Introduction to Hereditary Tumors

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10.1007/978-3-030-77633-6_26

In hereditary tumors, we can find constitutional alterations associated with somatic loss of function of wild-type alleles.

Genetic Aspects of Peripheral Nervous System Tumors

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10.1155/2021/9981028

Retinoblastoma (RB) is the most common primary intraocular malignant tumor in infants and the prototype of human hereditary tumors.

Correlation between Family RB1 Gene Pathogenic Variant with Clinical Features and Prognosis of Retinoblastoma under 5 Years Old

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10.1155/2021/1392386

Purpose Recently, pheochromocytomas and paragangliomas (PPGLs) have been strongly suspected as hereditary tumors, as approximately 40% of patients carry germline mutations.

Recurrent Germline Mutations of CHEK2 as a New Susceptibility Gene in Patients with Pheochromocytomas and Paragangliomas

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10.1111/jog.14861

This review article outlines the current status and future prospects of precision medicine for gynecologic hereditary tumors focusing on HBOC and LS.

Precision medicine for hereditary tumors in gynecologic malignancies

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10.1200/jco.2021.39.15_suppl.e22523

e22523 Background: Hereditary tumors are generally monogenic diseases with autosomal dominant inheritance.

Clinical and genetic characterization of cancer patients with multiple germline variants.

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10.1002/jgc4.1390

Pheochromocytoma and paraganglioma are frequently hereditary tumors commonly associated with succinate dehydrogenase (SDHx) pathogenic variants (PV).

Genetic testing for pheochromocytoma and paraganglioma: SDHx carriers’ experiences

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10.1038/s41419-021-03731-7

Thus, our study raises the importance of cortical hormone in PPGL development, and GIPC2 as a novel tumor suppressor provides a unified molecular mechanism for the tumorigenesis of both sporadic and hereditary tumors of Clusters 1A and 2A concerning SDHB and RET, but not tumors of Cluster 1B concerning VHL and other clusters.

GIPC2 is an endocrine-specific tumor suppressor gene for both sporadic and hereditary tumors of RET- and SDHB-, but not VHL-associated clusters of pheochromocytoma/paraganglioma

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10.5772/INTECHOPEN.96126

Paragangliomas and pheochromocytoma (PPGLs) are hereditary tumors in about 40% of cases.

Metastatic Paragangliomas and Pheochromocytomas: An Epigenetic View

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10.1007/s00535-021-01776-1

The professional committee created 18 CQs and statements concerning the current concept and diagnosis/treatment of various colorectal polyps, including their epidemiology, screening, pathophysiology, definition and classification, diagnosis, management, practical treatment, complications, and surveillance after treatment, and other colorectal lesions (submucosal tumors, nonneoplastic polyps, polyposis, hereditary tumors, ulcerative colitis-associated tumors/carcinomas).

Evidence-based clinical practice guidelines for management of colorectal polyps

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Hereditary Tumors
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What is/are Hereditary Tumors?

Hereditary Tumors - In hereditary tumors, we can find constitutional alterations associated with somatic loss of function of wild-type alleles. [1] Retinoblastoma (RB) is the most common primary intraocular malignant tumor in infants and the prototype of human hereditary tumors. [2] Purpose Recently, pheochromocytomas and paragangliomas (PPGLs) have been strongly suspected as hereditary tumors, as approximately 40% of patients carry germline mutations. [3] This review article outlines the current status and future prospects of precision medicine for gynecologic hereditary tumors focusing on HBOC and LS. [4] e22523 Background: Hereditary tumors are generally monogenic diseases with autosomal dominant inheritance. [5] Pheochromocytoma and paraganglioma are frequently hereditary tumors commonly associated with succinate dehydrogenase (SDHx) pathogenic variants (PV). [6] Thus, our study raises the importance of cortical hormone in PPGL development, and GIPC2 as a novel tumor suppressor provides a unified molecular mechanism for the tumorigenesis of both sporadic and hereditary tumors of Clusters 1A and 2A concerning SDHB and RET, but not tumors of Cluster 1B concerning VHL and other clusters. [7] Paragangliomas and pheochromocytoma (PPGLs) are hereditary tumors in about 40% of cases. [8] The professional committee created 18 CQs and statements concerning the current concept and diagnosis/treatment of various colorectal polyps, including their epidemiology, screening, pathophysiology, definition and classification, diagnosis, management, practical treatment, complications, and surveillance after treatment, and other colorectal lesions (submucosal tumors, nonneoplastic polyps, polyposis, hereditary tumors, ulcerative colitis-associated tumors/carcinomas). [9]