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Dopaminergic Innervation sentence examples within ventral tegmental area

ventral tegmental area

Frontostriatal structures, such as the nucleus accumbens (NAcb), the medial prefrontal cortex (mPFC), and their dopaminergic innervation from the ventral tegmental area (VTA) have been implicated in impulse control and attention.

Temporally Specific Roles of Ventral Tegmental Area Projections to the Nucleus Accumbens and Prefrontal Cortex in Attention and Impulse Control

ventral tegmental area

The prefrontal cortex (PFC) and nucleus accumbens (NAcc) receive dopaminergic innervation from the ventral tegmental area (VTA).

Effects of inactivation of the ventral tegmental area on prefronto-accumbens activity and sexual motivation in male rats

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Dopaminergic Innervation sentence examples within Striatal Dopaminergic Innervation

Striatal Dopaminergic Innervation

Both motor and cognitive recovery and partial circadian rhythm recovery correlate with the degree of graft integration into the host environment as well as with in-vivo levels of striatal dopaminergic innervation and function.

Induced Cognitive Impairments Reversed by Grafts of Neural Precursors: a Longitudinal Study in a Macaque Model of Parkinson’s Disease

Striatal Dopaminergic Innervation

Immunohistochemical and biochemical analyses showed that oral subchronic OTA treatment induced loss of striatal dopaminergic innervation and dopaminergic cell dysfunction responsible for motor impairments.

Oral subchronic exposure to the mycotoxin ochratoxin A induces key pathological features of Parkinson´s disease in mice six months after the end of the treatment.

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Dopaminergic Innervation sentence examples within Increased Dopaminergic Innervation

Increased Dopaminergic Innervation

Here, we show an increased dopaminergic innervation of the ischemic territory during the first week after stroke induced in Wistar rats subjected to transient occlusion of the middle cerebral artery (tMCAO) for 120 min.

Plasticity-Enhancing Effects of Levodopa Treatment after Stroke

Increased Dopaminergic Innervation

In rodent models, exposure to 17‐OHPC significantly increased dopaminergic innervation of the medial prefrontal cortex (mPFC), an aberrant pattern of connectivity that may underlie deficits in cognitive flexibility observed in adulthood.

Sex differences in dopamine innervation and microglia are altered by synthetic progestin in neonatal medial prefrontal cortex

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10.1016/B978-0-12-817988-8.00026-9

In this chapter we describe the maturational changes occurring in the adolescent prefrontal cortex and the organizational role that dopaminergic innervation plays in this process.

Adolescent dopamine development

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10.3390/ijms22105326

Previous studies showed that pharmacological depletion of dopamine significantly reduces Rhes mRNA levels in rodents, non-human primates and Parkinson’s disease (PD) patients, suggesting a link between dopaminergic innervation and physiological Rhes mRNA expression.

Involvement of the Protein Ras Homolog Enriched in the Striatum, Rhes, in Dopaminergic Neurons’ Degeneration: Link to Parkinson’s Disease

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10.1002/mdc3.13192

One imaging modality used for diagnosis is dopamine transporter (DaT) imaging, which usually demonstrate loss of dopaminergic innervation to the striatum, occasionally with severe lateralization.

Asymmetric Distribution of Dopamine Transporters in Premorbid Corticobasal Syndrome—A Case Report

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10.1016/j.bbr.2020.113067

Examination of levels of TH, as a reflection of dopaminergic innervation, found no difference in either gender post-TBI in the PFC, but a significant increase in the limbic system (nucleus accumbens) in males, but not females, chronically post-TBI, suggesting an imbalance between the regions.

Injury during adolescence leads to sex-specific executive function deficits in adulthood in a pre-clinical model of mild traumatic brain injury

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10.20944/PREPRINTS202104.0379.V1

Previous studies showed that pharmacological depletion of dopamine significantly reduces Rhes mRNA levels in rodents, non-human primates and Parkinson’s disease (PD) patients, suggesting a link between dopaminergic innervation and physiological Rhes mRNA expression.

Involvement of the Protein Ras Homolog Enriched in the Striatum, Rhes, in Dopaminergic Neurons Degeneration: Link to Parkison’s Disease

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10.1002/mds.27535

Dopaminergic innervation of the putamen is essentially absent in PD subjects within 4 years after diagnosis, indicating that any neuroprotective therapy, including STN DBS, will require intervention within the immediate postdiagnosis interval.

BDNF provides many routes toward STN DBS‐mediated disease modification

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10.3389/fnins.2019.00360

These results indicate that H1R blockade at E14–E16 favors the differentiation of dopaminergic neurons, but hampers their migration, leading to a decrease in dopaminergic innervation of the striatum in post-natal life.

The Systemic Administration of the Histamine H1 Receptor Antagonist/Inverse Agonist Chlorpheniramine to Pregnant Rats Impairs the Development of Nigro-Striatal Dopaminergic Neurons

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10.22037/IJPR.2019.2344

In addition, within the hippocampus, DA receptors are mostly located in the dorsal part (CA1) and dopaminergic innervations are predominant in this sub-region.

The Involvement of Intra-Hippocampal Dopamine Receptors in the Conditioned Place Preference Induced By Orexin Administration into the Rat Ventral Tegmental Area

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10.1177/1073858419834517

The brain circuits processing these signals are widespread and include the prefrontal cortex, amygdala and striatum, and their dopaminergic innervation.

How Reward and Aversion Shape Motivation and Decision Making: A Computational Account

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10.1523/JNEUROSCI.0623-19.2019

1 KO mice exhibit no deficit in social engagement, working memory, sensorimotor gating, native locomotor activity, and dopaminergic innervation.

Disturbed Prefrontal Cortex Activity in the Absence of Schizophrenia-Like Behavioral Dysfunction in Arc/Arg3.1 Deficient Mice

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10.1002/dneu.22660

Furthermore, the increase in H3K4me1 at the Arc locus requires dopamine receptor signaling, and the priming of Arc expression correlates with the dopaminergic innervation pattern in the frontal cortex.

Developmental Exposure to Psychostimulant Primes Activity‐dependent Gene Induction in Frontal Cortex

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10.1007/978-3-319-97897-0_15

Altered sweating appears to occur in two forms: that related to dopaminergic innervation (“off” sweating) and the other being non-dopaminergic.

Treatment of Drenching Sweats in Parkinson’s Disease

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10.1016/j.mcn.2019.06.002

We used tyrosine hydroxylase (TH) expression as a proxy for dopaminergic innervation and showed that TH onset occurs at E13 and progressively increased until postnatal stages following an 'inside-out' pattern.

Embryonic and postnatal development of mouse olfactory tubercle

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Dopaminergic Innervation
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What is/are Dopaminergic Innervation?

Dopaminergic Innervation - In this chapter we describe the maturational changes occurring in the adolescent prefrontal cortex and the organizational role that dopaminergic innervation plays in this process. [1] Previous studies showed that pharmacological depletion of dopamine significantly reduces Rhes mRNA levels in rodents, non-human primates and Parkinson’s disease (PD) patients, suggesting a link between dopaminergic innervation and physiological Rhes mRNA expression. [2] One imaging modality used for diagnosis is dopamine transporter (DaT) imaging, which usually demonstrate loss of dopaminergic innervation to the striatum, occasionally with severe lateralization. [3] Examination of levels of TH, as a reflection of dopaminergic innervation, found no difference in either gender post-TBI in the PFC, but a significant increase in the limbic system (nucleus accumbens) in males, but not females, chronically post-TBI, suggesting an imbalance between the regions. [4] Previous studies showed that pharmacological depletion of dopamine significantly reduces Rhes mRNA levels in rodents, non-human primates and Parkinson’s disease (PD) patients, suggesting a link between dopaminergic innervation and physiological Rhes mRNA expression. [5] Dopaminergic innervation of the putamen is essentially absent in PD subjects within 4 years after diagnosis, indicating that any neuroprotective therapy, including STN DBS, will require intervention within the immediate postdiagnosis interval. [6] These results indicate that H1R blockade at E14–E16 favors the differentiation of dopaminergic neurons, but hampers their migration, leading to a decrease in dopaminergic innervation of the striatum in post-natal life. [7] In addition, within the hippocampus, DA receptors are mostly located in the dorsal part (CA1) and dopaminergic innervations are predominant in this sub-region. [8] The brain circuits processing these signals are widespread and include the prefrontal cortex, amygdala and striatum, and their dopaminergic innervation. [9] 1 KO mice exhibit no deficit in social engagement, working memory, sensorimotor gating, native locomotor activity, and dopaminergic innervation. [10] Furthermore, the increase in H3K4me1 at the Arc locus requires dopamine receptor signaling, and the priming of Arc expression correlates with the dopaminergic innervation pattern in the frontal cortex. [11] Altered sweating appears to occur in two forms: that related to dopaminergic innervation (“off” sweating) and the other being non-dopaminergic. [12] We used tyrosine hydroxylase (TH) expression as a proxy for dopaminergic innervation and showed that TH onset occurs at E13 and progressively increased until postnatal stages following an 'inside-out' pattern. [13]

ventral tegmental area

Frontostriatal structures, such as the nucleus accumbens (NAcb), the medial prefrontal cortex (mPFC), and their dopaminergic innervation from the ventral tegmental area (VTA) have been implicated in impulse control and attention. [1] The prefrontal cortex (PFC) and nucleus accumbens (NAcc) receive dopaminergic innervation from the ventral tegmental area (VTA). [2] Histological analyses revealed reduced dopaminergic innervation from the ventral tegmental area to the olfactory tubercle, consistent with altered impulsivity neurocircuitry. [3]

Striatal Dopaminergic Innervation

Both motor and cognitive recovery and partial circadian rhythm recovery correlate with the degree of graft integration into the host environment as well as with in-vivo levels of striatal dopaminergic innervation and function. [1] Immunohistochemical and biochemical analyses showed that oral subchronic OTA treatment induced loss of striatal dopaminergic innervation and dopaminergic cell dysfunction responsible for motor impairments. [2] I read with interest the article by Honkanen and colleagues on the question of correlation between striatal dopaminergic innervation and dopamine transporter (DAT) imaging as assessed by putamen tyrosine hydroxylase–positive axon counts and DAT single-photon emission computed tomography (SPECT). [3] Identifying new strategies to protect these cells from degeneration and retain striatal dopaminergic innervation is therefore of great importance. [4]

Increased Dopaminergic Innervation

Here, we show an increased dopaminergic innervation of the ischemic territory during the first week after stroke induced in Wistar rats subjected to transient occlusion of the middle cerebral artery (tMCAO) for 120 min. [1] In rodent models, exposure to 17‐OHPC significantly increased dopaminergic innervation of the medial prefrontal cortex (mPFC), an aberrant pattern of connectivity that may underlie deficits in cognitive flexibility observed in adulthood. [2] Increased dopaminergic innervation of the striatum and improved behavioral recovery were observed with the addition of BDNF. [3]