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10.1007/s00213-021-05989-3
However, there was insufficient evidence for the association between antibiotic intake after birth and ADHD risk.
However, there was insufficient evidence for the association between antibiotic intake after birth and ADHD risk.
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10.1177/1087054720914352
Quantifying early health services use among children later diagnosed with ADHD could help us understand the early life impact of the disorder and uncover health care utilization patterns associated with higher ADHD risk.
Quantifying early health services use among children later diagnosed with ADHD could help us understand the early life impact of the disorder and uncover health care utilization patterns associated with higher ADHD risk.
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10.1017/CTS.2021.707
5-methoxytryptophol (5-MTX), a neuroendocrine molecule which also has antioxidant and immunomodulatory properties, was inversely associated with acetaminophen and ADHD risk.
5-methoxytryptophol (5-MTX), a neuroendocrine molecule which also has antioxidant and immunomodulatory properties, was inversely associated with acetaminophen and ADHD risk.
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10.1101/2021.02.16.21251737
If confirmed, these findings support previous evidence that maternal ASD or neuroticism may be possible targets for intervention to help break the chain of the intergenerational transmission of ADHD risk.
If confirmed, these findings support previous evidence that maternal ASD or neuroticism may be possible targets for intervention to help break the chain of the intergenerational transmission of ADHD risk.
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10.1016/j.envint.2021.106549
Simultaneously decreasing all constituents of common-detect OPE-phthalate mixture, specifically DPHP, DNBP, and 6 phthalate metabolites, by a quartile resulted in an ADHD risk ratio of 0.
Simultaneously decreasing all constituents of common-detect OPE-phthalate mixture, specifically DPHP, DNBP, and 6 phthalate metabolites, by a quartile resulted in an ADHD risk ratio of 0.
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10.1007/s00787-021-01860-0
Increased ADHD risk in children in terms of hazard ratios (HRs) and 95% confidence intervals (CIs) was associated with prenatal exposure to paternal autoimmune diseases, including Sjögren’s syndrome (HR: 8.
Increased ADHD risk in children in terms of hazard ratios (HRs) and 95% confidence intervals (CIs) was associated with prenatal exposure to paternal autoimmune diseases, including Sjögren’s syndrome (HR: 8.
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10.1101/2021.08.06.21261679
In analyses of the polygenic architecture, we found higher polygenic score (PGS) of ADHD risk variants in persistent ADHD (mean PGS=0.
In analyses of the polygenic architecture, we found higher polygenic score (PGS) of ADHD risk variants in persistent ADHD (mean PGS=0.
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10.1002/ajmg.b.32844
Interestingly, a causal effect from cannabis use to ADHD risk was also observed for the first time.
Interestingly, a causal effect from cannabis use to ADHD risk was also observed for the first time.
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10.1101/2021.03.30.21254492
We used genetic variants from alcohol metabolising genes (alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH)) as proxies for fetal alcohol exposure to investigate their association with offspring ADHD risk around age 7-8.
We used genetic variants from alcohol metabolising genes (alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH)) as proxies for fetal alcohol exposure to investigate their association with offspring ADHD risk around age 7-8.
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10.1093/ACREFORE/9780190632366.013.181
While genetic factors contribute to ASD and ADHD risk, the environmental contribution to ASD and ADHD has been recognized as having potentially equal importance, which raises the hope for early prevention and intervention.
While genetic factors contribute to ASD and ADHD risk, the environmental contribution to ASD and ADHD has been recognized as having potentially equal importance, which raises the hope for early prevention and intervention.
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10.3390/ijerph18094939
Dose–response analysis for parental age and ADHD risk was performed.
Dose–response analysis for parental age and ADHD risk was performed.
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10.1101/2021.03.25.21254087
We investigated associations between maternal prenatal smoking, alcohol and caffeine consumption with childhood ADHD risk accounting for shared familial factors.
We investigated associations between maternal prenatal smoking, alcohol and caffeine consumption with childhood ADHD risk accounting for shared familial factors.
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10.33774/COE-2021-XZ72R
Although ADHD is highly heritable, environmental factors also appear to contribute to ADHD risk.
Although ADHD is highly heritable, environmental factors also appear to contribute to ADHD risk.
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10.1101/2021.08.18.21262211
We derived ADHD-PRS and identified 25 ADHD risk-factors in Danish national registers.
We derived ADHD-PRS and identified 25 ADHD risk-factors in Danish national registers.
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10.25972/OPUS-23626
The study of ADHD risk genes such as ADGRL3 requires the gene to be first identified using human studies.
The study of ADHD risk genes such as ADGRL3 requires the gene to be first identified using human studies.
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