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In a healthy population, iron excess may not be a major concern; however, in persons with high oxidative stress and dyslipidaemia, iron excess may place them at greater risk.
In a healthy population, iron excess may not be a major concern; however, in persons with high oxidative stress and dyslipidaemia, iron excess may place them at greater risk.
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Indeed, due to the high reactivity with oxygen and the formation of free radicals, iron excess may cause oxidative damage of cells that are extremely vulnerable to this condition because the normal elevated ROS production and the paucity in antioxidant enzyme activities.
Indeed, due to the high reactivity with oxygen and the formation of free radicals, iron excess may cause oxidative damage of cells that are extremely vulnerable to this condition because the normal elevated ROS production and the paucity in antioxidant enzyme activities.
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10.1021/acs.biochem.1c00586
In response, it is becoming increasingly evident that bacteria have evolved Fe2+ efflux pumps to deal with conditions of ferrous iron excess and to prevent intracellular oxidative stress.
In response, it is becoming increasingly evident that bacteria have evolved Fe2+ efflux pumps to deal with conditions of ferrous iron excess and to prevent intracellular oxidative stress.
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10.18410/JEBMH/2021/287
In a healthy population, iron excess may not be a major concern; however, in persons with high oxidative stress and dyslipidaemia, iron excess may place them at greater risk.
In a healthy population, iron excess may not be a major concern; however, in persons with high oxidative stress and dyslipidaemia, iron excess may place them at greater risk.
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10.1007/s00277-021-04639-0
And by extension, targeting miR-let-7d, miR-122, and miR-200 might serve as novel sensitive, specific and non-invasive predictor biomarkers for cellular damage under condition of tissue iron excess.
And by extension, targeting miR-let-7d, miR-122, and miR-200 might serve as novel sensitive, specific and non-invasive predictor biomarkers for cellular damage under condition of tissue iron excess.
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10.1007/s12098-021-03812-7
The endocrine effects of iron excess have thus far been studied extensively in the pancreas.
The endocrine effects of iron excess have thus far been studied extensively in the pancreas.
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10.1002/dvdy.372
Iron homeostasis is tightly regulated to balance the iron requirement for erythropoiesis and other vital cellular functions, while preventing cellular injury from iron excess.
Iron homeostasis is tightly regulated to balance the iron requirement for erythropoiesis and other vital cellular functions, while preventing cellular injury from iron excess.
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10.3390/membranes11080571
Iron excess in tissues results in increased oxidative damage.
Iron excess in tissues results in increased oxidative damage.
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10.1016/j.abb.2021.109031
Since iron excess is accompanied by increased expression of iron-storage protein, ferritin, we examined whether ferritin has an effect on the ryanodine receptor - isoform 2 (RYR2), which is one of the major components of Ca2+ signaling.
Since iron excess is accompanied by increased expression of iron-storage protein, ferritin, we examined whether ferritin has an effect on the ryanodine receptor - isoform 2 (RYR2), which is one of the major components of Ca2+ signaling.
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10.1111/1751-7915.13939
The best system revealed that the four human and soya bean ferritins possess a novel function as anti-ageing proteins in conditions of iron excess.
The best system revealed that the four human and soya bean ferritins possess a novel function as anti-ageing proteins in conditions of iron excess.
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10.1093/tropej/fmab088
, neither iron deficiency (ID) nor iron excess was measured.
, neither iron deficiency (ID) nor iron excess was measured.
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10.3390/nu13072221
Studies on maternal iron status above normal, or iron excess, suggest deleterious effects on infant growth, cognition, and childhood Type 1 diabetes.
Studies on maternal iron status above normal, or iron excess, suggest deleterious effects on infant growth, cognition, and childhood Type 1 diabetes.
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10.1016/j.jbc.2021.100835
While SLC39A14 imports manganese into the liver and other organs under physiological conditions, it imports iron under conditions of iron excess.
While SLC39A14 imports manganese into the liver and other organs under physiological conditions, it imports iron under conditions of iron excess.
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10.1038/s41467-021-24333-z
Only maternal exposure to both iron excess and inflammation, but not either condition alone, causes embryo malformations and demise.
Only maternal exposure to both iron excess and inflammation, but not either condition alone, causes embryo malformations and demise.
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10.1038/s41598-021-99030-4
Mitotracker staining showed that the mitochondrial activity was enriched in ΔferS under both iron excess and iron depletion.
Mitotracker staining showed that the mitochondrial activity was enriched in ΔferS under both iron excess and iron depletion.
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10.3390/jof7040272
Phenotyping under different iron concentrations revealed detrimental effects on spore swelling and hyphal formation under iron depletion, but yeast-like morphology under iron excess.
Phenotyping under different iron concentrations revealed detrimental effects on spore swelling and hyphal formation under iron depletion, but yeast-like morphology under iron excess.
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10.3390/cells10112841
Indeed, due to the high reactivity with oxygen and the formation of free radicals, iron excess may cause oxidative damage of cells that are extremely vulnerable to this condition because the normal elevated ROS production and the paucity in antioxidant enzyme activities.
Indeed, due to the high reactivity with oxygen and the formation of free radicals, iron excess may cause oxidative damage of cells that are extremely vulnerable to this condition because the normal elevated ROS production and the paucity in antioxidant enzyme activities.
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10.1152/ajpendo.00116.2021
These findings also suggest that if Mɸs are capable of storing iron properly, they have a pronounced ability to withstand iron excess without evoking overt collateral damage and associated insulin resistance that may be age dependent.
These findings also suggest that if Mɸs are capable of storing iron properly, they have a pronounced ability to withstand iron excess without evoking overt collateral damage and associated insulin resistance that may be age dependent.
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10.1177/20458940211055996
Both SCA and β-thalassemia intermedia present with intra and extravascular hemolysis, and because SCA and β-thalassemia intermedia share features of extravascular hemolysis, macrophage iron excess and anemia we sought to characterize the common features of the PH phenotype, cardiac mechanics, and function as well as lung and right ventricular metabolism.
Both SCA and β-thalassemia intermedia present with intra and extravascular hemolysis, and because SCA and β-thalassemia intermedia share features of extravascular hemolysis, macrophage iron excess and anemia we sought to characterize the common features of the PH phenotype, cardiac mechanics, and function as well as lung and right ventricular metabolism.
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10.3389/fimmu.2021.635899
Emerging evidence indicates that reduced iron intake and low systemic iron levels are associated with the pathogenesis of colorectal cancer, suggesting that optimal iron intake must be carefully balanced to avoid both iron deficiency and iron excess.
Emerging evidence indicates that reduced iron intake and low systemic iron levels are associated with the pathogenesis of colorectal cancer, suggesting that optimal iron intake must be carefully balanced to avoid both iron deficiency and iron excess.
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10.1186/s12866-021-02320-0
Parasutterella excrementihominis , Bacteroides vulgatus , and Alistipes finegoldii , were more abundant with the iron excess diet.
Parasutterella excrementihominis , Bacteroides vulgatus , and Alistipes finegoldii , were more abundant with the iron excess diet.
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10.1099/mic.0.000838
5), iron starvation (bipyridyl) and iron excess (FeCl3), and antibiotic stress (tetracycline and ciprofloxacin).
5), iron starvation (bipyridyl) and iron excess (FeCl3), and antibiotic stress (tetracycline and ciprofloxacin).
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10.3389/fnins.2019.00386
By promoting erythropoiesis, EPO influences iron metabolism and induces shifts in iron pool which may ameliorate conditions of free iron excess and iron accumulation.
By promoting erythropoiesis, EPO influences iron metabolism and induces shifts in iron pool which may ameliorate conditions of free iron excess and iron accumulation.
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10.1096/fj.201901106R
Our objective was to get a better understanding of the mechanisms leading to iron excess in HA.
Our objective was to get a better understanding of the mechanisms leading to iron excess in HA.
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10.1172/jci127341
The increasing prevalence of indiscriminate iron supplementation during pregnancy also raises concerns about the potential adverse effects of iron excess.
The increasing prevalence of indiscriminate iron supplementation during pregnancy also raises concerns about the potential adverse effects of iron excess.
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10.1002/ajh.25366
The bone morphogenetic protein (BMP)‐SMAD signaling pathway is a key transcriptional regulator of hepcidin in response to tissue iron stores, serum iron, erythropoietic drive and inflammation to increase the iron supply when needed for erythropoiesis, but to prevent the toxicity of iron excess.
The bone morphogenetic protein (BMP)‐SMAD signaling pathway is a key transcriptional regulator of hepcidin in response to tissue iron stores, serum iron, erythropoietic drive and inflammation to increase the iron supply when needed for erythropoiesis, but to prevent the toxicity of iron excess.
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10.1093/jn/nxz166
2), suggesting that iron excess enhanced purine catabolism.
2), suggesting that iron excess enhanced purine catabolism.
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10.1093/pch/pxz148
Due to a lack of iron excretory mechanism, the possibility of iron excess also exists.
Due to a lack of iron excretory mechanism, the possibility of iron excess also exists.
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10.1182/blood-2018-06-815894
Conversely, hepcidin production is induced by iron loading and inflammation to prevent the toxicity of iron excess and limit its availability to pathogens.
Conversely, hepcidin production is induced by iron loading and inflammation to prevent the toxicity of iron excess and limit its availability to pathogens.
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10.1093/gerona/gly270
Iron excess can be prevented or treated but diagnosis is often delayed or missed.
Iron excess can be prevented or treated but diagnosis is often delayed or missed.
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10.1093/cdn/nzz048.P11-072-19
Objectives
Our prior work demonstrated that dietary iron excess in early life results in iron overload in both liver and hippocampus in pre-weanling piglets.
Objectives
Our prior work demonstrated that dietary iron excess in early life results in iron overload in both liver and hippocampus in pre-weanling piglets.
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10.1038/s41598-019-42765-y
We modulated the iron status by adding either the iron chelator Deferoxamine (DFO) for iron deficiency, or ferric ammonium citrate for iron excess, and followed the emergence of developing haematopoietic progenitors.
We modulated the iron status by adding either the iron chelator Deferoxamine (DFO) for iron deficiency, or ferric ammonium citrate for iron excess, and followed the emergence of developing haematopoietic progenitors.
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10.5897/JTEHS2019.0429
More studies are recommended to evaluate the beneficial effects of quercetin with iron excess.
More studies are recommended to evaluate the beneficial effects of quercetin with iron excess.
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10.1093/jn/nxy227
OBJECTIVE
We hypothesized that early-life iron excess causes systemic and central nervous system iron overload, and compromises social behavior.
OBJECTIVE
We hypothesized that early-life iron excess causes systemic and central nervous system iron overload, and compromises social behavior.
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10.1038/S42255-019-0063-6
Here Lim and colleagues show that iron excess activates Nrf2 via mitochondrial reactive oxygen species, enhancing the expression of Bmp6 in liver sinusoidal endothelial cells, which in turn promotes hepcidin expression by hepatocytes, decreasing systemic iron levels.
Here Lim and colleagues show that iron excess activates Nrf2 via mitochondrial reactive oxygen species, enhancing the expression of Bmp6 in liver sinusoidal endothelial cells, which in turn promotes hepcidin expression by hepatocytes, decreasing systemic iron levels.
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10.15372/SSMJ20190111
The role of multiple transfusions in formation of iron excess in patients with myelodysplastic syndrome was demonstrated.
The role of multiple transfusions in formation of iron excess in patients with myelodysplastic syndrome was demonstrated.
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10.3760/CMA.J.ISSN.1001-9030.2019.01.008
Objective
To investigate the effects of stress caused by iron excess on biologic activity of murine myoblast C2C12 cells and related mechanism.
Objective
To investigate the effects of stress caused by iron excess on biologic activity of murine myoblast C2C12 cells and related mechanism.
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10.1186/s41938-019-0185-x
solanacearum density after corn coincided with the high ratio of fluorescent pseudomonads, endospores, and actinomycetes, being most clear in the poor soils (Wardan) and less clear under iron excess at Ganoub El-Tahrir as well as the clay soils.
solanacearum density after corn coincided with the high ratio of fluorescent pseudomonads, endospores, and actinomycetes, being most clear in the poor soils (Wardan) and less clear under iron excess at Ganoub El-Tahrir as well as the clay soils.
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10.1097/ACO.0000000000000825
Iron excess, typically because of genetic defects, can cause toxicity to tissues and, like iron deficiency, impair wound and injury repair.
Iron excess, typically because of genetic defects, can cause toxicity to tissues and, like iron deficiency, impair wound and injury repair.
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10.3390/nu11030674
Considering that iron excess is able to negatively alter the microbiome, the use of alginate as a food supplement could be useful in colonic-iron chelation.
Considering that iron excess is able to negatively alter the microbiome, the use of alginate as a food supplement could be useful in colonic-iron chelation.
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10.3390/cells8060603
While chronic hepatitis C virus infection drives deficiencies in micronutrients such as zinc, selenium, vitamin A and B12, it also stimulates copper and iron excess; these micronutrients influence antioxidant, inflammatory and immune responses to HCV.
While chronic hepatitis C virus infection drives deficiencies in micronutrients such as zinc, selenium, vitamin A and B12, it also stimulates copper and iron excess; these micronutrients influence antioxidant, inflammatory and immune responses to HCV.
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10.1021/acschemneuro.8b00263
Because of the serious neurologic consequences of iron deficiency and iron excess in the brain, interest in the iron status of the central nervous system has increased significantly in the past decade.
Because of the serious neurologic consequences of iron deficiency and iron excess in the brain, interest in the iron status of the central nervous system has increased significantly in the past decade.
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10.1016/j.tracli.2018.08.006
Hemochromatoses, mostly but not exclusively related to the HFE gene, correspond to systemic iron overload of genetic origin in which iron excess is the consequence of hepcidin deficiency, hepcidin being the hormone regulating negatively plasma iron.
Hemochromatoses, mostly but not exclusively related to the HFE gene, correspond to systemic iron overload of genetic origin in which iron excess is the consequence of hepcidin deficiency, hepcidin being the hormone regulating negatively plasma iron.
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10.1186/s13104-019-4362-5
ObjectivesThis study was conducted to establish a method for early, quick and cheap screening of iron excess tolerance in rice (Oryza sativa L.
ObjectivesThis study was conducted to establish a method for early, quick and cheap screening of iron excess tolerance in rice (Oryza sativa L.
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10.1016/j.fsi.2019.08.021
Besides, iron excess also increased enteritis morbidity and impaired immune function of fish under infection of A.
Besides, iron excess also increased enteritis morbidity and impaired immune function of fish under infection of A.
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10.31413/NATIVA.V7I1.6075
EXCESS OF IRON ON THE DEVELOPMENT OF CITRUS ROOTSTOCKS ABSTRACT: The objective of this work was evaluated the effects of iron excess on the emergence and initial development of citrus rootstock genotypes.
EXCESS OF IRON ON THE DEVELOPMENT OF CITRUS ROOTSTOCKS ABSTRACT: The objective of this work was evaluated the effects of iron excess on the emergence and initial development of citrus rootstock genotypes.
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Keywords related to Iron
Iron Catalyzed
Iron Reducing
Iron Phthalocyanine
Iron Ore Coal
Iron Promoted
Iron Thin
Iron Doped Hydroxyapatite
Iron Chromium
Iron Line
Iron Reduction
Iron Produced
Iron Halogenases
Iron Starvation
Iron Catalyzed Reductive
Iron Sulfide
Iron Sulfur Proteins
Iron Addition
Iron Pincer
Iron Assimilation
Iron Repletion
Iron Telluride
Iron Accumulation
Iron Sucrose
Iron Phases
Iron Deficient Patients
Iron Meteorites
Iron Aluminide
Iron Mineralization
Iron Chalcogenide
Iron Magnesium
Iron Values
Iron Stress
Iron Sulfur Cluster
Iron Caused
Iron Phosphide
Iron Nanocluster
Iron Speciation
Iron Determination
Iron Doped Biochar
Iron Mineralogy
Iron Rich
Iron Titanium
Iron Bound
Iron Indices
Iron Catalyzed Dehydrogenative
Iron Sulfur Protein
Iron Quadrangle
Iron Nanoparticle
Iron Garnets
Iron Nanoparticles
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Keywords related to Excess
Boundary Excess
Negative Excess
Enantiomeric Excess
Chronic Excess
Divergence Excess
Deuterium Excess
Surface Excess
Associated Excess
Low Energy Excess
Lithium Excess
Gev Excess
X Ray Excess
Androgen Excess
Predicting Excess
Recoil Excess
Diphoton Excess
Soft Dielectron Excess
Maxillary Excess
Reducing Excess
Entropy Excess
Considering Excess
Base Excess
Xenon1t Excess
Containing Excess
Air Excess
Long Term Excess
Industrial Excess
Positron Excess
Hydrated Excess
Cortisol Excess
Covid 19 Excess
Hormone Excess
Preventing Excess
Substantial Excess
Large Excess
Weight Excess
Center Excess
Iodine Excess
Currency Excess
Residual Excess
Aldosterone Excess
Prevent Excess
Confer Excess
Explaining Excess
Much Excess
Mineralocorticoid Excess
Gamma Ray Excess
Testosterone Excess
Glucocorticoid Excess
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