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10.1111/bph.15364

Its effect on folic acid‐induced acute kidney injury, however, remains unknown.

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10.1097/ALN.0000000000002906

WHAT WE ALREADY KNOW ABOUT THIS TOPIC Nitrous oxide exposure can induce spinal cord neurodegeneration and related neuropathy in humans rendered susceptible by genetic or acquired risk factorsThe effects of nitrous oxide on the regeneration of nervous system following trauma has not been previously reported WHAT THIS ARTICLE TELLS US THAT IS NEW: In in vitro and in vivo experimental models of male rats, nitrous oxide exposure impairs folic acid-induced axonal regeneration of dorsal root and retinal ganglion neuronsThe beneficial effects of folic acid on functional recovery following spinal cord contusion in male rats are hindered by co-administration of nitrous oxideThese experiments suggest that nitrous oxide can interfere with axonal regeneration and functional recovery following central nervous system injury BACKGROUND:: Nitrous oxide can induce neurotoxicity.

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10.1016/j.tox.2019.152303

Experimental folic acid-induced AKI is comparable to human AKI and is repeatedly reported to study the pathogenic pathway of human AKI and study the therapeutic efficacy of different agents against AKI.

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10.1016/j.kint.2018.11.043

MMP-7 was induced in renal tubules following ischemia/ reperfusion injury or cisplatin administration, and in folic acid-induced AKI.

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10.1038/s41581-019-0228-2

In the folic acid-induced neprophathy (FAN) mouse model of kidney fibrosis, Mir33–/– mice had lower expression of fibrosis markers (α-smooth muscle actin, fibronectin and collagen) and lower levels of blood urea nitrogen (BUN) and creatinine than wild-type (WT) mice.

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10.15407/UBJ91.03.019

In our work, the effect of glutathione on the activity of respiratory chain enzymes and the content of free Sh-groups in rat liver mitochondria was examined with the use of folic acid-induced nephropathy model.

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Folic Acid Derived Folic Acid Targeted Folic Acid Functionalized Folic Acid Conjugated Folic Acid Modified Folic Acid Explore More

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Fluoride Induced Palsy Induced Decays Induced Aerosol Induced Mixing Induced Fibrosis Induced Polymer Induced Alleviates Hypoxia Induced Chloride Induced Freunds Adjuvant Induced Gels Induced Against Contrast Induced Thrombin Induced Factor B1 Induced Phase Induced Anisotropy Induced Staphylococcus Aureus Induced Systemic Inflammation Induced Fusion Induced Receptor Induced Mediates Hypoxia Induced Anxiety Induced Astrocytes Induced Mediates Cd Induced Splitting Induced Inhibit Lipopolysaccharide Induced Fractions Induced Fractures Induced Doxorubicin Induced Nociception Induced Anomaly Induced Neurons Induced Exposure Induced Dysplasia Induced Chemotherapy Induced Spinal Anesthesia Induced Subsidence Induced Contrast Agent Induced Neovascularization Induced Be Induced Polymyxin B Induced Cerebral Ischemia Induced Propionic Acid Induced Covid 19 Pandemic Induced Compound 48 80 Induced Membranes Induced Fluid Induced Tacrolimus Induced Diabetes Induced Inhibits Pdgf Bb Induced Explore More

Folic Acid Induced

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Sep 18, 2024

What is/are Folic Acid Induced?

Folic Acid Induced - Its effect on folic acid‐induced acute kidney injury, however, remains unknown. ^[1] WHAT WE ALREADY KNOW ABOUT THIS TOPIC Nitrous oxide exposure can induce spinal cord neurodegeneration and related neuropathy in humans rendered susceptible by genetic or acquired risk factorsThe effects of nitrous oxide on the regeneration of nervous system following trauma has not been previously reported WHAT THIS ARTICLE TELLS US THAT IS NEW: In in vitro and in vivo experimental models of male rats, nitrous oxide exposure impairs folic acid-induced axonal regeneration of dorsal root and retinal ganglion neuronsThe beneficial effects of folic acid on functional recovery following spinal cord contusion in male rats are hindered by co-administration of nitrous oxideThese experiments suggest that nitrous oxide can interfere with axonal regeneration and functional recovery following central nervous system injury BACKGROUND:: Nitrous oxide can induce neurotoxicity. ^[2] Experimental folic acid-induced AKI is comparable to human AKI and is repeatedly reported to study the pathogenic pathway of human AKI and study the therapeutic efficacy of different agents against AKI. ^[3] MMP-7 was induced in renal tubules following ischemia/ reperfusion injury or cisplatin administration, and in folic acid-induced AKI. ^[4] In the folic acid-induced neprophathy (FAN) mouse model of kidney fibrosis, Mir33–/– mice had lower expression of fibrosis markers (α-smooth muscle actin, fibronectin and collagen) and lower levels of blood urea nitrogen (BUN) and creatinine than wild-type (WT) mice. ^[5] In our work, the effect of glutathione on the activity of respiratory chain enzymes and the content of free Sh-groups in rat liver mitochondria was examined with the use of folic acid-induced nephropathy model. ^[6]