Introduction to Receptor Endocytosis
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Receptor Endocytosis sentence examples within Ampa Receptor Endocytosis
PKMζ maintains synaptic potentiation by preventing AMPA receptor endocytosis and promoting stabilisation of dendritic spine growth.
PKMζ maintains synaptic potentiation by preventing AMPA receptor endocytosis and promoting stabilisation of dendritic spine growth.
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Arc protein is localized to excitatory synapses, where it controls AMPA receptor endocytosis, and to the nucleus, where it associates with Tip60, a subunit of a chromatin modifying complex.
Arc protein is localized to excitatory synapses, where it controls AMPA receptor endocytosis, and to the nucleus, where it associates with Tip60, a subunit of a chromatin modifying complex.
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Receptor Endocytosis sentence examples within Mediated Receptor Endocytosis
The authors propose utilizing agonist-mediated receptor endocytosis to downregulate CRFR2.
The authors propose utilizing agonist-mediated receptor endocytosis to downregulate CRFR2.
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The resultant information will greatly facilitate the study of clathrin- and caveolae-mediated receptor endocytosis and signaling process and also our home-built dual-color T-Rex STED microscope with this line-switching imaging mode provides a precise and convenient way to study subcellular-scale protein interactions.
The resultant information will greatly facilitate the study of clathrin- and caveolae-mediated receptor endocytosis and signaling process and also our home-built dual-color T-Rex STED microscope with this line-switching imaging mode provides a precise and convenient way to study subcellular-scale protein interactions.
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Receptor Endocytosis sentence examples within Metabolic Receptor Endocytosis
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
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These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
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Receptor Endocytosis sentence examples within Regulate Receptor Endocytosis
, S799T) is part of a motif that regulates receptor endocytosis; however, due to the conservative nature of serine to threonine substitution, it is unclear if this mutation would alter CSF3R function.
, S799T) is part of a motif that regulates receptor endocytosis; however, due to the conservative nature of serine to threonine substitution, it is unclear if this mutation would alter CSF3R function.
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show that mitotic proteins and the SHP2-MAPK pathway regulate receptor endocytosis and insulin signaling feedback, identifying a potential role for SHP2 inhibitors to treat diabetes.
show that mitotic proteins and the SHP2-MAPK pathway regulate receptor endocytosis and insulin signaling feedback, identifying a potential role for SHP2 inhibitors to treat diabetes.
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Receptor Endocytosis sentence examples within Wnt Receptor Endocytosis
We review recent investigations that uncover new cell physiological effects through the regulation of Wnt receptor endocytosis, Wnt-induced stabilization of proteins (Wnt-STOP), macropinocytosis, increase in lysosomal activity, and metabolic changes.
We review recent investigations that uncover new cell physiological effects through the regulation of Wnt receptor endocytosis, Wnt-induced stabilization of proteins (Wnt-STOP), macropinocytosis, increase in lysosomal activity, and metabolic changes.
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Impaired Wnt receptor endocytosis due to loss of the ubiquitin ligase RNF43 gives rise to Wnt-hypersensitive tumours that are susceptible to anti-Wnt-based therapy.
Impaired Wnt receptor endocytosis due to loss of the ubiquitin ligase RNF43 gives rise to Wnt-hypersensitive tumours that are susceptible to anti-Wnt-based therapy.
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10.1007/978-1-0716-1044-2_3
This has been widely exploited to study constitutive endocytosis or ligand-induced receptor endocytosis.
This has been widely exploited to study constitutive endocytosis or ligand-induced receptor endocytosis.
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10.1146/annurev-cellbio-120319-023657
We review recent investigations that uncover new cell physiological effects through the regulation of Wnt receptor endocytosis, Wnt-induced stabilization of proteins (Wnt-STOP), macropinocytosis, increase in lysosomal activity, and metabolic changes.
We review recent investigations that uncover new cell physiological effects through the regulation of Wnt receptor endocytosis, Wnt-induced stabilization of proteins (Wnt-STOP), macropinocytosis, increase in lysosomal activity, and metabolic changes.
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10.1016/j.ejphar.2021.174203
We mainly focused on the characteristics, early evaluation of GLP-1 receptor endocytosis and effects of optimization for endocytosis on druggability.
We mainly focused on the characteristics, early evaluation of GLP-1 receptor endocytosis and effects of optimization for endocytosis on druggability.
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10.1073/pnas.2011023118
Previous studies show that β-arrestin signaling and receptor endocytosis are modulated by the plasma membrane phosphoinositide lipid phosphatidylinositol-(4, 5)-bisphosphate (PI(4,5)P2).
Previous studies show that β-arrestin signaling and receptor endocytosis are modulated by the plasma membrane phosphoinositide lipid phosphatidylinositol-(4, 5)-bisphosphate (PI(4,5)P2).
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10.3390/biom11081088
Depending on the multivalent, engineered ligand used, the clustering of FGFR1 into diverse supramolecular complexes enhances the efficiency and modifies the mechanism of receptor endocytosis, alters FGFR1 lifetime and modifies receptor signaling, ultimately determining cell fate.
Depending on the multivalent, engineered ligand used, the clustering of FGFR1 into diverse supramolecular complexes enhances the efficiency and modifies the mechanism of receptor endocytosis, alters FGFR1 lifetime and modifies receptor signaling, ultimately determining cell fate.
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10.1038/s41467-020-20624-z
We demonstrate that GABARAP functions to stabilize GABA A Rs via promoting its trafficking pathway instead of blocking receptor endocytosis.
We demonstrate that GABARAP functions to stabilize GABA A Rs via promoting its trafficking pathway instead of blocking receptor endocytosis.
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10.1016/j.freeradbiomed.2021.07.001
The small GTPase Arf6 regulates many cellular processes, including cytoskeletal remodeling, receptor endocytosis, and pathogen phagocytosis.
The small GTPase Arf6 regulates many cellular processes, including cytoskeletal remodeling, receptor endocytosis, and pathogen phagocytosis.
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10.1101/2021.02.03.429614
In VSMCs, LRRC8C knockdown (siRNA) recapitulated the effects of siLRRC8A, inhibiting TNFα-induced extracellular and endosomal O2•- production, receptor endocytosis, NF-κB activation, and proliferation.
In VSMCs, LRRC8C knockdown (siRNA) recapitulated the effects of siLRRC8A, inhibiting TNFα-induced extracellular and endosomal O2•- production, receptor endocytosis, NF-κB activation, and proliferation.
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10.1038/s41598-021-90271-x
The transmembrane aminopeptidase CD13 is highly expressed in cells of the myeloid lineage, regulates dynamin-dependent receptor endocytosis and recycling and is a necessary component of actin cytoskeletal organization.
The transmembrane aminopeptidase CD13 is highly expressed in cells of the myeloid lineage, regulates dynamin-dependent receptor endocytosis and recycling and is a necessary component of actin cytoskeletal organization.
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10.1002/chem.202100328
In the RN46A immortalized serotonergic neuronal cell line, extracellular serotonin enhances transferrin receptor endocytosis, even in the presence of broad‐spectrum serotonin receptor and transporter inhibitors.
In the RN46A immortalized serotonergic neuronal cell line, extracellular serotonin enhances transferrin receptor endocytosis, even in the presence of broad‐spectrum serotonin receptor and transporter inhibitors.
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10.3389/fnsys.2021.698240
The authors propose utilizing agonist-mediated receptor endocytosis to downregulate CRFR2.
The authors propose utilizing agonist-mediated receptor endocytosis to downregulate CRFR2.
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10.1038/s41440-021-00669-3
The sorting nexin 1 (SNX) family is composed of a diverse group of cytoplasmic and membrane-associated proteins that are involved in various aspects of receptor endocytosis and trafficking through endosomes [2–4].
The sorting nexin 1 (SNX) family is composed of a diverse group of cytoplasmic and membrane-associated proteins that are involved in various aspects of receptor endocytosis and trafficking through endosomes [2–4].
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10.1186/s12967-021-02807-4
Co-immunoprecipitation was performed using the FGFR4 antibody to identify the endosomal complex formation and receptor endocytosis.
Co-immunoprecipitation was performed using the FGFR4 antibody to identify the endosomal complex formation and receptor endocytosis.
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10.1002/adbi.202101008
Despite intensive studies on the TGFβ pathway over the last decades, the necessity of TGFβ receptor endocytosis for downstream TGFβ signaling responses is a subject of debate.
Despite intensive studies on the TGFβ pathway over the last decades, the necessity of TGFβ receptor endocytosis for downstream TGFβ signaling responses is a subject of debate.
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10.3390/ph14070674
This review provides a deeper understanding of the pertinent principles of receptor endocytosis for the currently approved ADCs.
This review provides a deeper understanding of the pertinent principles of receptor endocytosis for the currently approved ADCs.
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10.1124/molpharm.121.000363
Cmpd-6 potentiates β-arrestin1– but not Gs-protein–mediated high-affinity binding of carvedilol at the β2AR and β-arrestin–mediated cellular functions in response to carvedilol including extracellular signal-regulated kinase phosphorylation, receptor endocytosis, and trafficking into lysosomes.
Cmpd-6 potentiates β-arrestin1– but not Gs-protein–mediated high-affinity binding of carvedilol at the β2AR and β-arrestin–mediated cellular functions in response to carvedilol including extracellular signal-regulated kinase phosphorylation, receptor endocytosis, and trafficking into lysosomes.
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10.1142/s1793545821500176
The resultant information will greatly facilitate the study of clathrin- and caveolae-mediated receptor endocytosis and signaling process and also our home-built dual-color T-Rex STED microscope with this line-switching imaging mode provides a precise and convenient way to study subcellular-scale protein interactions.
The resultant information will greatly facilitate the study of clathrin- and caveolae-mediated receptor endocytosis and signaling process and also our home-built dual-color T-Rex STED microscope with this line-switching imaging mode provides a precise and convenient way to study subcellular-scale protein interactions.
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10.1101/2021.01.18.427180
Mechanistically, direct binding of KAMPs to cell surface TLR2 and TLR co-receptors CD14 and MD-2 not only blocks their bacterial ligand docking sites, but also reduces cell surface availability of TLR2 and TLR4 through promotion of receptor endocytosis.
Mechanistically, direct binding of KAMPs to cell surface TLR2 and TLR co-receptors CD14 and MD-2 not only blocks their bacterial ligand docking sites, but also reduces cell surface availability of TLR2 and TLR4 through promotion of receptor endocytosis.
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10.1515/revneuro-2020-0105
PKMζ maintains synaptic potentiation by preventing AMPA receptor endocytosis and promoting stabilisation of dendritic spine growth.
PKMζ maintains synaptic potentiation by preventing AMPA receptor endocytosis and promoting stabilisation of dendritic spine growth.
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10.1016/j.bbamcr.2021.119124
It is unclear whether autophagy participates in the receptor endocytosis and modulates Shh signaling transduction.
It is unclear whether autophagy participates in the receptor endocytosis and modulates Shh signaling transduction.
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10.1093/ijnp/pyab011
Brain region–specific microinjections of the Tat-GluA23Y peptide into the nucleus accumbens, medial prefrontal cortex, or central or basolateral amygdala demonstrated that disruption of glutamate α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor endocytosis in the central amygdala also potentiated the LI effect.
Brain region–specific microinjections of the Tat-GluA23Y peptide into the nucleus accumbens, medial prefrontal cortex, or central or basolateral amygdala demonstrated that disruption of glutamate α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor endocytosis in the central amygdala also potentiated the LI effect.
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10.1113/JP281577
LRRC8C knockdown inhibited TNFα‐induced O2•− production, receptor endocytosis, nuclear factor‐κB (NF‐κB) activation and proliferation while LRRC8D knockdown enhanced NF‐κB activation.
LRRC8C knockdown inhibited TNFα‐induced O2•− production, receptor endocytosis, nuclear factor‐κB (NF‐κB) activation and proliferation while LRRC8D knockdown enhanced NF‐κB activation.
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10.48091/gsr.v1i2.19
Their findings suggest that GPR40 induces NMDA receptor endocytosis via direct interaction with NR2A and NR2B subunits of postsynaptic NMDA receptors.
Their findings suggest that GPR40 induces NMDA receptor endocytosis via direct interaction with NR2A and NR2B subunits of postsynaptic NMDA receptors.
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10.1016/J.COEMR.2020.09.002
GRK-mediated phosphorylation of GPCRs promotes arrestin binding to uncouple the receptor from G protein, a process called desensitization, and for many GPCRs, arrestin binding also promotes receptor endocytosis and intracellular signaling.
GRK-mediated phosphorylation of GPCRs promotes arrestin binding to uncouple the receptor from G protein, a process called desensitization, and for many GPCRs, arrestin binding also promotes receptor endocytosis and intracellular signaling.
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10.1073/pnas.2025846118
Our data reveal that GPCR-promoted activation of Gq at the plasma membrane induces its translocation to endosomes independently of β-arrestin engagement and receptor endocytosis.
Our data reveal that GPCR-promoted activation of Gq at the plasma membrane induces its translocation to endosomes independently of β-arrestin engagement and receptor endocytosis.
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10.3389/fcell.2021.733688
However, there have been conflicting data on the role of death receptor endocytosis in apoptotic TRAIL signaling and possible cell type-specific differences in TRAIL signaling have been proposed.
However, there have been conflicting data on the role of death receptor endocytosis in apoptotic TRAIL signaling and possible cell type-specific differences in TRAIL signaling have been proposed.
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10.1101/833988
Arc protein is localized to excitatory synapses, where it controls AMPA receptor endocytosis, and to the nucleus, where it associates with Tip60, a subunit of a chromatin modifying complex.
Arc protein is localized to excitatory synapses, where it controls AMPA receptor endocytosis, and to the nucleus, where it associates with Tip60, a subunit of a chromatin modifying complex.
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10.1016/j.bioorg.2018.12.009
Lately, there has been a growing interest in its cytosolic C-terminal SAM domain (EphA2-SAM) as it engages protein modulators of receptor endocytosis and stability.
Lately, there has been a growing interest in its cytosolic C-terminal SAM domain (EphA2-SAM) as it engages protein modulators of receptor endocytosis and stability.
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10.1038/s41598-019-47478-w
Other GPCRs showed agonist-induced GPCR/14-3-3 interaction signal increases that occur later than agonist-induced GPCR/β-arrestin interaction signals, indicating that GPCR/14-3-3 interaction occurred after receptor endocytosis.
Other GPCRs showed agonist-induced GPCR/14-3-3 interaction signal increases that occur later than agonist-induced GPCR/β-arrestin interaction signals, indicating that GPCR/14-3-3 interaction occurred after receptor endocytosis.
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10.1101/668434
Furthermore, changes in the AMPA receptor endocytosis pathway in both protein synthesis and protein phosphorylation upon LTD were identified, whereby Intersectin-1 was validated as a vital player in this pathway.
Furthermore, changes in the AMPA receptor endocytosis pathway in both protein synthesis and protein phosphorylation upon LTD were identified, whereby Intersectin-1 was validated as a vital player in this pathway.
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10.32867/inakidney.v2i3.31
NGAL is also a protease-resistant polypetide; it is released from the distal tubule, secreted to the urine or returned to the plasma (back leak), freely filtered in the glomerulus, reabsorbed in the proximal tubule through the megalin receptor endocytosis or secreted to urine.
NGAL is also a protease-resistant polypetide; it is released from the distal tubule, secreted to the urine or returned to the plasma (back leak), freely filtered in the glomerulus, reabsorbed in the proximal tubule through the megalin receptor endocytosis or secreted to urine.
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10.1016/j.jbior.2018.09.003
LGALS3 also modulates T cell receptor endocytosis and impairs interferon mediated chemokine production by binding glycosylated interferon.
LGALS3 also modulates T cell receptor endocytosis and impairs interferon mediated chemokine production by binding glycosylated interferon.
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10.1016/j.abb.2019.07.004
Hence, the present study revealed that the APT-DOX-PLGA-PVP NPs improved the therapeutic efficiency through the nucleolin receptor endocytosis targeted drug release.
Hence, the present study revealed that the APT-DOX-PLGA-PVP NPs improved the therapeutic efficiency through the nucleolin receptor endocytosis targeted drug release.
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10.1038/s12276-019-0284-5
During ligand-mediated receptor endocytosis, the small GTPase Rab5 functions in vesicle fusion and trafficking.
During ligand-mediated receptor endocytosis, the small GTPase Rab5 functions in vesicle fusion and trafficking.
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10.1124/mol.118.112987
Although retrieval mechanisms, such as receptor endocytosis, have been extensively investigated, fewer studies have explored mechanisms of receptor maturation and delivery to the plasma membrane.
Although retrieval mechanisms, such as receptor endocytosis, have been extensively investigated, fewer studies have explored mechanisms of receptor maturation and delivery to the plasma membrane.
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10.1177/1073858419844497
A recent study by Awasthi and others (2019) shows that Syt3 Ca2+-dependently modulates the postsynaptic receptor endocytosis and thereby promotes the long-term depression (LTD) and the decay of long-term potentiation (LTP).
A recent study by Awasthi and others (2019) shows that Syt3 Ca2+-dependently modulates the postsynaptic receptor endocytosis and thereby promotes the long-term depression (LTD) and the decay of long-term potentiation (LTP).
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10.1021/acschemneuro.9b00659
Although the role of membrane lipids has been extensively studied in the context of the function, organization and dynamics of GPCRs, their role in receptor endocytosis remains largely unexplored.
Although the role of membrane lipids has been extensively studied in the context of the function, organization and dynamics of GPCRs, their role in receptor endocytosis remains largely unexplored.
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10.1016/j.metabol.2019.153990
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
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10.1152/ajpheart.00741.2018
We hypothesized that ASK1 is required for TNFα receptor endocytosis and subsequent inflammatory TNFα signaling.
We hypothesized that ASK1 is required for TNFα receptor endocytosis and subsequent inflammatory TNFα signaling.
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10.1016/j.jaci.2018.05.003
R321 inhibits only the early phase of extracellular signal‐regulated kinase 1/2 activation and not the late phase generally associated with &bgr;‐arrestin recruitment and receptor endocytosis, promoting CCR3 internalization and degradation.
R321 inhibits only the early phase of extracellular signal‐regulated kinase 1/2 activation and not the late phase generally associated with &bgr;‐arrestin recruitment and receptor endocytosis, promoting CCR3 internalization and degradation.
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10.1080/19420889.2019.1586048
However, whether and how AMPA receptor endocytosis and LTD at specific synapses are causally linked to learning and memory in vivo remains unclear.
However, whether and how AMPA receptor endocytosis and LTD at specific synapses are causally linked to learning and memory in vivo remains unclear.
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10.1016/j.cellsig.2019.03.022
However, the intrinsic mechanisms of receptor endocytosis and contemporaneous intracellular signaling are not well understood.
However, the intrinsic mechanisms of receptor endocytosis and contemporaneous intracellular signaling are not well understood.
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10.1016/j.bbrc.2019.03.204
In vitro studies attribute cooperation to forced receptor endocytosis/degradation and inhibition of both DNA synthesis and cell migration.
In vitro studies attribute cooperation to forced receptor endocytosis/degradation and inhibition of both DNA synthesis and cell migration.
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10.1074/jbc.RA119.007485
We discover that C5apep acts as a full agonist for Gαi coupling as measured by cAMP response and extracellular signal-regulated kinase 1/2 phosphorylation, but it displays partial agonism for βarr recruitment and receptor endocytosis.
We discover that C5apep acts as a full agonist for Gαi coupling as measured by cAMP response and extracellular signal-regulated kinase 1/2 phosphorylation, but it displays partial agonism for βarr recruitment and receptor endocytosis.
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10.1091/mbc.E18-08-0512
Activation of the epidermal growth factor (EGF) receptor (EGFR) at the cell surface initiates signaling through the RAS-RAF-MAPK/ERK1/2 pathway and receptor endocytosis.
Activation of the epidermal growth factor (EGF) receptor (EGFR) at the cell surface initiates signaling through the RAS-RAF-MAPK/ERK1/2 pathway and receptor endocytosis.
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10.1038/s41375-019-0688-1
, S799T) is part of a motif that regulates receptor endocytosis; however, due to the conservative nature of serine to threonine substitution, it is unclear if this mutation would alter CSF3R function.
, S799T) is part of a motif that regulates receptor endocytosis; however, due to the conservative nature of serine to threonine substitution, it is unclear if this mutation would alter CSF3R function.
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10.1101/748574
Impaired Wnt receptor endocytosis due to loss of the ubiquitin ligase RNF43 gives rise to Wnt-hypersensitive tumours that are susceptible to anti-Wnt-based therapy.
Impaired Wnt receptor endocytosis due to loss of the ubiquitin ligase RNF43 gives rise to Wnt-hypersensitive tumours that are susceptible to anti-Wnt-based therapy.
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10.1101/836437
Receptor endocytosis is important for signal activation and transduction.
Receptor endocytosis is important for signal activation and transduction.
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10.1111/adb.12793
We also observed that the inhibitory effect of the UCS retrieval‐extinction procedure is mediated by regulation of AMPA receptor endocytosis in the BLA.
We also observed that the inhibitory effect of the UCS retrieval‐extinction procedure is mediated by regulation of AMPA receptor endocytosis in the BLA.
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10.1016/j.cellsig.2019.03.012
Our screen has identified potential PCTAIRE-1 substrates (AP2-Associated Kinase 1 (AAK1), dynamin 1, and synaptojanin 1) in brain that have been shown to regulate crucial steps of receptor endocytosis, and are involved in control of neuronal synaptic transmission.
Our screen has identified potential PCTAIRE-1 substrates (AP2-Associated Kinase 1 (AAK1), dynamin 1, and synaptojanin 1) in brain that have been shown to regulate crucial steps of receptor endocytosis, and are involved in control of neuronal synaptic transmission.
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10.1111/tra.12628
This review explores what is known about receptor sorting at the plasma membrane, protein partners that control receptor endocytosis, and the ways in which receptor sorting at the plasma membrane regulates downstream trafficking and signaling.
This review explores what is known about receptor sorting at the plasma membrane, protein partners that control receptor endocytosis, and the ways in which receptor sorting at the plasma membrane regulates downstream trafficking and signaling.
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10.1038/s41467-019-09318-3
show that mitotic proteins and the SHP2-MAPK pathway regulate receptor endocytosis and insulin signaling feedback, identifying a potential role for SHP2 inhibitors to treat diabetes.
show that mitotic proteins and the SHP2-MAPK pathway regulate receptor endocytosis and insulin signaling feedback, identifying a potential role for SHP2 inhibitors to treat diabetes.
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10.1096/fj.201800936R
SUMOylation of the M1 mAChR played an important role in increasing the ligand‐binding affinity to M1 mAChR, signaling efficiencies, and receptor endocytosis.
SUMOylation of the M1 mAChR played an important role in increasing the ligand‐binding affinity to M1 mAChR, signaling efficiencies, and receptor endocytosis.
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10.1101/793000
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
These studies suggest that C2CD5 represents a new protein regulated by metabolic cues and involved in metabolic receptor endocytosis.
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10.1158/1538-7445.AM2019-4543
The pSTY data shows that RXDX106 deregulates phosphorylation of proteins involved in PI3K signaling, receptor endocytosis and cell migration pathways in H1299 cells.
The pSTY data shows that RXDX106 deregulates phosphorylation of proteins involved in PI3K signaling, receptor endocytosis and cell migration pathways in H1299 cells.
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10.1038/s41563-019-0367-7
Localized epidermal growth factor presentation enables dynamic regulation over proliferation, intracellular mitogen-activated protein kinase signalling and subcellularly resolved receptor endocytosis.
Localized epidermal growth factor presentation enables dynamic regulation over proliferation, intracellular mitogen-activated protein kinase signalling and subcellularly resolved receptor endocytosis.
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10.1016/j.celrep.2019.01.049
Phosphorylation of heptahelical receptors is thought to regulate G protein signaling, receptor endocytosis, and non-canonical signaling via recruitment of β-arrestins.
Phosphorylation of heptahelical receptors is thought to regulate G protein signaling, receptor endocytosis, and non-canonical signaling via recruitment of β-arrestins.
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Keywords related to Receptor
Receptor Homolog
Receptor Progesterone Receptor
Receptor T
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Receptor Induced
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Receptor Positive Her2 Negative
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Receptor Plasticity
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Receptor Cck1r
Receptor Neuron
Receptor Transduced
Receptor X2
Receptor Selectivity
Receptor 21
Receptor Related
Receptor Drug
Receptor Nkg2d
Receptor Reveals
Receptor Isoforms
Receptor Complex
Receptor 4 Signaling
Receptor Gamma
Receptor Imaging
Receptor Superfamily
Receptor Splice
Receptor Transgenic
Receptor Engineered T
Receptor Potential
Receptor Downregulator
Receptor Upregulation
Receptor Mediated Mechanisms
Receptor Mediating
Receptor Availability
Receptor Hace2
Receptor Coactivators
Receptor Downregulation
Receptor Mediated Calcium
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Receptor Antagonists Inverse
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Receptor Ir
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Keywords related to Endocytosis
Dependent Endocytosis
Caveolae Mediated Endocytosis
Clathrin Independent Endocytosis
Regulates Endocytosis
Egfr Endocytosis
Integrin Endocytosis
Cellular Endocytosis
Receptor Mediated Endocytosis
Inhibits Endocytosis
Presynaptic Endocytosis
Vesicle Endocytosis
Junction Endocytosis
Bulk Endocytosis
Apical Endocytosis
Clathrin Mediated Endocytosis
Mediated Endocytosis
Promoting Endocytosis
Gpcr Endocytosis
Cell Endocytosis
Clathrin Dependent Endocytosis
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Receptor Endocytosis
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