually occur due to an intracranial extension of the tumour, and they include isolated or multiple cranial nerve palsies, orbital apex or cavernous sinus syndromes and papilloedema or unilateral optic disc swelling. ^[12] If there is no suggestive history or symptoms and signs indicative of one of the five topographical syndromes: brainstem syndrome, elevated intracranial pressure syndrome, petrous apex syndrome, cavernous sinus syndrome or orbital syndrome, the patient can be classified as a case of isolated sixth nerve palsy. ^[13] In terms of the clinical manifestations of the eight patients, two were affected with Guillain-Barre syndrome (GBS), two with Cavernous sinus syndrome, one with Miller Fisher syndrome, one with both GBS and spinal cord demyelination, one with Bulbar paralysis, and one with chronic inflammatory demyelinating polyneuropathy (CIDP). ^[14] A middle-aged lady who presented with headaches and sudden onset of left cavernous sinus syndrome was found to have a left persistent primitive trigeminal artery (PPTA) on scans. ^[15] CASE REPORTS We report the management of two cases of life-threatening cavernous sinus dural arteriovenous fistula revealed by a typical cavernous sinus syndrome. ^[16] Purpose To report a Rathke’s cleft cyst (RCC) presenting as unilateral incomplete cavernous sinus syndrome and disc edema Method Case report with literature review Results We present an 18-year-old girl who has been experiencing intermittent binocular diplopia for 1 month. ^[17] Here, we report a 47-year-old woman with only cavernous sinus syndrome as first manifestation. ^[18]

Carotid Sinus Syndrome

Knowledge of these variants is critical for treatment planning of atherosclerotic carotid artery disease, management of carotid body tumours and carotid body denervation procedures in case of carotid sinus syndrome. ^[1] It is important to recognize the presentation of carotid sinus syndrome (CSS) as a potential Introduction Syncope is a common cause of hospitalization in the elderly. ^[2] Three mechanistic patterns of reflex syncope are known in such patients: carotid sinus syndrome, glossopharyngeal neuralgia syndrome, and parapharyngeal space lesions syncope syndrome. ^[3] Objectives: This article reviews the impact of asymptomatic carotid artery stenosis on management of syncope, and the role of carotid Doppler ultrasound in carotid sinus syndrome and in syncope. ^[4] On detailed investigations, he was diagnosed with carotid sinus syndrome (CSS). ^[5] Introduction: Carotid sinus syndrome (CSS) is a hypersensitivity of the carotid sinus manifested by atrioventricular sinus bradycardia or decreased arterial pressure of at least 50 mmHg. ^[6] Symptoms due to bulky disease were neck discomfort (100%), tumor bleeding (43%), tracheal obstruction (14%), and carotid sinus syndrome (28%). ^[7] This article sorts out the research literature on carotid sinus related death at home and abroad in recent years, systematically reviews the anatomic structure of the carotid sinus nerve, the clinical epidemiology of carotid sinus syndrome, and the research on the death mechanism of carotid sinus injury at home and abroad in recent years, in order to provide references for forensic pathology research and prosecution. ^[8] 5%), cardioinhibitory carotid sinus syndrome (17. ^[9] The patient was diagnosed with carotid sinus syndrome (CSS) as the free jejunum was dilated when consuming breakfast, which may have caused carotid sinus hypersensitivity and induced a medullary reflex. ^[10] Background Carotid sinus syndrome accounts for one third of patients who presents with unexplained syncope. ^[11] The results of a later interview led us to assume that she had carotid sinus syndrome. ^[12] Cardiac pacing may reduce recurrent syncope in cardioinhibitory carotid sinus syndrome. ^[13] Diagnoses: The patient was diagnosed with a well-differentiated squamous cell carcinoma of the tongue along with massive cervical lymph node metastasis and carotid sinus syndrome. ^[14] Interest has been renewed in the anatomy and physiology of the carotid sinus nerve (CSN) and its targets (carotid sinus and carotid body, CB), due to recent proposals of surgical procedures for a series of common pathologies, such as carotid sinus syndrome, hypertension, heart failure, and insulin resistance. ^[15]

Coronary Sinus Syndrome

This defect is rare and part of a wide spectrum of unroofed coronary sinus syndromes. ^[1] The ultrasonographic findings of the fetus were as follows: (1) fetal growth restriction; (2) cleft lip and palate; (3) bilateral renal hypoplasia; (4) lateral ventriculomegaly; (5) single umbilical artery; (6) absent stomach; (7) coronary sinus dilatation with persistent left superior vena cava, ventricular septal defect and unroofed coronary sinus syndrome. ^[2] Left superior vena cava draining into the left atrium in the absence of coronary sinus is an anomaly that can appear in heterotaxy syndrome and unroofed coronary sinus syndrome. ^[3] Objective To analyze the clinical features and surgical treatment of unroofed coronary sinus syndrome(UCSS) in children, and to provide evidence for the diagnosis and treatment of such diseases during the perio-perative period. ^[4] 无顶冠状静脉窦综合征(unroofed coronary sinus syndrome, UCSS)是一种较为复杂的先天性心脏畸形, 本文报道了1例Kirklin分型Ⅳ型或者解剖分型Ⅲb型的患者, 并结合手术及超声心动图资料分析了UCSS诊治的难点与要点。. ^[5]

sinus syndrome secondary

In this case report, we describe the case of a patient presenting with a cavernous sinus syndrome secondary to a presumed meningioma. ^[1] OBJECTIVE To describe a case of silent sinus syndrome secondary to malignancy and discuss the pertinent clinical findings. ^[2]

sinus syndrome presented

Case report 2: An 85yearold man with hypertension, chronic kidney disease, prostate cancer and sick sinus syndrome presented with acute respiratory failure due to COVID-19 pneumonia requiring mechanical ventilation. ^[1] A 74-year-old woman with a history of primary hyperparathyroidism, thyroid nodules, atrial fibrillation and pacemaker placement for sick sinus syndrome presented with fatigue, constipation and persistent lower extremity oedema. ^[2]