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dopaminergic nerve terminal

METH injections induced hyperthermia, an increase in plasma HMGB1 concentration, degeneration of dopaminergic nerve terminals, accumulation of microglia, and extracellular release of neuronal HMGB1 in the striatum.

Neuroprotective Effects of Anti-high Mobility Group Box-1 Monoclonal Antibody Against Methamphetamine-Induced Dopaminergic Neurotoxicity.

dopaminergic nerve terminal

Furthermore, carbonyl scavenging treatments, such as hydralazine, were found to protect dopaminergic nerve terminals in a model of Parkinson’s disease, and to alleviate mechanical hyperalgesia in animal models of multiple sclerosis and spinal cord injury.

Pathological role of acrolein in neurotrauma and neurodegeneration

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Dopaminergic Nerve sentence examples within dopaminergic nerve cell

dopaminergic nerve cell

Proliferating cells, immature neurons, and dopaminergic nerve cells were immunohistochemically detected using antibodies against bromodeoxyuridine, doublecortin, and tyrosine hydroxylase.

Hippocampal Neurogenesis, Dopaminergic Neurons of the Substantia Nigra, and Behavior after Intranasal Administration of Native α-Synuclein Protein to Ageing Mice

dopaminergic nerve cell

Objectives: Parkinson's Disease (PD) is a form of neurodegenerative disease that is caused the progressive weakening of dopaminergic nerve cells that affects a large number of people around the world.

A comparative and comprehensive study of prediction of Parkinson’s disease

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10.1007/s12640-021-00402-5

METH injections induced hyperthermia, an increase in plasma HMGB1 concentration, degeneration of dopaminergic nerve terminals, accumulation of microglia, and extracellular release of neuronal HMGB1 in the striatum.

Neuroprotective Effects of Anti-high Mobility Group Box-1 Monoclonal Antibody Against Methamphetamine-Induced Dopaminergic Neurotoxicity.

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10.1134/S181971242101013X

Proliferating cells, immature neurons, and dopaminergic nerve cells were immunohistochemically detected using antibodies against bromodeoxyuridine, doublecortin, and tyrosine hydroxylase.

Hippocampal Neurogenesis, Dopaminergic Neurons of the Substantia Nigra, and Behavior after Intranasal Administration of Native α-Synuclein Protein to Ageing Mice

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10.1016/j.neulet.2021.135873

The loss of dopaminergic nerves and dopamine deficiency leads to motor symptoms characterized by rigidity, tremor, and bradykinesia.

Metals associated neurodegeneration in Parkinson’s disease: Insight to physiological, pathological mechanisms and management

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10.25394/PGS.14933226.V1

Furthermore, carbonyl scavenging treatments, such as hydralazine, were found to protect dopaminergic nerve terminals in a model of Parkinson’s disease, and to alleviate mechanical hyperalgesia in animal models of multiple sclerosis and spinal cord injury.

Pathological role of acrolein in neurotrauma and neurodegeneration

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10.11591/ijeecs.v23.i3.pp1748-1760

Objectives: Parkinson's Disease (PD) is a form of neurodegenerative disease that is caused the progressive weakening of dopaminergic nerve cells that affects a large number of people around the world.

A comparative and comprehensive study of prediction of Parkinson’s disease

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Dopaminergic Nerve - METH injections induced hyperthermia, an increase in plasma HMGB1 concentration, degeneration of dopaminergic nerve terminals, accumulation of microglia, and extracellular release of neuronal HMGB1 in the striatum. [1] Proliferating cells, immature neurons, and dopaminergic nerve cells were immunohistochemically detected using antibodies against bromodeoxyuridine, doublecortin, and tyrosine hydroxylase. [2] The loss of dopaminergic nerves and dopamine deficiency leads to motor symptoms characterized by rigidity, tremor, and bradykinesia. [3] Furthermore, carbonyl scavenging treatments, such as hydralazine, were found to protect dopaminergic nerve terminals in a model of Parkinson’s disease, and to alleviate mechanical hyperalgesia in animal models of multiple sclerosis and spinal cord injury. [4] Objectives: Parkinson's Disease (PD) is a form of neurodegenerative disease that is caused the progressive weakening of dopaminergic nerve cells that affects a large number of people around the world. [5]

dopaminergic nerve terminal

METH injections induced hyperthermia, an increase in plasma HMGB1 concentration, degeneration of dopaminergic nerve terminals, accumulation of microglia, and extracellular release of neuronal HMGB1 in the striatum. [1] Furthermore, carbonyl scavenging treatments, such as hydralazine, were found to protect dopaminergic nerve terminals in a model of Parkinson’s disease, and to alleviate mechanical hyperalgesia in animal models of multiple sclerosis and spinal cord injury. [2]

dopaminergic nerve cell

Proliferating cells, immature neurons, and dopaminergic nerve cells were immunohistochemically detected using antibodies against bromodeoxyuridine, doublecortin, and tyrosine hydroxylase. [1] Objectives: Parkinson's Disease (PD) is a form of neurodegenerative disease that is caused the progressive weakening of dopaminergic nerve cells that affects a large number of people around the world. [2]