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Identification of bioactive metabolites in human iPSC-derived dopaminergic neurons with PARK2 mutation: Altered mitochondrial and energy metabolism



In vitro genome editing rescues parkinsonism phenotypes in induced pluripotent stem cells-derived dopaminergic neurons carrying LRRK2 p.G2019S mutation



Modelling Parkinson’s Disease: iPSCs towards Better Understanding of Human Pathology



Establishment of an in vitro model for analyzing mitochondrial ultrastructure in PRKN-mutated patient iPSC-derived dopaminergic neurons



The AKT modulator A-443654 reduces α-synuclein expression and normalizes ER stress and autophagy



Phenotypic manifestation of α-synuclein strains derived from Parkinson’s disease and multiple system atrophy in human dopaminergic neurons


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10.1016/j.brainresbull.2021.07.016

Restoring lost nigrostriatal fibers in Parkinson’s disease based on clinically-inspired design criteria


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10.1080/15548627.2019.1598754

The PARK10 gene USP24 is a negative regulator of autophagy and ULK1 protein stability



CCAAT/enhancer binding protein δ is a transcriptional repressor of α-synuclein



Impact of Sleep–Wake-Associated Neuromodulators and Repetitive Low-Frequency Stimulation on Human iPSC-Derived Neurons



Special issue on stem cell and tissue engineering in development, disease, and repair



Homogenous generation of dopaminergic neurons from multiple hiPSC lines by transient expression of transcription factors



A modulator of wild-type glucocerebrosidase improves pathogenic phenotypes in dopaminergic neuronal models of Parkinson’s disease



An integrated transcriptomics and proteomics analysis reveals functional endocytic dysregulation caused by mutations in LRRK2


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