Introduction to Chronic Postoperative Hypoparathyroidism

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Chronic Postoperative Hypoparathyroidism sentence examples

10.14341/OMET12729

The chronic postoperative hypoparathyroidism accounts approximately 75% of patients; the genetic, autoimmune or idiopathic forms are significantly less common.

Daily calcium profile in diagnosis of hypo- and hypercalcemia in patients with chronic hypoparathyroidism. clinical case series

10.14412/2074-2711-2021-3-82-87

We present a case report of a woman with convulsions, which developed due to dissociative (conversion motor) disorder, with chronic postoperative hypoparathyroidism, receiving standard treatment and having normo- and hypercalcemia.

Differential diagnosis of convulsions in the structure of dissociative (conversion motor) disorder and chronic postoperative hypoparathyroidism: case report and literature review

10.1016/j.bone.2019.06.025

One patient had chronic postoperative hypoparathyroidism and one patient remained normocalcemic 4 years after surgery.

Hyperparathyroidism and parathyroidectomy in X-linked hypophosphatemia patients.

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10.1210/JS.2019-SUN-526

One patient had chronic postoperative hypoparathyroidism, and one patient remains normocalcemic 4 years after surgery.

SUN-526 Prevalence of Hyperparathyroidism in X-Linked Hypophosphatemia

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An Overview of Chronic Postoperative Hypoparathyroidism

Chronic Postoperative Hypoparathyroidism
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What is/are Postoperative Hypoparathyroidism?

Postoperative Hypoparathyroidism - Postoperative hypoparathyroidism is still an urgent problem, as it requires additional treatment and prolongs hospital-stay. [1] OBJECTIVE to determine the usefulness of high urine output as a low-cost early predictor for postoperative hypoparathyroidism. [2] Notwithstanding the actual surgical skill, postoperative hypoparathyroidism still represents its most frequent complication. [3] We present a case report of a 38-year-old woman with postoperative hypoparathyroidism treated with rhPTH who subsequently developed calciphylactic lesions on her abdomen. [4] Advances in intraoperative technology to optimize the vascularization of the parathyroid glands can help to predict and prevent patients from a postoperative hypoparathyroidism. [5] The postoperative hypoparathyroidism rate in the CNs group was significantly lower than that in the control group (P<0. [6] The procedure is generally safe, although a minority of cases could present complications as postoperative hypoparathyroidism or recurrent laryngeal nerve dysfunction [1]. [7] We aimed to investigate the prevalence of postoperative hypoparathyroidism (PoH), the relevant factors, and predictors of transient or permanent hypoparathyroidism. [8] The clinical characteristics of patients with postoperative hypoparathyroidism who recover parathyroid function more than 12 months after surgery have not been studied. [9] In the case of surgery, postoperative hypoparathyroidism was significantly more frequent following more extensive surgical procedures. [10] In this study, we aimed to evaluate the relationship between the recovery time of parathyroid gland function and patient characteristics, preoperative and postoperative electrolyte changes, and intraoperative parathyroid findings in patients with postoperative hypoparathyroidism. [11] BACKGROUND To investigate how number of autotransplanted parathyroid glands (PGs) affects the incidence of postoperative hypoparathyroidism and the recovery of parathyroid function. [12] Background Definitions of postoperative hypoparathyroidism (hypoPT) have never reached consent until the American Thyroid Association (ATA) statement was released, with new characteristics and challenges. [13] However, this modality did not result in the reduction of postoperative hypoparathyroidism or hypocalcemia risk. [14] This study aimed to explore the relationship between parathyroid gland autotransplantation and postoperative hypoparathyroidism. [15] However, as predicted, LT completely eliminated the risk of postoperative hypoparathyroidism. [16] We wished to investigate the association between number of parathyroids identified intraoperatively during TT, and incidence of incidental parathyroidectomy, and postoperative hypoparathyroidism. [17] BACKGROUND Postoperative hypoparathyroidism (PH) is the most common complication after total thyroidectomy. [18] RESULTS The incidence of postoperative hypoparathyroidism (PTH < 15pg/mL) was significantly lower in the NIRAF group during the hospitalization (88 patients: 33. [19] This method could be a cost- and labor-effective alternative to frozen sections to reduce the incidence of postoperative hypoparathyroidism and improve the outcome of primary hyperparathyroidism in low-resource areas. [20] Introduction: Postoperative hypoparathyroidism is one of the most common complications after total thyroidectomy. [21] The authors hypothesized that (1) for CEC patients undergoing PLE with thyroid lobectomy, definitive chemoradiotherapy (CRT) would have some effect on postoperative hypothyroidism, and (2) PLTE may also have some effect on postoperative hypoparathyroidism. [22] The aim of this paper was to determine whether the rate of postoperative hypoparathyroidism (HPT) is influenced by whether surgery is staged. [23] Postoperative hypoparathyroidism was defined as low parathyroid hormone (PTH) levels (<1. [24] Postoperative hypoparathyroidism/hypocalcemia is the most prevalent complication following thyroid and parathyroid surgeries. [25] When compared with other PHPT patients, PC patients were more frequently male and had higher preoperative blood calcium and PTH and lower phosphate levels, larger and heavier parathyroids excised, lower postoperative calcium, and a higher rate of postoperative hypoparathyroidism. [26] Postoperative hypoparathyroidism requiring long-term calcium and 1alpha(OH) D3 treatment was reported in 5% of patients in group A*3/4 and 26% of patients in group B*7/8 (P=0. [27] A study form consisting of patient information on possible risk factors for osteoporosis such as gender, age, menopausal status, smoking, family history of osteoporosis, preoperative thyroid hormone status, postoperative hypoparathyroidism history, mean serum TSH levels, and duration of TSH suppression was created and filled out for each participant. [28] Injury to parathyroid glands during thyroid and parathyroid surgery is common and postoperative hypoparathyroidism represents a serious complication. [29] Objective: To analyze the challenges and the impact of specialized care in diagnosis, treatment and in follow-up of patients with postoperative hypoparathyroidism, with potential to improve teaching and further research possibilities in the subject. [30] 22, respectively), whereas the LC&R group patients had significantly longer operation time, higher blood loss, and a higher rate of postoperative hypoparathyroidism compared with the L group (P <. [31] This is mostly attributed to the lower rates of surgical complications, such as postoperative hypoparathyroidism and airway obstruction. [32] There were five (4, PTxT+A; 1, SPTx) patients with postoperative hypoparathyroidism, one with persistent HPT (SPTx) and 13 with normal parathyroid function (4, TPTx+A; 9, SPTx) six months after PTx. [33] Postoperative hypoparathyroidism is the major complication after surgery for benign thyroid disease, thus requiring more awareness. [34] Conclusions The risk of morbidity concerning the recurrent nerve injury and postoperative hypoparathyroidism increases with the extent of surgery. [35] Conclusion The application of nano-carbon technology in thyroidectomy can completely clear the lymph nodes, reduce the occurrence of incidental parathyroidectomy, and reduce the incidence of postoperative hypoparathyroidism, low parathyroid hormone and hypocalcemia. [36] No association was found between postoperative hypoparathyroidism and autotransplantation of a parathyroid gland. [37] ImportancePostoperative hypoparathyroidism remains a relevant problem after thyroid surgery. [38] The prevalence of postoperative hypoparathyroidism has been studied in registries and in surgical series with highly variable and imprecise results. [39] For the prevention of postoperative hypoparathyroidism, in addition to carefully accounting for anatomical and topographic-anatomical features, a «stress-test» and a method of double visual-instrumental recording of the parathyroid gland-induced fluorescence were used. [40]

lymph node dissection

Female sex, neck dissection, the yield of lymph node dissection and incidental parathyroidectomy were significantly associated with postoperative hypoparathyroidism. [1] Univariate analysis showed that postoperative hypoparathyroidism was associated with cervical lymph node dissection, gender, surgical range, operation time, central region lymph node dissection and preoperative PTH level (P 1). [2]

Chronic Postoperative Hypoparathyroidism

The chronic postoperative hypoparathyroidism accounts approximately 75% of patients; the genetic, autoimmune or idiopathic forms are significantly less common. [1] We present a case report of a woman with convulsions, which developed due to dissociative (conversion motor) disorder, with chronic postoperative hypoparathyroidism, receiving standard treatment and having normo- and hypercalcemia. [2] One patient had chronic postoperative hypoparathyroidism and one patient remained normocalcemic 4 years after surgery. [3] One patient had chronic postoperative hypoparathyroidism, and one patient remains normocalcemic 4 years after surgery. [4]

Transient Postoperative Hypoparathyroidism

1%) with transient postoperative hypoparathyroidism, and 21 patients (3. [1] The image-based NIRAF device has been shown in one multicenter prospective study to lower the risk of transient postoperative hypoparathyroidism [3]. [2] Transient postoperative hypoparathyroidism with hypocalcaemia was reported in 8 (9. [3]

Permanent Postoperative Hypoparathyroidism

Accidental damage or removal of the PG may result in temporary or permanent postoperative hypoparathyroidism and hypocalcemia. [1] 4%) and permanent postoperative hypoparathyroidism (2. [2]

Developed Postoperative Hypoparathyroidism

In both groups participants who developed postoperative hypoparathyroidism (parathormone (iPTH) <1. [1] The time intervals for thyroid disorders were 320 cases developed postoperative hypoparathyroidism in eight weeks, 480 cases the second month, and 1000 cases in the first year after surgery. [2]

Preventing Postoperative Hypoparathyroidism

Identification of the parathyroid glands during surgery is crucial for preventing postoperative hypoparathyroidism. [1] Recognition of normal parathyroid glands using ultrasound can be valuable for preventing postoperative hypoparathyroidism and in increasing the accuracy of postsurgical ultrasound surveillance. [2]