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Atp Supply sentence examples within pentose phosphate pathway



Regulation of amino acid and nucleotide metabolism by crustacean hyperglycemic hormone in the muscle and hepatopancreas of the crayfish Procambarus clarkia



UHPLC-Q-TOF/MS based metabolomics reveal the mechanism of MeJA in delaying the deterioration of Agaricus bisporus.


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UHPLC-Q-TOF/MS based metabolomics reveal the mechanism of MeJA in delaying the deterioration of Agaricus bisporus.



Ultrahigh-Pressure Liquid Chromatography-Quadrupole-Time-of-Flight Mass Spectrometry-Based Metabolomics Reveal the Mechanism of Methyl Jasmonate in Delaying the Deterioration of Agaricus bisporus


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10.1007/s00424-019-02304-0

Isoproterenol enhances force production in mouse glycolytic and oxidative muscle via separate mechanisms


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10.1007/978-981-13-8946-7_28

Targeting Mitochondria for Therapy of Cardiovascular Disease



Mitochondria supply ATP to the ER through a mechanism antagonized by cytosolic Ca2+



Combined docosahexaenoic acid and thyroid hormone supplementation as a protocol supporting energy supply to precondition and afford protection against metabolic stress situations


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10.1002/cbic.201900612

The Multifaceted Inhibitory Effects of an Alkylquinolone on the Diatom Phaeodactylum tricornutum


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10.1016/j.molbiopara.2019.02.002

Control and regulation of the pyrophosphate-dependent glucose metabolism in Entamoeba histolytica.


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10.1016/j.cbpc.2019.01.004

Acute crude oil exposure alters mitochondrial function and ADP affinity in cardiac muscle fibers of young adult Mahi-mahi (Coryphaena hippurus).



UPLC-QTOF/MS-based metabolomics reveals the mechanism of chronic unpredictable mild stress-induced hypertension in rats.



First person – Topaz Altman


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10.1016/j.bpc.2019.106208

Integration of demand and supply sides in the ATP energy economics of cells.


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10.3390/molecules24081583

Oxidative Stress: A Key Modulator in Neurodegenerative Diseases


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10.1016/j.bbabio.2019.06.017

Mitochondrial respirasome works as a single unit and the cross-talk between complexes I, III2 and IV stimulates NADH dehydrogenase activity.


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